Mobility & Flexibility - Joint Movement
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Mobility & Flexibility - Joint Movement
The human body retains an individual, natural level of mobility and flexibility to ensure all of its structures are functioning properly. The bones, muscles, ligaments, tendons, and other tissues work together to allow a range of movement and maintaining proper fitness and balanced nutrition can help keep the body functioning properly. According to Dr. Alex Jimenez’s compilation of articles pertaining to mobility and flexibility, individuals who don’t stretch their body often can experience shortened or stiffened muscles which decrease their ability to move effectively. For more information, please feel free to contact us at (915) 850-0900 or text to call Dr. Jimenez personally at (915) 850-0900. https://www.dralexjimenez.com  Book Appointment Today: https://bit.ly/Book-Online-Appointment
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March 28, 2018 9:19 PM
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Chiropractic Helps Tendonitis In El Paso, TX. | Doctor Of Chiropractic

Chiropractic Helps Tendonitis In El Paso, TX. | Doctor Of Chiropractic | Mobility & Flexibility - Joint Movement | Scoop.it

Chiropractic Helps: Tendonitis is a condition that occurs when a tendon that connects bone to muscle is injured, overused, or used improperly. The result is inflammation, pain and sometimes swelling. In the case of an injury where the tendon is stressed to otherwise injured, soft tissue around the area may also become involved. Each year more than 4 million people in the U.S. seek medical treatment for symptoms of tendonitis.

 

Common types of tendonitis include runner’s knee, tennis elbow, and pitcher’s shoulder. However, other tendons in the body can also experience tendonitis symptoms. The Achilles tendon, ankle, wrist, and even fingers can all become inflamed from the condition.

 

Often a primary care physician diagnoses the injury, but once there is a definitive diagnosis, the best treatment is one that is natural and holistic. Chiropractic care is an outstanding choice for treatment.

Chiropractic Helps: Thorough Healing

Tendonitis often recurs in many people. This is due, at least in part to improper or incomplete healing. Many people will resume normal activities as soon as the pain subsides when they should continue caring for the injury so that it can heal completely. This leads to inflammation of the area again as the original injury is aggravated, but it can also lead to re-injury in that same area. Chiropractic care can help tendonitis heal completely and help prevent re-injury to the area.

Chiropractic Helps: Tendonitis

Since tendonitis symptoms can mimic other serious conditions, the first step in treatment is confirming the diagnosis. The doctor may use X-rays, MRIs, or CAT scans to make a positive diagnosis of tendonitis.

 

From there, the chiropractor will work with the patient to develop a treatment plan that is natural and effective, tailored to the patient’s unique needs. At the core of this treatment is a targeted plan that treats the cause of the problem, not just the symptoms.

 

If there is a great deal of inflammation the chiropractor may employ inflammation reducing techniques such as bracing, rest, ice, electrical muscle stimulation, and other therapies. This helps to loosen the tendon and lessen the inflammation so that treatment can progress.

 

Once the inflammation is under control, the chiropractor may begin massage, physical therapy, joint manipulation, or strengthening exercises. If the patient has diminished or limited joint mobility then joint manipulation or adjustments may be the best option.

Chiropractic Helps: Length Of Treatment

The initial pain may subside or even disappear within the first two to three weeks. However, that does not mean that the patient is out of the woods. This is where many patients go wrong. They mistakenly believe that once the pain is gone the condition is healed.

 

This is not true. While the inflammation may indeed decrease over the first three weeks, the complete healing takes around six weeks and often longer. Scar tissue is formed during this time which helps the body repair the injury. Once the injury is repaired though, that scar tissue must be broken down in order for the area to regain its mobility and flexibility.

 

The chiropractor will use various techniques including massage and ultrasound to break down the scar tissue. Mild stretches may be incorporated into the treatment plan. Once the tissues are fully healed, exercise can aid in breaking down the scar tissue even more. These exercises will work the muscles but they won’t involve the tendons.

Chiropractic Helps: Healing Tendonitis For Good

Tendonitis can be healed completely and a good chiropractor can help. Most chiropractors take a whole body approach, including diet, specific supplements, and lifestyle choices that will help the healing process of tendonitis. Once chiropractic treatment is complete for tendonitis, the patient can usually return to normal activities without the risk of re-injury or re-inflammation.

 

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. A primary care physician diagnoses tendonitis, but once there is a definitive diagnosis, the best treatment is one that is natural and holistic. Chiropractic helps and is an outstanding choice for treatment. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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March 19, 2018 8:50 PM
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Carpal Tunnel Pain Treatment El Paso, TX | Ottis Hamlet

Carpal Tunnel Pain Treatment El Paso, TX | Ottis Hamlet | Mobility & Flexibility - Joint Movement | Scoop.it

Carpal Tunnel Pain: Ottis Hamlet depends largely on the use of his hands to carry out his important craftsmanship in the city of San Antonio, TX. However, Mr. Hamlet developed painful symptoms in both of his arms as a result of carpal tunnel syndrome, which tremendously affected his ability to engage in his job. Fortunately, Ottis Hamlet met Dr. Alex Jimenez during a visit to El Paso, TX and he received chiropractic treatment for his carpal tunnel syndrome, avoiding the need for surgery.

 

Carpal Tunnel Pain Treatment El Paso, TX | Ottis Hamlet

 

Carpal tunnel syndrome is a medical condition caused by the compression of the median nerve which travels through the wrist and into the carpal tunnel. Common symptoms include pain, tingling sensations and numbness, in the thumb, index finger, middle finger, and the thumb side of the ring fingers. Symptoms normally start gradually and during the evening. Symptoms can extend throughout the arm and weakened grip strength may also occur. Carpal tunnel syndrome can be diagnosed based on symptoms.

 

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Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Carpal tunnel pain is caused by the compression of the median nerve which travels through the wrist and into the carpal tunnel. Symptoms include pain, tingling sensations and numbness. Chiropractic treatment benefits carpal tunnel syndrome & avoids the need for surgery. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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February 27, 2018 5:23 PM
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Yoga Increases Effectiveness Of Chiropractic Treatment | El Paso, TX

Yoga Increases Effectiveness Of Chiropractic Treatment | El Paso, TX | Mobility & Flexibility - Joint Movement | Scoop.it

Yoga  increases the effectiveness of chiropractic treatment hasn't taken the world by storm, but it has gained steadily in popularity in the last decade. Originally developed thousands of years ago, the practice of breathing, posing, and stretching offers a variety of important health benefits. Reduced stress and blood pressure, increased flexibility and stamina, greater balance, and a better understanding of breathing techniques are all fabulous results of practicing yoga.

 

In addition to the normal exercise benefits yoga offers, the practice serves as treatment for a variety of injuries and health conditions. Yoga embodies the recognition of the link between physical, emotional, and spiritual wellness, and reaches its fans in deeper ways than other forms of traditional exercise.

 

Although the two disciplines come from entirely different histories, the concept that yoga heals the body in its entirety closely mirrors the concept behind chiropractic care. These similar foundations offer enormous benefits to those suffering from a variety of injuries and conditions that seek help from a chiropractor. By incorporating yoga sessions into their treatment, chiropractic patients often reap great rewards.

 

Here are four reasons why:

Yoga Primes The Body For Healing

Practicing yoga stretches and elongates the body's muscles, releasing tension and stress. Before patients visit their chiropractors, yoga can serve to warm up their bodies and clarify their minds, so the chiropractor can dig into the root problem. Yoga complements chiropractic treatment by readying the body to heal itself.

Yoga Increases Joint & Ligament Strength

Dealing with a health condition or injury is frustrating and can seem like it takes forever to heal. Implementing yoga into a recovery plan helps strengthen joints and ligaments, which aids in promoting healing and cutting down the time it takes to get better.

 

Yoga works on the body as a whole and promotes greater well-being from head to toe. A stronger body absorbs chiropractic care more productively and deeply than one that is stiff, sore, and unhealthy.

Yoga Increases Range Of Motion

Depending on the severity of the individual's specific condition, chiropractic patients may need several visits to "prep" their bodies before the main issue can even be addressed. Yoga sessions increase a body's flexibility and help with the range of motion in the neck, back, hips, and other joints. By going into a chiropractic treatment equipped with better range of motion, the patient is more pliable, and the visit is able to offer more in-depth adjustments, for greater results.

Prevents Future Injury

Many reasons individuals seek chiropractic treatment are for recurring issues. Yoga provides an ongoing way for patients dealing with chronic issues to manage and reduce instances of pain, inflammation, and other symptoms. A regimen of yoga coupled with chiropractic care keeps the body aligned, its balance level, the muscles stretched and de-stressed, and the joints operating efficiently.

 

Both also serve to keep posture correct, and ligaments strong. All of these results create a body that is less susceptible to future injury, and less inclined for previous medical conditions to resurface. Individuals enjoy greater mobility longer, all without changing their active lifestyles.

 

Yoga has long been touted as the answer for many of the body's issues, and it's smart for individuals, whether dealing with current issues or in prime health, to check out the basics of yoga. Chiropractic care coupled with yoga offers a great many benefits to patients who are dealing with medical conditions or injury. Ask your chiropractor for an evaluation of your health issues, and whether adding yoga to your overall health plan would help you lead a better, more active life.

Chiropractic Keeps Nastia Liukin's Body In Balance

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Chiropractic care coupled with yoga increases many benefits to patients who are dealing with medical conditions or injury. Ask your chiropractor for an evaluation of your health issues, and whether adding yoga to your overall health plan would help you lead a better, more active life. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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February 26, 2018 6:43 PM
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Benefits Plantar Fasciitis Sufferers Chiropractic Treatment | El Paso, TX

Benefits Plantar Fasciitis Sufferers Chiropractic Treatment | El Paso, TX | Mobility & Flexibility - Joint Movement | Scoop.it

One of the most difficult medical conditions to spell is also one of the most common. Plantar fasciitis is the most common cause of heel pain. A person is afflicted with this medical condition when the tissue tears in the long ligament that runs along the bottom of the foot, called the plantar fascia ligament. The resulting symptoms include pain and inflammation that can be acute and often ongoing.

