American Autoimmune Related Diseases Association (AARDA) announced today its latest Autoimmune Heroes – the 22 Scientific Advisory Board (SAB) members who have loyally and generously donated their time and talent to helping AARDA realize its...
AUTOIMMUNITY
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Vitiligo is an Autoimmune Disease Targeting the Skin https://t.co/gaGIMXieZd
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Type I interferon is a potent substance. As such, the induction, transmission, and resolution of the type I interferon–mediated immune response are tightly regulated. As defined, the type I interferonopathies represent discrete examples of a disturbance of the homeostatic control of this system caused by Mendelian mutations. Considering the complexity of the interferon response, the identification of further monogenic diseases belonging to this disease grouping seems likely, with the recognition of type I interferonopathies becoming of increasing clinical importance as treatment options are developed based on an understanding of disease pathology and innate immune signaling. Definition of the type I interferonopathies indicates that autoinflammation can be both interferon and noninterferon related, and that a primary disturbance of the innate immune system can “spill over” into autoimmunity in some cases. Indeed, that several non-Mendelian disorders, most particularly systemic lupus erythematosus and dermatomyositis, are also characterized by an up-regulation of type I interferon signaling suggests the possibility that insights derived from this work will have relevance to a broader field of clinical medicine.
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Researchers at the University of Liverpool have taken a major step forward in understanding the causes of a disorder which causes chronic pain in sufferers.
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"Parallel use of 16S profiling & virome sequencing in stool samples from children with islet autoimmunity...":
https://t.co/FjqcJ7Fh3G
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Editorial: Vascular Inflammation in Systemic Autoimmunity: Giuseppe A. Ramirez, Cornelia Weyand, Augusto Vaglio,…… https://t.co/ZXzeCd3IMa
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Credit: EPFL
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The activation of Fcγ receptors by autoantibody immune complexes plays a pathogenic role in multiple autoimmune diseases. In an attempt to systematically evaluate potential therapeutic approaches, Ortiz et al .
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Type 1 Diabetes
In the case of autoimmune diseases, such as type 1 diabetes, so-called “exhausted” T cells may be the answer to stopping disease. Long et al.
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#type1diabetes: #TCellexhaustion markers correlate with patients’ response to #antiCD3 #teplizumab @sciencemagazine… https://t.co/VY6u3fs9An
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A group of researchers from UMass Medical School has isolated immune cells from the islets of nine donors with type 1 diabetes to understand and prevent the immune-mediated destruction of beta cells.
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Diagnosis and treatment of autoimmunity has mainly relied on adaptive immunity. Infection
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Perspective on autoimmunity: a view from the ANCA vasculitis looking glass Air date: Wednesday, November 2, 2016, 3:00:00 PM Category: WALS - Wednesda
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Semin Immunopathol. 2016 Nov 25. [Epub ahead of print] Review
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IL-23 assists the transition of autoimmunity to inflammatory disease @NatRevRheumatol @NatImmunol https://t.co/uSyppq7j4A
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Clin Exp Immunol. 2016 Nov 23. doi: 10.1111/cei.12903. [Epub ahead of print] Review
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IDO2: A Pathogenic Mediator of Inflammatory Autoimmunity
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By photolabelling intestinal cells, Krebs and colleagues provide direct evidence that
Gilbert C FAURE's insight:
anca nephritis
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Catastrophic antiphospholipid syndrome (CAPS): Descriptive analysis of 500 patients from the International CAPS Registry analysis of 50
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Airway abnormalities and lung tissue citrullination are found in both rheumatoid arthritis (RA) patients and individuals at-risk for disease development.
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Fascinating work by Martin Kriegel - gut bacteria as possible initiators of autoimmunity in APS https://t.co/DHdyufktEX
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Speakers now available online #8thAutoimmunity2017 Conference held on #Rhodes, #Geece https://t.co/qqCCOPiz0K #Autoimmunity #autoimmune
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The human body is made up of 37 trillion cells and billions of these die every single day. The body has special cells, called macrophages, which consume dying cells to prevent them from building up in your body.
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