Plantar Fasciitis

It's estimated that 2 million Americans suffer from plantar fasciitis. However, many different factors cause the condition.

 

A foot trauma from an injury such as a fall can bring about the condition. Other causes are wearing ill-fitting or non-supporting footwear, prolonged standing, and arthritis. Once afflicted with plantar fasciitis, the sufferer often changes their gait to avoid foot pain, bringing on secondary issues such as misalignment and joint stress.

 

While there are several modes of treatment options, chiropractic care offers multiple unique benefits to those who suffer from plantar fasciitis. Here are four specific ways chiropractic care effectively treats plantar fasciitis.

Chiropractic Adjustments Can Reduce Stress In The Plantar fascia

When the ligament is stressed, it can cause tiny tears that brings on plantar fasciitis. Sufferers who don't take measures to repair this damage often experience ongoing pain and inflammation. A chiropractor, over a series of visits, is able to adjust the foot and heel so the ligament starts to relax, which in return, promotes healing and diminishes the instances of dealing with the condition again down the road.

Chiropractic Care Helps Minimize Secondary Bodily Injury Due To Compensation

As mentioned above, individuals dealing with the pain of plantar fasciitis frequently adapt their gait to avoid painful steps, causing stress and weight to fall on other parts of the feet, ankles, and joints. This may eventually cause issues with strained muscles and sore joints.

 

Chiropractic treatment not only deals with the symptoms, but treats the root of the problem. Patients who commit to chiropractic care see the plantar fasciitis decrease in severity. In addition, the chiropractor helps re-train them to walk and stand correctly, taking care of the secondary issues.

Additional At Home Exercises Promote Healing

Patients can help their situations in addition to visiting their chiropractor by taking advantage of regular home therapy exercises. Part of chiropractic care for plantar fasciitis includes a regular recommendation of exercises that stretches and heals the plantar fascia as well as secondary affected areas. For maximum results, patients need to make sure they perform the exercises correctly and diligently stick to the rehabilitation plan.

Chiropractic Works Well In Conjunction With Other Treatments

Chiropractic treatment for plantar fasciitis complements other treatments. Chiropractic visits paired with massage, physical therapy, and more invasive treatment such as injections to offer pain management, increased mobility, and faster healing. Talk with your chiropractor to see what other treatments may complement your current care.

 

The not so great news is plantar fasciitis's typical recovery time is several months. The great news is that committing to a combination of chiropractic visits and therapy exercises heals 9 out of 10 cases.

 

Plantar fasciitis is a common issue that millions of people face, but it doesn't have to control your activity level or hinder your lifestyle. Consult a chiropractor and work together to lay out a plan of chiropractic adjustments, at-home rehab, and possibly other complementary forms of treatments. It may take time, but plantar fasciitis sufferers can eventually reach a point where they are pain free and their mobility is unhindered!

Jerry Rice Credits Chiropractic Treatment

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX Treatment. Plantar fasciitis is the most common cause of heel pain. A person is afflicted with this medical condition when the tissue tears in the long ligament that runs along the bottom of the foot, called the plantar fascia ligament. The resulting symptoms include pain and inflammation. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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February 19, 2018 8:22 PM
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Ankle Injuries: Chiropractic Care Rehab | El Paso Chiropractor

Ankle Injuries: Chiropractic Care Rehab | El Paso Chiropractor | Mobility & Flexibility - Joint Movement | Scoop.it

An ankle can be injured as glamorously as falling off a $400 platform stiletto heel, stepping off a Parisian curb, or as mundanely as tripping over a toy truck, or falling over a rock on your way to the mailbox. No matter the cause, ankle injuries are painful and problematic, and cause recurring problems if left untreated.

 

The poor ankle sure has it rough. It supports a person's entire body weight, twists and turns many times a day, and maintains proper balance. This heavy responsibility takes its toll. Emergency rooms treat approximately one million patients ever year for ankle injuries.

 

Ankles are technically "the joint where the foot joins the leg."  In reality, there are more moving parts involved than that simplified definition allows. Multiple bones and two separate joints actually converge in the ankle area, which increases the chance of an ankle injury.

Ankle Injuries:

 

Sprains:

When you roll your ankle outward, the movement damages the ligaments on the outside of the ankle. This is a common sports injury and, unfortunately, once you have sprained your ankle it's more likely to recur. Up to half of the people who suffer from a sprained ankle will sprain it again.

Strains:

There are two tendons in the ankle that are commonly strained, usually over stretching from overuse or trauma.

Fractures:

This injury happens when one or more of the three bones in the ankle is injured. While less common than a sprain or strain, a fractured ankle may also involve damaged ligaments and require surgery.

 

Many instances of ankle injuries are avoidable. Be sure to wear proper shoes when exercising or participating in sports, avoid uneven walking surfaces, and keep stairways and floors in your home clear of clutter

 

And, ladies, avoid the really high heels. We know, we know, they are just so cute! :-)

 

Even with ankle-protecting precautions, you still may end up on your rump in the grass nursing your swollen ankle. What should you do if you injure your ankle? There are several forms of treatment for an ankle injury depending on its severity.

 

Rest and ice: For mild injuries, stay off your ankle and use ice packs to reduce the swelling. Rest allows the injured area to heal faster.

 

Visit a doctor: If you experience severe pain, swelling, and are unable to put weight on your ankle, see a doctor, as some ankle injuries grow worse without treatment. Injuries may require a brace, cast, or even surgery.

 

See a chiropractor: Patients frequently see strongly positive results in ankle injuries from a series of chiropractic treatments. Chiropractors understand the way the ankle is built, and use chiropractic adjustments to reduce pain and inflammation and promote faster healing.

 

Exercise rehab: Once you are healed, it's vital to build up the ankle's strength to avoid re-injury. Your chiropractor can lay out an exercise routine that you can employ into your regular workouts that will improve your balance and increase mobility. Performing these moves helps dramatically decrease dealing with this again down the road.

 

Ankle injuries are common and, whether or not you maintain an active lifestyle, you may end up suffering from one. By visiting a chiropractor on the front end, you can better plan a course of treatment that will heal your ankle quickly, reduce the pain effectively, and minimize the chance of a recurrence.

Golf & Chiropractic Care

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Ankles are technically "the joint where the foot joins the leg."  In reality, there are more moving parts involved than that simplified definition allows. Multiple bones and two separate joints actually converge in the ankle area, which increases the chance of ankle injuries. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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January 15, 2018 5:08 PM
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Muscle Energy Techniques (MET): Introduction | El Paso Back Clinic ®

Muscle Energy Techniques (MET): Introduction | El Paso Back Clinic ® | Mobility & Flexibility - Joint Movement | Scoop.it

Muscle Energy Techniques: A revolution has taken place in manipulative therapy involving a movement away from high velocity/low amplitude thrusts (HVT – now commonly known as ‘mobilization with impulse’ and characteristic of most chiropractic and, until recently, much osteopathic manipulation) towards gentler methods which take far more account of the soft tissue component (DiGiovanna 1991, Lewit 1999, Travell & Simons 1992).

 

Greenman (1996) states that: ‘Early [osteopathic] techniques did speak of muscle relaxation with soft tissue procedures, but specific manipulative approaches to muscle appear to be 20th century phenomena.’ One such approach – which targets the soft tissues primarily, although it also makes a major contribution towards joint mobilization – has been termed muscle energy technique (MET) in osteopathic medicine. There are a variety of other terms used to describe this approach, the most general (and descriptively accurate) of which was that used by chiropractor Craig Liebenson (1989, 1990) when he described muscle energy techniques as ‘active muscular relaxation techniques’. Muscle energy techniques evolved out of osteopathic procedures developed by pioneer practitioners such as T. J. Ruddy (1961), who termed his approach ‘resistive duction’, and Fred Mitchell Snr (1967). As will become clear in this chapter, there also exists a commonality between Muscle energy techniques and various procedures used in orthopaedic and physiotherapy methodology, such as proprioceptive neuromuscular facilitation (PNF). Largely due to the work of experts in physical medicine such as Karel Lewit (1999), MET has evolved and been refined, and now crosses all interdisciplinary boundaries.

 

MET has as one of its objectives the induced relaxation of hypertonic musculature and, where appropriate (see below), the subsequent stretching of the muscle. This objective is shared with a number of ‘stretching’ systems, and it is necessary to examine and to compare the potential benefits and drawbacks of these various methods (see Box 1.1).

 

MET, as presented in this book, owes most of its development to osteopathic clinicians such as T. J. Ruddy (1961) and Fred Mitchell Snr (1967), with more recent refinements deriving from the work of people such as Karel Lewit (1986, 1999) and Vladimir Janda (1989) of the former Czechoslovakia, both of whose work will be referred to many times in this text.

 

T. J. Ruddy (1961)

 

In the 1940s and 50s, osteopathic physician T. J. Ruddy developed a treatment method involving patient-induced, rapid, pulsating contractions against resistance which he termed ‘rapid resistive duction’. It was in part this work which Fred Mitchell Snr used as the basis for the evolution of MET (along with PNF methodology, see Box 1.1). Ruddy’s method called for a series of rapid, low amplitude muscle contractions against resistance, at a rate a little faster than the pulse rate. This approach is now known as pulsed MET, rather than the tongue-twisting ‘Ruddy’s rapid resistive duction’.

 

As a rule, at least initially, these patient-directed pulsating contractions involve an effort towards the barrier, using antagonists to shortened structures. This approach can be applied in all areas where sustained contraction muscle energy technique procedures are appropriate, and is particularly useful for self-treatment, following instruction from a skilled practitioner. Ruddy suggests that the effects include improved local oxygenation, venous and lymphatic circulation, as well as a positive influence on both static and kinetic posture, because of the effects on proprioceptive and interoceptive afferent pathways.

 

Ruddy’s work formed part of the base on which Mitchell Snr and others constructed MET and aspects of its clinical application are described in Chapter 3.

 

Fred Mitchell Snr

 

No single individual was alone responsible for MET, but its inception into osteopathic work must be credited to F. L. Mitchell Snr, in 1958. Since then his son F. Mitchell Jnr (Mitchell et al 1979) and many others have evolved a highly sophisticated system of manipulative methods (F. Mitchell Jnr, tutorial on biomechanical procedures, American Academy of Osteopathy, 1976) in which the patient ‘uses his/her muscles, on request, from a precisely controlled position in a specific direction, against a distinctly executed counterforce’.

 

Philip Greenman


Professor of biomechanics Philip Greenman (1996) states that:

 

The function of any articulation of the body which can be moved by voluntary muscle action, either directly or indirectly, can be influenced by muscle energy procedures ... . Muscle energy techniques can be used to lengthen a shortened, contractured or spastic muscle; to strengthen a physiologically weakened muscle or group of muscles; to reduce localized edema, to relieve passive congestion, and to mobilize an articulation with restricted mobility.

 

Sandra Yale


Osteopathic physician Sandra Yale (in DiGiovanna 1991) extols MET’s potential in even fragile and severely ill patients:

 

Muscle energy techniques are particularly effective in patients who have severe pain from acute somatic dysfunction, such as those with a whiplash injury from a car accident, or a patient with severe muscle spasm from a fall. MET methods are also an excellent treatment modality for hospitalized or bedridden patients. They can be used in older patients who may have severely restricted motion from arthritis, or who have brittle osteoporotic bones.

 

Edward Stiles


Among the key MET clinicians is Edward Stiles, who elaborates on the theme of the wide range of MET application (Stiles 1984a, 1984b). He states that:

 

Basic science data suggests the musculoskeletal system plays an important role in the function of other systems. Research indicates that segmentally related somatic and visceral structures may affect one another directly, via viscerosomatic and somaticovisceral reflex pathways. Somatic dysfunction may increase energy demands, and it can affect a wide variety of bodily processes; vasomotor control, nerve impulse patterns (in facilitation), axionic flow of neurotrophic proteins, venous and lymphatic circulation and ventilation. The impact of somatic dysfunction on various combinations of these functions may be associated with myriad symptoms and signs. A possibility which could account for some of the observed clinical effects of manipulation.

 

As to the methods of manipulation he now uses clinically, Stiles states that he employs muscle energy methods on about 80% of his patients, and functional techniques (such as strain/counterstrain) on 15–20%. He uses high velocity thrusts on very few cases. The most useful manipulative tool available is, he maintains, muscle energy techniques.

 

J. Goodridge and W. Kuchera


Modern osteopathic refinements of MET – for example the emphasis on very light contractions which has strongly influenced this text – owe much to physicians such as John Goodridge and William Kuchera, who consider that (Goodridge & Kuchera 1997):

 

Localization of force is more important than intensity. Localization depends on palpatory proprioceptive perception of movement (or resistance to movement) at or about a specific articulation ... . Monitoring and confining forces to the muscle group or level of somatic dysfunction involved are important for achieving desirable changes. Poor results are most often due to improperly localized forces, often with excessive patient effort.

Early Sources Of Muscle Energy Techniques

MET emerged squarely out of osteopathic tradition, although a synchronous evolution of treatment methods, involving isometric contraction and stretching, was taking place independently in physical therapy, called PNF (see Box 1.1).

 

Fred Mitchell Snr (1958) quoted the words of the developer of osteopathy, Andrew Taylor Still: ‘The attempt to restore joint integrity before soothingly restoring muscle and ligamentous normality was putting the cart before the horse.’ As stated earlier, Mitchell’s work drew on the methods developed by Ruddy; however, it is unclear whether Mitchell Snr, when he was refining MET methodology in the early 1950s, had any awareness of proprioceptive neuromuscular facilitation (PNF), a method which had been developed a few years earlier, in the late 1940s, in a physical therapy context (Knott & Voss 1968).

 

PNF method tended to stress the importance of rotational components in the function of joints and muscles, and employed these using resisted (isometric) forces, usually involving extremely strong contractions. Initially, the focus of PNF related to the strengthening of neurologically weakened muscles, with attention to the release of muscle spasticity following on from this, as well as to improving range of motion at intervertebral levels (Kabat 1959, Levine et al 1954) (see Box 1.1).

Postisometric Relaxation & Reciprocal Inhibition: Two Forms Of MET (Box 1.2)

A term much used in more recent developments of muscle energy techniques is postisometric relaxation (PIR), especially in relation to the work of Karel Lewit (1999). The term postisometric relaxation refers to the effect of the subsequent reduction in tone experienced by a muscle, or group of muscles, after brief periods during which an isometric contraction has been performed.

The terms proprioceptive neuromuscular facilitation (PNF) and postisometric relaxation (PIR) (the latent hypotonic state of a muscle following isometric activity) therefore represent variations on the same theme. A further variation involves the physiological response of the antagonists of a muscle which has been isometrically contracted – reciprocal inhibition (RI).

 

 

When a muscle is isometrically contracted, its antagonist will be inhibited, and will demonstrate reduced tone immediately following this. Thus the antagonist of a shortened muscle, or group of muscles, may be isometrically contracted in order to achieve a degree of ease and additional movement potential in the shortened tissues.

 

Sandra Yale (in DiGiovanna 1991) acknowledges that, apart from the well understood processes of reciprocal inhibition, the precise reasons for the effectiveness of MET remain unclear – although in achieving PIR the effect of a sustained contraction on the Golgi tendon organs seems pivotal, since their response to such a contraction seems to be to set the tendon and the muscle to a new length by inhibiting it (Moritan 1987). Other variations on this same theme include ‘hold–relax’ and ‘contract–relax’ techniques (see Box 1.1).

 

Lewit & Simons (1984) agree that while reciprocal inhibition is a factor in some forms of therapy related to postisometric relaxation techniques, it is not a factor in PIR itself, which is a phenomenon resulting from a neurological loop, probably involving the Golgi tendon organs (see Figs 1.1 and 1.2).

 

Liebenson (1996) discusses both the benefits of, and the mechanisms involved in, use of muscle energy techniques (which he terms ‘manual resistance techniques’, or MRT):

 

Two aspects to MRT [i.e. MET by another name] are their ability to relax an overactive muscle ... and their ability to enhance stretch of a shortened muscle or its associated fascia when connective tissue or viscoelastic changes have occurred.

 

Two fundamental neurophysiological principles account for the neuromuscular inhibition that occurs during application of these techniques. The first is postcontraction inhibition [also known as postisometric relaxation, or PIR], which states that after a muscle is contracted, it is automatically in a relaxed state for a brief, latent, period. The second is reciprocal inhibition (RI) which states that when one muscle is contracted, its antagonist is automatically inhibited.

 

Liebenson suggests that there is evidence that the receptors responsible for PIR lie within the muscle and not in the skin or associated joints (Robinson 1982).

 

Where pain of an acute or chronic nature makes controlled contraction of the muscles involved difficult, the therapeutic use of the antagonists can patently be of value. Thus modern MET incorporates both postisometric relaxation and reciprocal inhibition methods, as well as aspects unique to itself, such as isokinetic techniques, described later.

 

A number of researchers, including Karel Lewit of Prague (Lewit 1999), have reported on the usefulness of aspects of MET in the treatment of trigger points, and this is seen by many to be an excellent method of treating these myofascial states, and of achieving the restoration of a situation where the muscle in which the trigger lies is once more capable of achieving its full resting length, with no evidence of shortening.

 

Travell & Simons (1992) mistakenly credited Lewit with developing MET, stating that ‘The concept of applying post-isometric relaxation in the treatment of myofascial pain was presented for the first time in a North American journal in 1984 [by Lewit]’. In fact Mitchell Snr had described the method some 25 years previously, a fact acknowledged by Lewit (Lewit & Simons 1984).

Key Points About Modern Muscle Energy Techniques

MET methods all employ variations on a basic theme. This primarily involves the use of the patient’s own muscular efforts in one of a number of ways, usually in association with the efforts of the therapist:

 

1. The operator’s force may exactly match the effort of the patient (so producing an isometric contraction) allowing no movement to occur – and producing as a result a physiological neurological response (via the Golgi tendon organs) involving a combination of:

 

— reciprocal inhibition of the antagonist(s) of the muscle(s) being contracted, as well as

— postisometric relaxation of the muscle(s) which are being contracted.

  1. The operator’s force may overcome the effort of the patient, thus moving the area or joint in the direction opposite to that in which the patient is attempting to move it (this is an isotonic eccentric contraction, also known as an isolytic contraction).
  2. The operator may partially match the effort of the patient, thus allowing, although slightly retarding, the patient’s effort (and so producing an isotonic concentric, isokinetic, contraction).

Other variables may be also introduced, for example involving:

 

l Whether the contraction should commence with the muscle or joint held at the resistance barrier or short of it – a factor decided largely on the basis of the degree of chronicity or acuteness of the tissues involved

  • How much effort the patient uses – say, 20% of strength, or more, or less
  • The length of time the effort is held – 7–10 seconds, or more, or less (Lewit (1999) favours 7– 10 seconds; Greenman (1989), Goodridge & Kuchera (1997) all favour 3–5 seconds)
  • Whether, instead of a single maintained contraction, to use a series of rapid, low amplitude contractions (Ruddy’s rhythmic resisted duction method, also known as pulsed muscle energy techniques)
  • The number of times the isometric contraction (or its variant) is repeated – three repetitions are thought to be optimal (Goodridge & Kuchera 1997)
  • The direction in which the effort is made – towards the resistance barrier or away from it, thus involving either the antagonists to the muscles or the actual muscles (agonists) which require ‘release’ and subsequent stretching (these variations are also known as ‘direct’ and ‘indirect’ approaches, see p. 8)
  • Whether to incorporate a held breath and/or specific eye movements to enhance the effects of the contraction – desirable if possible, it is suggested (Goodridge & Kuchera 1997, Lewit 1999)
  • What sort of resistance is offered (for example by the operator, by gravity, by the patient, or by an immovable object)
  • Whether the patient’s effort is matched, overcome or not quite matched – a decision based on the precise needs of the tissues – to achieve relaxation, reduction in fibrosis or tonifying/ reeducation
  • Whether to take the muscle or joint to its new barrier following the contraction, or whether or not to stretch the area/muscle(s) beyond the barrier – this decision is based on the nature of the problem being addressed (does it involve shortening? fibrosis?) and its degree of chronicity
  • Whether any subsequent (to a contraction) stretch is totally passive, or whether the patient should participate in the movement, the latter being thought by many to be desirable in order to reduce danger of stretch reflex activation (Mattes 1995)
  • Whether to utilize Muscle energy techniques alone, or in a sequence with other modalities such as the positional release methods of strain/counterstrain, or the ischaemic compression/inhibitory pressure techniques of neuromuscular technique (NMT) – such decisions will depend upon the type of problem being addressed, with myofascial trigger point treatment frequently benefiting from such combinations (see description of integrated neuromuscular inhibition (INIT), p. 197 (Chaitow 1993)).

Greenman summarises the requirements for the successful use of MET in osteopathic situations as ‘control, balance and localisation’. His suggested basic elements of MET include the following:

  • A patient/active muscle contraction, which
    — commences from a controlled position
    — is in a specific direction (towards or away from a restriction barrier)
  • The operator applies distinct counterforce (to meet, not meet, or to overcome the patient’s force)
  • The degree of effort is controlled (sufficient to obtain an effect but not great enough to induce trauma or difficulty in controlling the effort).

What is done subsequent to the contraction may involve any of a number of variables, as will be explained.

 

The essence of MET then is that it uses the energy of the patient, and that it may be employed in one or other of the manners described above with any combination of variables depending upon the particular needs of the case. Goodridge (one of the first osteopaths to train with Mitchell Snr in 1970) summarises as follows: ‘Good results [with MET] depend on accurate diagnosis, appropriate levels of force, and sufficient localization. Poor results are most often caused by inaccurate diagnosis, improperly localized forces, or forces that are too strong’ (Goodridge & Kuchera 1997) (see also Box 1.3).

 

Using agonist or antagonist? (Box 1.4)

 

As mentioned, a critical consideration in MET, apart from degree of effort, duration and frequency of use, involves the direction in which the effort is made. This may be varied, so that the operator’s force is directed towards overcoming the restrictive barrier (created by a shortened muscle, restricted joint, etc.); or indeed opposite forces may be used, in which the operator’s counter-effort is directed away from the barrier.

 

There is general consensus among the various osteopathic experts already quoted that the use of postisometric relaxation is more useful than reciprocal inhibition in normalizing hypertonic musculature. This, however, is not generally held to be the case by experts such as Lewit and Janda, who see specific roles for the reciprocal inhibition variation.

 

 

Osteopathic clinicians such as Stiles and Greenman believe that the muscle which requires stretching (the agonist) should be the main source of ‘energy’ for the isometric contraction, and suggest that this achieves a more significant degree of relaxation, and so a more useful ability to subsequently stretch the muscle, than would be the case were the relaxation effect being achieved via use of the antagonist (i.e. using reciprocal inhibition).

 

Following on from an isometric contraction – whether agonist or antagonist is being used – there is a refractory, or latency, period of approximately 15 seconds during which there can be an easier (due to reduced tone) movement towards the new position (new resistance barrier) of a joint or muscle.

Variations On The Muscle Energy Techniques Theme

Liebenson (1989, 1990) describes three basic variations which are used by Lewit and Janda as well as by himself in a chiropractic rehabilitation setting.

 

Lewit’s (1999) modification of MET, which he calls postisometric relaxation, is directed towards relaxation of hypertonic muscle, especially if this relates to reflex contraction or the involvement of myofascial trigger points. Liebenson (1996) notes that ‘this is also a suitable method for joint mobilisation when a thrust is not desirable’.

 

Lewit’s postisometric relaxation method 

 

(Lewit 1999)

 

  1. The hypertonic muscle is taken, without force or ‘bounce’, to a length just short of pain, or to the point where resistance to movement is first noted (Fig. 1.3).
  2. The patient gently contracts the affected hypertonic muscle away from the barrier (i.e. the agonist is used) for between 5 and 10 seconds, while the effort is resisted with an exactly equal counterforce. Lewit usually has the patient inhale during this effort.
  3. This resistance involves the operator holding the contracting muscle in a direction which would stretch it, were resistance not being offered.
  4. The degree of effort, in Lewit’s method, is minimal. The patient may be instructed to think in terms of using only 10 or 20% of his available strength, so that the manoeuvre is never allowed to develop into a contest of strength between the operator and the patient.
  5. After the effort, the patient is asked to exhale and to let go completely, and only when this is achieved is the muscle taken to a new barrier with all slack removed but no stretch – to the extent that the relaxation of the hypertonic muscles will now allow.
  6. Starting from this new barrier, the procedure is repeated two or three times.
  7. In order to facilitate the process, especially where trunk and spinal muscles are involved, Lewit usually asks the patient to assist by looking with his eyes in the direction of the contraction during the contracting phase, and in the direction of stretch during the stretching phase of the procedure.

The key elements in this approach, as in most MET, involve precise positioning, as well as taking out slack and using the barrier as the starting and ending points of each contraction.

 

What Is Happening?

Karel Lewit, discussing MET methods (Lewit 1999), states that medullary inhibition is not capable of explaining their effectiveness. He considers that the predictable results obtained may relate to the following facts:

  • During resistance using minimal force (isometric contraction) only a very few fibers are active, the others being inhibited
  • During relaxation (in which the shortened musculature is taken gently to its new limit without stretching) the stretch reflex is avoided – a reflex which may be brought about even by passive and non-painful stretch (see Mattes’ views p. 3).

He concludes that this method demonstrates the close connection between tension and pain, and between relaxation and analgesia.

 

The use of eye movements as part of the methodology is based on research by Gaymans (1980) which indicates, for example, that flexion is enhanced by the patient looking downwards, and extension by the patient looking upwards. Similarly, sidebending is facilitated by looking towards the side involved. These ideas are easily proved by self-experiment: an attempt to flex the spine while maintaining the eyes in an upwards (towards the forehead) looking direction will be found to be less successful than an attempt made to flex while looking downwards. These eye-direction aids are also useful in manipulation of the joints.

 

Effects of Muscle energy techniques


Lewit (1999) discusses the element of passive muscular stretch in MET and maintains that this factor does not always seem to be essential. In some areas, self-treatment, using gravity as the resistance factor, is effective, and such cases sometimes involve no element of stretch of the muscles in question. Stretching of muscles during MET, according to Lewit (1999), is only required when contracture due to fibrotic change has occurred, and is not necessary if there is simply a disturbance in function. He quotes results in one series of patients in his own clinic in which 351 painful muscle groups, or muscle attachments, were treated by MET (using postisometric relaxation) in 244 patients. Analgesia was immediately achieved in 330 cases and there was no effect in only 21 cases. These are remarkable results by any standards.

 

Lewit suggests, as do many others, that trigger points and ‘fibrositic’ changes in muscle will often disappear after MET contraction methods. He further suggests that referred local pain points, resulting from problems elsewhere, will also disappear more effectively than where local anaesthesia or needling (acupuncture) methods are employed.

 

Janda’s postfacilitation stretch method


Janda’s variation on this approach (Janda 1993), known as ‘postfacilitation stretch’, uses a different starting position for the contraction and also a far stronger isometric contraction than that suggested by Lewit and most osteopathic users of Muscle energy techniques:

 

  1. The shortened muscle is placed in a mid-range position about halfway between a fully stretched and a fully relaxed state.
  2. The patient contracts the muscle isometrically, using a maximum degree of effort for 5–10 seconds while the effort is resisted completely.
  3. On release of the effort, a rapid stretch is made to a new barrier, without any ‘bounce’, and this is held for at least 10 seconds.
  4. The patient relaxes for approximately 20 seconds and the procedure is repeated between three and five times more.

Some sensations of warmth and weakness may be anticipated for a short while following this more vigorous approach.

 

Reciprocal inhibition variation


This method, which forms a component of PNF methodology (see Box 1.1) and Muscle energy techniques, is mainly used in acute settings, where tissue damage or pain precludes the use of the more usual agonist contraction, and also commonly as an addition to such methods, often to conclude a series of stretches whatever other forms of MET have been used (Evjenth & Hamberg 1984):

  1. The affected muscle is placed in a mid-range position.
  2. The patient is asked to push firmly towards the restriction barrier and the operator either completely resists this effort (isometric) or allows a movement towards it (isotonic). Some degree of rotational or diagonal movement may be incorporated into the procedure.
  3. On ceasing the effort, the patient inhales and exhales fully, at which time the muscle is passively lengthened.

Liebenson notes that ‘a resisted isotonic effort towards the barrier is an excellent way in which to facilitate afferent pathways at the conclusion of treatment with active muscular relaxation techniques or an adjustment (joint). This can help reprogram muscle and joint proprioceptors and thus re-educate movement patterns.’ (See Box 1.2.)

 

Strengthening variation


Another major muscle energy variation is to use what has been called isokinetic contraction (also known as progressive resisted exercise). In this the patient starts with a weak effort but rapidly progresses to a maximal contraction of the affected muscle(s), introducing a degree of resistance to the operator’s effort to put the joint, or area, through a full range of motion. The use of isokinetic contraction is reported to be a most effective method of building strength, and to be superior to high repetition, lower resistance exercises (Blood 1980). It is also felt that a limited range of motion, with good muscle tone, is preferable (to the patient) to having a normal range with limited power. Thus the strengthening of weak musculature in areas of permanent limitation of mobility is seen as an important contribution in which isokinetic contractions may assist.

 

Isokinetic contractions not only strengthen the fibres which are involved, but also have a training effect which enables them to operate in a more coordinated manner. There is often a very rapid increase in strength. Because of neuromuscular recruitment, there is a progressively stronger muscular effort as this method is repeated. Isokinetic contractions, and accompanying mobilisation of the region, should take no more than 4 seconds at each contraction in order to achieve maximum benefit with as little fatiguing as possible, either of the patient or the operator. Prolonged contractions should be avoided. The simplest, safest, and easiest-to-handle use of isokinetic methods involves small joints, such as those in the extremities. Spinal joints may be more difficult to mobilise while muscular resistance is being fully applied.

 

The options available in achieving increased strength via these methods therefore involve a choice between either a partially resisted isotonic contraction, or the overcoming of such a contraction, at the same time as the full range of movement is being introduced (note that both isotonic concentric and eccentric contractions will take place during the isokinetic movement of a joint). Both of these options should involve maximum contraction of the muscles by the patient. Home treatment of such conditions is possible, via self-treatment, as in other MET methods.

Isolytic Muscle Energy Techniques

Another application of the use of isotonic contraction occurs when a direct contraction is resisted and overcome by the operator (Fig. 1.4). This has been termed isolytic contraction, in that it involves the stretching, and sometimes the breaking down, of fibrotic tissue present in the affected muscles. Adhesions of this type are reduced by the application of force by the operator which is just greater than that being exerted by the patient. This procedure can be uncomfortable, and the patient should be advised of this. Limited degrees of effort are therefore called for at the outset of isolytic contractions. This is an isotonic eccentric contraction, in that the origins and insertions of the muscles involved will become further separated, despite the patient’s effort to approximate them. In order to achieve the greatest degree of stretch (in the condition of myofascial fibrosis, for example), it is necessary for the largest number of fibers possible to be involved in the isotonic contraction. Thus there is a contradiction in that, in order to achieve this large involvement, the degree of contraction should be a maximal one, and yet this is likely to produce pain, which is contraindicated. It may also, in many instances, be impossible for the operator to overcome.

 

 

This stretches the muscles which are contracting (TFL shown in example) thereby inducing a degree of controlled microtrauma, with the aim of increasing the elastic potential of shortened or fibrosed tissues.

 

The patient should be instructed to use about 20% of possible strength on the first contraction, which is resisted and overcome by the operator, in a contraction lasting 3–4 seconds. This is then repeated, but with an increased degree of effort on the part of the patient (assuming the first effort was relatively painless). This continuing increase in the amount of force employed in the contracting musculature may be continued until, hopefully, a maximum contraction effort is possible, again to be overcome by the operator. In some muscles, of course, this may require a heroic degree of effort on the part of the operator, and alternative methods are therefore desirable. Deep tissue techniques, such as neuromuscular technique, would seem to offer such an alternative. The isolytic manoeuvre should have as its ultimate aim a fully relaxed muscle, although this will not always be possible.

Why Fibrosis Occurs Naturally

An article in the Journal of the Royal Society of Medicine (Royal Society of Medicine 1983) discusses connective tissue changes:

 

Aging affects the function of connective tissue more obviously than almost any organ system. Collagen fibrils thicken, and the amounts of soluble polymer decrease. The connective tissue cells tend to decline in number, and die off. Cartilages become less elastic, and their complement of proteoglycans changes both quantitatively and qualitatively. The interesting question is how many of these processes are normal, that contribute blindly and automatically, beyond the point at which they are useful? Does prevention of aging, in connective tissues, simply imply inhibition of cross linking in collagen fibrils, and a slight stimulation of the production of chondroitin sulphate proteoglycan?

 

The effects of various soft tissue approaches such as NMT and Muscle energy techniques will impact directly on these tissues as well as on the circulation and drainage of the affected structures, which suggests that the ageing process can be influenced. Destruction of collagen fibrils, however, is a serious matter (for example when using isolytic stretches), and although the fibrous tissue may be replaced in the process of healing, scar-tissue formation is possible, and this makes repair inferior to the original tissues, both in functional and structural terms. An isolytic contraction has the ability to break down tight, shortened tissues and the replacement of these with superior material will depend, to a large extent, on the subsequent use of the area (exercise, etc.), as well as the nutritive status of the individual. Collagen formation is dependent on adequate vitamin C, and a plentiful supply of amino acids such as proline, hydroxyproline and arginine. Manipulation, aimed at the restoration of a degree of normality in connective tissues, should therefore take careful account of nutritional requirements.

 

The range of choices in stretching, irrespective of the form of prelude to this – strong or mild isometric contraction, starting at or short of the barrier – therefore covers the spectrum from all- passive to all-active, with many variables in between.

Putting It Together

Many may prefer to use the variations, as described above, within individual settings. The recommendation of this text, however, is that they should be ‘mixed and matched’ so that elements of all of them may be used in any given setting, as appropriate. Lewit’s (1999) approach seems ideal for more acute and less chronic conditions, while Janda’s (1989) more vigorous methods seem ideal for hardy patients with chronic muscle shortening.

 

Muscle energy techniques offers a spectrum of approaches which range from those involving hardly any active contraction at all, relying on the extreme gentleness of mild isometric contractions induced by breath-holding and eye movements only, all the way to the other extreme of full-blooded, total- strength contractions. Subsequent to isometric contractions – whether strong or mild – there is an equally sensitive range of choices, involving either energetic stretching or very gentle movement to a new restriction. We can see why Sandra Yale (in DiGiovanna 1991) speaks of the usefulness of MET in treating extremely ill patients.

 

Many patients present with a combination of recent dysfunction (acute in terms of time, if not in degree of pain or dysfunction) overlaid on chronic changes which have set the scene for their acute current problems. It seems perfectly appropriate to use methods which will deal gently with hypertonicity, and more vigorous methods which will help to resolve fibrotic change, in the same patient, at the same time, using different variations on the theme of MET. Other variables can be used which focus on joint restriction, or which utilise RI should conditions be too sensitive to allow PIR methods, or variations on Janda’s more vigorous stretch methods (see Box 1.1).

 

Discussion of common errors in application of Muscle energy techniques will help to clarify these thoughts.

Why Muscle Energy Techniques Might Be Ineffective At Times

Poor results from use of Muscle energy techniques may relate to an inability to localize muscular effort sufficiently, since unless local muscle tension is produced in the precise region of the soft tissue dysfunction, the method is likely to fail to achieve its objectives. Also, of course, underlying pathological changes may have taken place, in joints or elsewhere, which make such an approach of short-term value only, since such changes will ensure recurrence of muscular spasms, sometimes almost immediately.

 

MET will be ineffective, or cause irritation, if excessive force is used in either the contraction phase or the stretching phase.

 

The keys to successful application of Muscle energy techniques therefore lie in a precise focusing of muscular activity, with an appropriate degree of effort used in the isometric contraction, for an adequate length of time, followed by a safe movement through the previous restriction barrier, usually with patient assistance.

 

Use of variations such as stretching chronic fibrotic conditions following an isometric contraction and use of the integrated approach (INIT) mentioned earlier in this chapter represent two examples of further adaptations of Lewit’s basic approach which, as described above, is ideal for acute situations of spasm and pain.

To Stretch Or To Strengthen?

Marvin Solit (1963), a former pupil of Ida Rolf, describes a common error in application of Muscle energy techniques – treating the ‘wrong’ muscles the ‘wrong’ way:

 

As one looks at a patient’s protruding abdomen, one might think that the abdominal muscles are weak, and that treatment should be geared towards strengthening them. By palpating the abdomen, however, one would not feel flabby, atonic muscles which would be the evidence of weakness; rather, the muscles are tight, bunched and shortened. This should not be surprising because here is an example of muscle working overtime maintaining body equilibrium. In addition these muscles are supporting the sagging viscera, which normally would be supported by their individual ligaments. As the abdominal muscles are freed and lengthened, there is a general elevation of the rib cage, which in turn elevates the head and neck.

 

Attention to tightening and hardening these supposedly weak muscles via exercise, observes Solit, results in no improvement in posture, and no reduction in the ‘pot-bellied’ appearance. Rather, the effect is to further depress the thoracic structures, since the attachments of the abdominal muscles, superiorly, are largely onto the relatively mobile, and unstable, bones of the rib cage. Shortening these muscles simply achieves a degree of pull on these structures towards the stable pelvic attachments below.

 

The approach to this problem adopted by Rolfers is to free and loosen these overworked and only apparently weakened tissues. This allows for a return to some degree of normality, freeing the tethered thoracic structures, and thus correcting the postural imbalance. Attention to the shortened, tight musculature, which will also be inhibiting their antagonist muscles, should be the primary aim. Exercise is not suitable at the outset, before this primary goal is achieved.

 

The common tendency in some schools of therapy to encourage the strengthening of weakened muscle groups in order to normalise postural and functional problems is also discussed by Vladimir Janda (1978). He expresses the reasons why this approach is ‘putting the cart before the horse’: ‘In pathogenesis, as well as in treatment of muscle imbalance and back problems, tight muscles play a more important, and perhaps even primary, role in comparison to weak muscles’ (Fig. 1.5). He continues with the following observation:

 

Clinical experience, and especially therapeutic results, support the assumption that (according to Sherrington’s law of reciprocal innervation) tight muscles act in an inhibitory way on their antagonists. Therefore, it does not seem reasonable to start with strengthening of the weakened muscles, as most exercise programmes do. It has been clinically proved that it is better to stretch tight muscles first. It is not exceptional that, after stretching of the tight muscles, the strength of the weakened antagonists improves spontaneously, sometimes immediately, sometimes within a few days, without any additional treatment.

 

This sound, well-reasoned, clinical and scientific observation, which directs our attention and efforts towards the stretching and normalizing of those tissues which have shortened and tightened, seems irrefutable, and this theme will be pursued further in Chapter 2.

 

Muscle energy techniques are designed to assist in this endeavor and, as discussed above, also provides an excellent method for assisting in the toning of weak musculature, should this still be required, after the stretching of the shortened antagonists, by use of isotonic methods.

Tendons

Aspects of the physiology of muscles and tendons are worthy of a degree of review, in so far as Muscle energy techniques and its effects are concerned (see also Box 1.5). The tone of muscle is largely the job of the Golgi tendon organs. These detect the load applied to the tendon, via muscular contraction. Reflex effects, in the appropriate muscles, are the result of this information being passed from the Golgi tendon organ back along the cord. The reflex is an inhibitory one, and thus differs from the muscle spindle stretch reflex. Sandler (1983) describes some of the processes involved:

 

When the tension on the muscles, and hence the tendon, becomes extreme, the inhibitory effect from the tendon organ can be so great that there is sudden relaxation of the entire muscle under stretch. This effect is called the lengthening reaction, and is probably a protective reaction to the force which, if unprotected, can tear the tendon from its bony attachments. Since the Golgi tendon organs, unlike the [muscle] spindles, are in series with the muscle fibres, they are stimulated by both passive and active contractions of the muscles.

 

Pointing out that muscles can either contract with constant length and varied tone (isometrically), or with constant tone and varied length (isotonically), he continues: ‘In the same way as the gamma efferent system operates as a feedback to control the length of muscle fibers, the tendon reflex serves as a reflex to control the muscle tone’.

 

 

The relevance of this to soft tissue techniques is explained as follows:

 

In terms of longitudinal soft tissue massage, these organs are very interesting indeed, and it is perhaps the reason why articulation of a joint, passively, to stretch the tendons that pass over the joint, is often as effective in relaxing the soft tissues as direct massage of the muscles themselves. Indeed, in some cases, where the muscle is actively in spasm, and is likely to object to being pummelled directly, articulation, muscle energy techniques, or functional balance techniques, that make use of the tendon organ reflexes, can be most effective.

 

The use of this knowledge in therapy is obvious and Sandler explains part of the effect of massage on muscle: ‘The [muscle] spindle and its reflex connections constitute a feedback device which can operate to maintain constant muscle length, as in posture; if the muscle is stretched the spindle discharges increase, but if the muscle is shortened, without a change in the rate of gamma discharge, then the spindle discharge will decrease, and the muscle will relax.’

 

Sandler believes that massage techniques cause a decrease in the sensitivity of the gamma efferent, and thus increase the length of the muscle fibres rather than a further shortening of them; this produces the desired relaxation of the muscle. Muscle energy techniques provides for the ability to influence both the muscle spindles and also the Golgi tendon organs.

Joints & Muscle Energy Techniques

Bourdillon (1982) tells us that shortening of muscle seems to be a self-perpetuating phenomenon which results from an over-reaction of the gamma-neuron system. It seems that the muscle is incapable of returning to a normal resting length as long as this continues. While the effective length of the muscle is thus shortened, it is nevertheless capable of shortening further. The pain factor seems related to the muscle’s inability thereafter to be restored to its anatomically desirable length. The conclusion is that much joint restriction is a result of muscular tightness and shortening. The opposite may also apply where damage to the soft or hard tissues of a joint is a factor. In such cases the periarticular and osteophytic changes, all too apparent in degenerative conditions, are the major limiting factor in joint restrictions. In both situations, however, Muscle energy techniques may be useful, although more useful where muscle shortening is the primary factor.

 

The restriction which takes place as a result of tight, shortened muscles is usually accompanied by some degree of lengthening and weakening of the antagonists. A wide variety of possible permutations exists in any given condition involving muscular shortening which may be initiating, or be secondary to, joint dysfunction combined with weakness of antagonists. A combination of isometric and isotonic methods can effectively be employed to lengthen and stretch the shortened groups, and to strengthen and shorten the weak, overlong muscles.

 

Paul Williams (1965) stated a basic truth which is often neglected by the professions which deal with musculoskeletal dysfunction:

 

The health of any joint is dependent upon a balance in the strength of its opposing muscles. If for any reason a flexor group loses part, or all of its function, its opposing tensor group will draw the joint into a hyperextended position, with abnormal stress on the joint margins. This situation exists in the lumbar spine of modern man.

 

Lack of attention to the muscular component of joints in general, and spinal joints in particular, results in frequent inappropriate treatment of the joints thus affected. Correct understanding of the role of the supporting musculature would frequently lead to normalization of these tissues, without the need for heroic manipulative efforts. Muscle energy techniques and other soft tissue approaches focus attention on these structures and offer the opportunity to correct both the weakened musculature and the shortened, often fibrotic, antagonists.

 

More recently, Norris (1999) has pointed out that:

 

The mixture of tightness and weakness seen in the muscle imbalance process alters body segment alignment and changes the equilibrium point of a joint. Normally the equal resting tone of the agonist and antagonist muscles allows the joint to take up a balanced position where the joint surfaces are evenly loaded and the inert tissues of the joint are not excessively stressed. However if the muscles on one side of a joint are tight and the opposing muscles relax, the joint will be pulled out of alignment towards the tight muscle(s).

 

Such alignment changes produce weight-bearing stresses on joint surfaces, and result also in shortened soft tissues chronically contracting over time. Additionally such imbalances result in reduced segmental control with chain reactions of compensation emerging (see Ch. 2).

 

Several studies will be detailed (Chs 5 and 8) showing the effectiveness of Muscle energy techniques application in diverse population groups, including a Polish study on the benefits of Muscle energy techniques in joints damaged by haemophilia, and a Swedish study on the effects of Muscle energy techniques in treating lumbar spine dysfunction, as well as an American/Czech study involving myofascial pain problems. In the main, the results indicate a universal role in providing resolution or relief of such problems by means of the application of safe and effective muscle energy techniques.

 

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Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. El Paso, TX. Muscle energy techniques, manipulative therapy involves a movement away from high velocity/low amplitude thrusts (HVT – known as ‘mobilization with impulse’ and characteristic of most chiropractic manipulation) towards gentler methods. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

No comment yet.
Scooped by Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP
December 18, 2017 9:35 PM
Scoop.it!

4 Ways Chiropractic Can Help Carpal Tunnel Syndrome Sufferers

4 Ways Chiropractic Can Help Carpal Tunnel Syndrome Sufferers | Mobility & Flexibility - Joint Movement | Scoop.it

Carpal tunnel syndrome (CTS) is a serious, painful nerve injury that affects many people in the United States. CTS occurs when the median nerve, which runs down a person's forearm to his or her hand, gets compressed in the eight bones in the wrist called the carpal tunnel. This injury is frequently caused over time by repetitive motion such as assembly line work, and is the most expensive work-related injury. Symptoms of CTS include pain and numbness of the hand and wrist. While there are a variety of treatment options available to those who are afflicted with carpal tunnel syndrome up to and including surgery, chiropractic care has become a popular and effective option.

 

Here are four ways patients with carpal tunnel syndrome benefit from chiropractic care.

Chiropractic Care Is Documented To Work On Carpal Tunnel Syndrome.

While there are no guarantees that any one mode of treatment will work on every person, two studies have shown strong results that back the effectiveness of chiropractic treatment on CTS.

 

In both cases, the majority of participants showed significant improvement in several measures such as range of motion, finger sensation, and pain reduction. These studies provide evidence to people suffering from carpal tunnel syndrome. Patients can feel confident in improving their symptoms when choosing to pursue a chiropractor's care.

Chiropractors Provide A Less Invasive Treatment Option.

Carpal tunnel syndrome, particularly cases that have gone undiagnosed or treated for an extended period of time, that ends up being too painful and advanced to be handled with medicine may face surgery. However, chiropractic care often helps minimize that option as a last resort. Regular visits to a chiropractor can show positive results in the controlling and healing of many of the CTS symptoms.

Chiropractic Care Offers An Alternative To Drugs To Manage Pain

As with surgery, daily doses of medicine may be a less than ideal plan when managing carpal tunnel syndrome. Certain patients may suffer from issues with the medicine, CTS medicines may conflict with other medication, or they may simply not want to take daily medications.

 

If medicinal treatment is not an attractive option, a chiropractor appointment is the next logical step. Chiropractic care often helps CTS by adjusting the patient's elbow and spine.

 

Another common treatment is bracing. This technique limits the hand's movements with the goal of allowing the wrist and tendons to heal and recover.

 

A chiropractor who is experienced with carpal tunnel syndrome is able to review each case individually and make solid treatment recommendations that can alleviate the patient's dependence on managing the pain with drugs.

Allows Patients To Learn How To Manage Carpal Tunnel Syndrome.

Unfortunately, health problems that produce chronic pain can take a toll on the patient not only physically, but psychologically. Dealing with CTS can make a person feel powerless over their own body. Working with a chiropractor to relieve, control, and heal the symptoms of carpal tunnel syndrome empowers the patient to be able to feel ownership of managing and improving his or her health.

 

Workers who perform repetitive tasks daily as part of their job need to be especially aware of the symptoms of carpal tunnel syndrome. Feeling pain, numbness, tingling, or burning in their palms or fingers may be the first symptoms of CTS. The earlier it's diagnosed, the more effective less-invasive treatment will be.

 

Of all the carpal tunnel syndrome treatment choices, chiropractic care offers the dual benefits of being a highly effective treatment while still employing tactics that are not as invasive as other options. If you or a loved one are suffering from this condition, give us a call. We’re here to help!

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Carpal tunnel syndrome (CTS) is a serious, painful nerve injury that affects many people in the United States. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

good health's curator insight, January 15, 2024 9:02 AM

Acquista Online La Prescrizione Di Perdita Di Peso
Crediamo che i farmaci a volte possano essere molto urgenti da assumere. Se hai urgente bisogno di farmaci, possiamo anche fornirti una consegna espressa,


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Stretching: What Chiropractic Patients Should Know | El Paso Back Clinic®

Stretching: What Chiropractic Patients Should Know | El Paso Back Clinic® | Mobility & Flexibility - Joint Movement | Scoop.it

When you suffer from joint or muscle pain, it is important to work on maintaining as much flexibility as possible. The more flexible you are, the less likely you will be to further injure yourself. One of the best ways to improve your flexibility is by stretching before you are active.

 

However, you need to warm up your muscles before you stretch. If you stretch first, you can actually injure yourself by pushing your joints too far. Spend a few minutes doing some light activity before you stretch. This can be as simple as a brisk walk or some basic calisthenics.

 

If you have been seeing a chiropractor, he or she may be able to recommend some stretches for you. Otherwise, you can use some of these basic techniques. There are two basic forms of stretching, static and dynamic.

Static Stretching vs. Dynamic Stretching

Static stretches involve holding a position for a certain period of time to loosen up your muscles. These tend to be what most people think of when they think about stretching. However, dynamic stretches are also important. With these, you move parts of your body to work on your flexibility.

 

Many of the most effective stretches for back pain can be done right at home. For example, lie on your back with your knees bent. Grasp one knee in both hands and pull it up towards your chest. Hold this position for 30 seconds and then lower the knee to the starting pose. Repeat with the other knee. You can also do both knees at the same time.

 

A similar stretch begins in the same position as the previous one. However, instead of lifting your knee to your chest, roll both legs to one side so that your knees are as close to the floor as possible. Hold this position for 10 seconds, and then roll to the other side.

 

Another common stretch recommended by many chiropractors comes from yoga, where it is known as the "cat pose." Get down on the floor on your hands and knees, with your hands underneath your shoulders. First, let your abdomen drop down towards the floor. Then, reverse this movement by arching your back. Repeat this cycle three to five times.

 

More dynamic stretches can also be good for your muscle pain. Try doing hand walks to stretch your shoulders and abdominal muscles. Stand up straight and slowly lower your hands towards the floor. Walk your hands out in front of you until you are as far down as you can go. Then walk your hands back to the starting position.

 

A final stretch that can help your back muscles is known as the "scorpion." Lie face-down and stretch your arms out to the sides. First, slowly move your right foot towards your left arm. Then, move your left foot towards your right arm. Make sure to move in a slow and controlled fashion.

 

When you are suffering from muscle or joint pain, it is a good idea to stretch both in the morning and the evening. By incorporating these exercises into your daily routine, you can avoid many common injuries.

 

If you need further instruction regarding stretches, please give us a call so that you can schedule an appointment with our Doctor of Chiropractic.

 

This article is copyrighted by Blogging Chiros LLC for its Doctor of Chiropractic members and may not be copied or duplicated in any manner including printed or electronic media, regardless of whether for a fee or gratis without the prior written permission of Blogging Chiros, LLC.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Stretching, If you suffer from joint or muscle pain, it is important to work on maintaining as much flexibility as possible. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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Upper Trapezius: The Key To Optimal Function Of The Shoulder | El Paso Back Clinic® • 915-850-0900

Upper Trapezius: The Key To Optimal Function Of The Shoulder | El Paso Back Clinic® • 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it


Understanding The Upper Trapezius


Doctor of Chiropractic, Dr. Alex Jimenez explores the relevant anatomy and function of the UT, the role it has in musculoskeletal dysfunction and management ideas in the form of strengthening and loosening.


The upper trapezius (UT) is a major muscle that affects movement and stability of both the scapula and cervical spine. The coordination and interplay between the UT and other scapula muscles such as the lower and middle trapezius, serratus anterior, rhomboids, levator scapulae and pectoralis minor, have a large bearing on how the scapula moves and how stable it can become to allow proper gleno-humeral motion. With this in mind, dysfunction in the UT, either as weakness or hypertonicity, has been implicated in a host of shoulder pathologies and cervical spine syndromes.


Anatomy & Biomechanics


The gross anatomy and function of the UT is comprehensively discussed in a landmark study from the 1990’s(1). Johnson and Bogduk (1994) dissected the individual fascicular anatomy of the UT from the superior nuchal line near the occiput, all the way down to the C7 vertebrae, and also the thoracic portion of the
UT. What they found was that the UT was a multipennate muscle that had no osseous attachments between the occiput to the C7 spinous process. Instead the UT originated from the fascial structure known as the ‘ligamentum nuchae’.


The upper cervical fibres from C1 to C6 passed in a downward direction, and traversed to insert into the clavicle. Fibres from C7 and T1 passed horizontally to reach the acromion and spine of the scapula. Its thoracic fibres converged to the deltoid tubercle of the scapula (see Figure 1).


Figure 1: Anatomy of the upper trapezius

Note the thicker C7 and T1 fibres
The fascicle length of the UT fibres measured around 1cm from the occiput to C3, and from C3 to T12 the fibres were around 7-14cm. These fibres are primarily low-threshold type I fibres, suggesting that their role is not to produce power but instead act as a controller and stabiliser of the scapula.


The findings from the Johnson and Bogduk dissectionstudy paved the way for a different interpretation on the function of the UT in scapula movement. As only the upper fibres showed a vertical orientation, this suggested they may have only a slight role in scapula elevation. These fibres are relatively small compared to the lower fibres; volumetric studies have demonstrated that the fibres from C7, T1, and the lower half of ligamentum nuchae are the largest.


Johnson and Bodguk also found that the UT muscle has an angle and orientation, which are too small to be a significant elevator of the scapula when the arm is down by the side. They act primarily as an upward rotator of the scapula when co-ordinated with the lower and middle trapezius fibres.


They also work in a force couple along with serratus anterior to create upward rotation. Without the contribution from the lower trapezius and serratus anterior, the UT cannot create upward rotation in isolation. It needs to work in concert with the other upward rotators.


The mechanism of this force couple is via the action of the serratus anterior, which pulls the scapula into protraction. The lower trapezius contracts isometrically to fixate the middle medial border of the scapula so that the serratus anterior can then upwardly rotate the scapula.


UT then contributes towards upward rotation as the scapula has already begun upward rotation. The UT also elevates the scapula at the top of the shoulder flexion/abduction movement (see Figure 2).


Figure 2: Force couple of UT with middle/lower trapezius and serratus Anterior

Furthermore, the majority of the UT fibres attach onto the distal third of the clavicle, and due to their horizontal arrangement, they also rotate the clavicle medially – this rotation compresses and stabilises the sternoclavicular (SC) joint.


This SC joint compression allows load to be transferred away from the cervical spine and instead is directed towards the sternum and axial skeleton.


Johnson et al argue that the increased EMG activity of the UT does not occur to elevate the scapula but instead exists to draw the clavicle medially and upward at the same time to compress the SC joint(1). Therefore, the UT creates elevation by exerting an upward moment on the clavicle and increases the
compression loads at the sternoclavicular joint. This means that the cervical spine is not taking the load of the UT during shoulder elevation but the SC joint is.


The diagram below shows the angle of orientation of the fascicles, based on the work by Johnston and Boduk(1). A radiograph shows the direction of fibres. In this image the size and cross sectional area of the fibres are also shown based on the thickness of the lines. It can be seen that the fascicles of the lower half of the UT are much larger than the upper fibres.

 

The largest fibres are the transverse C6 and C7 fascicles.
In summary, the UT works synergistically with the other trapezius muscles (middle and lower) to produce a force couple on the scapula; therefore the UT has both a functional role in movement and stabilisation of the scapula. To highlight the individual and synergistic role of the trapezius muscle, the individual heads work in the following ways;


Figure 3: Orientation of fascicles(1)

UT draws the clavicle backwards, medially and upwards at the sternoclavicular joint. It helps to control the neutral positionof the scapula. It has some potential to produce movement to the cervical spine and contributes to the stability of the cervical spine.


Middle trapezius produces scapula retractionand upward rotation, as well as drawing the clavicle and scapula backwards and medially together with UT allowing scapula upward rotation. It controls or resists scapula protraction, and downward rotation.
Lower trapezius produces scapula upward rotation, external rotation, posterior tilt and depression. It controls or resists scapula elevation, abduction and downward rotation, as well as controlling the neutral position of the scapula.


Finally, the UT working with other cervical muscles can also laterally flex the cervical spine to the same side, rotate the cervical spine contralaterally and extend the cervical spine.


Dysfunction Of The Upper Trapezius


The UT has an important role to play in both scapula position and stability. This in turn will influence the gleno-humeral joint in positions of shoulder elevation, since scapula mobility has been linked with shoulder range of movement(2,3). During arm elevation, the scapula moves toward internal or external rotation, upward rotation, and posterior tilt. These actions are influenced by the upper/middle/lower trapezius and the serratus anterior as a force couple (mentioned above)(4). Lack of control of these muscles will break down the optimal scapula position and stabilisation/orientation of the shoulder joint. This scapula imbalance has been linked to shoulder pain(5). It must not be forgotten that other factors such as thoracic spine posture, capsule tightness in the shoulder and tightness in pectoralis major/ minor and latissimus dorsi may also influence scapula position(6,7).


Figure 4: Classic UT trigger points (from Travel and Simons(21))

Turgot et al (2016) studied the isometric strength of the three heads of the trapezius and linked it to electromagnetic assessment of scapula mechanics(8). They found that shoulders with stronger UT muscles showed greater upward scapular rotation at 30°, 60°, 90°, and 120° of elevation in the frontal plane. Shoulders with stronger middle trapezius had greater scapular upward rotation at 90° of elevation in the frontal plane. Shoulders with stronger lower trapezius showed greater scapular posterior tilt at 90° of elevation in the sagittal plane.
Numerous studies have identified an imbalance in activation between the UT and lower trapezius in patients with painful shoulders. Specifically, the imbalance identifies overactivity of the UT and underactivity in the lower trapezius(9-13). One study found that using a novel taping technique to inhibit the UT, reduced activation patterns of the UT in patients with shoulder pain(14). This aspect will be explained in detail later in the article.


Peat and Grahame investigated trapezius, serratus anterior and deltoid EMG in people with and without shoulder pathology(15). They found that in those with shoulder pathology, UT showed increased activity during arm elevation and lowering (between 40 to 100 degrees of arm elevation). Also, serratus anterior showed decreased activity at some humeral elevation angles (between 70 to 100 degrees) compared to healthy controls. This increase in UT activity has been found in numerous other studies on UT activation and shoulder pathology(9-13,15,16).


This finding may be associated with the increase in clavicular elevation or scapular elevation found in several clinical and kinematic studies(17).


The increased UT activation may be viewed as a common compensatory strategy used by people with shoulder pain and pathology to elevate their arm. However, the subsequent increase in clavicle elevation at the SC joint that may be produced by increased UT activity will result in scapular anterior tilt. This offsets the scapular posterior tilt produced at the AC joint by the lower trapezius, reducing the overall posterior tilt of the scapula on the thorax, which may be viewed as a potential mechanism to either cause or aggravate impingement symptoms.


Other authors have suggested that excess UT activity may attempt to compensate for a weak serratus anterior, and is believed to contribute to impingement through abnormal rotation
of the scapula(13). Evidence of increased UT activation combined with reduced SA activation (as well as evidence of increased superior translation of the scapula) has been demonstrated in persons with shoulder pain(13,15,18). Imbalances of force production of the serratus anterior and UT can result in a scapula elevation motion (or early shoulder shrugging).

 

This may cause excess superior translation of the scapula, with less efficient upward rotation and reduced posterior tilting. Clinical consequences of these alterations can include subacromial impingement, subacromial bursitis, and rotator cuff or biceps tendinitis, which can progress to rotator cuff tears. There may also be altered acromioclavicular joint forces and possible predisposition to degenerative changes(19).
In a study by Leong et al (2016) (20) it was shown that athletes with rotator cuff tendinopathy exhibited higher UT shear modulus during active arm holding than the asymptomatic athletes(20) (higher shear modulus suggests an increase in active and/or passive muscle tension measured on a specialised ultrasound). UT shear modulus was also higher in athletes with rotator cuff tendinopathy than in the asymptomatic athletes during the resting arm position at 0°of shoulder abduction.


The findings from such a study cannot however be used to determine whether the increased tension in UT is a result of rotator cuff tendinopathy or if it is a cause of the problem. It is clinically appreciated that athletes with painful rotator cuff may have a scapula at rest that appears downwardly rotated and anterior tilted, and often this is attributed to an overactive pectoralis minor. This position would place extra ‘stretch’ on the UT thus creating a situation whereby the UT had to counterbalance the drag effect of the scapula position.


Upper Trapezius Myofascial Trigger Points
 
Figure 5: UT shrug exercise (note the wide hand placement)

1. Wide Grip Shrug


In a study performed in Brazil, researchers attempted to correlate the anatomy of the trapezius and the accessory nerve entry points into the muscle with clinical locations of myofascial trigger points(21,22). What they found was that of the classic seven clinical points of myofascial trigger points, four correlated well to the actual anatomy of the accessory nerve and the trapezius muscle. These seven points with their characteristic referral patterns are shown in Figure 4.


Upper Trapezius, Neck Pain, Headaches & Nerve Pain
It was suggested in a study by Australian researchers that tightness in the UT is correlated to limited neural extensibility in the brachial plexus(23). Although it is difficult to prove causation, the suggestion is made that those with sensitised neural tissue may have reactive muscle tone in the UT. The weight of the arm at rest would provide a reasonable traction force to the brachial plexus, and the UT may react to protect the brachial plexus by increasing tone and lifting the scapula in order to reduce the traction effect on the nerves.
Figure 6: Single-arm overhead shrug start position (note angle of abduction)

2. Single-arm overhead shrug


What is also interesting are the morphological changes in the UT in the presence of neck pain and dysfunction.
The muscle fibre type changes from type I to type II, with fatty infiltration and higher percentage of grossly hypertrophied type I muscle mega-fibres, along with poor capillarisation(24-26). Finally, the famous ‘Janda’s Upper Crossed Syndrome’ has been implicated as a causative factor in cervicogenic headaches and patients; this condition is often present with tightness of the sternocleidomastoid, UT, levator, scalenes, suboccipitals, pectoralis minor, and pectoralis major(27-30).


Management Of Trapezius Dysfunction


*Strengthening

A group of Australian researchers investigated the use of a standard shoulder shrug (scapular elevation) at 0 degrees compared to a modified shrug performed with the arm elevated to 30 degrees (with the scapula in slight upward rotation). They found that the arm-elevated position elicited greater activation in the lower and upper trapezius. Therefore, traditional shrug exercises are not as effective as the 30-degree abduction position(31). Below are shown some exercises that may be used to retrain the UT in the presence of weakness and dysfunction.
Hold a barbell with the hand placed as far away as comfortably possible. This will place the arm in some abduction and the scapula in upward rotation. Slowly elevate the scapula towards the ear. Perform three sets of 20 slow repetitions with a hold. This is a great starter exercise for a patient with shoulder pathology because the arm is in relative neutral abduction.
Holding a dumbbell above the head and the body in a side sit position (this position opens up the shoulder and avoids impingement of the shoulder). Slowly elevate the scapula and rotate the dumbbell from internal to external rotation.
Perform three sets of 20 reps.


Hold two dumbbells overhead with the arm fully elevated (you need good shoulders for this). This is the same process as exercise 2 above but the exercise is performed bilaterally, and with more abduction of the shoulder.


3. Monkey Shrug

*Loosening


1. Upper trapezius stretch


The action of the UT is to upwardly rotate and retract the scapula; its role in elevation is questionable. The effect on the cervical spine is to laterally flex towards the same side, rotate away and finally to extend the cervical spine. Therefore, the best stretch position is one that encourages a combination of:


2. Upper trapezius trigger points


Scapula downward rotation, protraction, depression
Cervical contralateral flexion, flexion and ipsilateral rotation.
Above is an example of stretch to target the UT in a stretch and also ball placement for trigger point releases.


Hold the arm behind the back. This will create downward rotation of the scapula. Protract the scapula and depress the scapula actively. Pull the head to the opposite side and look over the same shoulder. Hold for ten seconds and repeat five times.


Place a ball (tennis, spikey, trigger ball) into any of the seven mentioned trigger points. Hold against the trigger for 30 seconds, move off and find another spot.


Conclusion


The UT is an important muscle of the scapula, which has a major role in scapulohumeral rhythm. Its key roles are scapula upward rotation, elevation and retraction. Its role as a scapula elevator is not as important. It is a muscle that may become either tight and hypertonic as a compensatory mechanism for other scapula imbalances, or it may become weak as it is dragged into a stretch position by poor scapula posture. Exercises have been presented that are designed to either strengthen the muscle or to release tightness and hypertonicity.
 
References
1. Clin Biomech (Bristol, Avon). ;9(1): 1994. 44-50
2. Am J Sports Med, 1998, 26: 325–337
3. Journal of the American Academy of Orthopaedic Surgeons. 2003, 11, 142–15
4. Clin Orthop Relat Res, 1996, (330): 3–12
5. Orthop Clin North Am, 2000, 31: 285–293
6. J Orthop Sports Phys Ther, 2009, 39: 90–104
7. J Phys Ther Sci, 2015, 27: 1739–1742
8. J. Phys. Ther. Sci. 2016. 28: 1864–1867
9. The Physician and Sportsmedicine, 2003. 31(7), 25–32
10. Mottram, S. L. (1997). Dynamic stability of the scapula. Manual Therapy, 2,123–131
11. Journal of Orthopaedic and Sports Physical Therapy, 1999. 29, 31–38
12. Am. J. Sports Med. 2003. 31; 542-549
13. Physical Therapy Mar. 2000. 80(3):276-291
14. Phys Ther Sport 10 (2), 45-50. 2009
15. Am J Phys Med 1977;56(5):223–40
16. J Electromyogr Kinesiol 2005;15(6): 576–86
17. Phys Ther 2006;86(8): 1075–90
18. J Orthop Sports Phys Ther. 1999. 29: 574–586, 1999
19. American Journal of Sports Med. 2004. 32(2); 484-493
20. Leong et al (2016) Increased Upper Trapezius Muscle Stiffness in Overhead Athletes with Rotator Cuff Tendinopathy. PLoSONE 11(5)
21. Travel and Simons (1999) Myofascial pain and dysfunction. Volume 1. Upper half of the body. Second edition. Williams and Wilkins. Baltimore.
22. Akamatsu et al (2015) Biomed Research International 2015.
23. Australian Journal of Physiotherapy. 1994. 40(2); 99-103
24. Manual Therapy. 2008. 13: 258–265
25. Spine. 2004. Vol 29(13) 1 July:1436-1440
26. Journal of Electromyography and Kinesiology. 2008. 18:255–261
27. Cephalalgia, 1994. 14(4): p. 273-9
28. Cephalalgia, 1999. 19(3): p. 179-85
29. Man Ther, 2006. 11(2): p. 118-29
30. The International Journal of Sports Physical Therapy. 2011. 6(3); 254-266
31. Clinical Biomechanics. 2014. 29(2): 201-205

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

The upper trapezius (UT) is a major muscle that affects movement and stability of both the scapula and cervical spine. 

For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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Scooped by Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP
April 26, 2017 4:42 PM
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Walking Increases Blood Supply to the Brain

Walking Increases Blood Supply to the Brain | Mobility & Flexibility - Joint Movement | Scoop.it

Just put one foot in front of the other and you’ll boost your brain at the same time.


That’s the conclusion of a small study that found the impact of a foot while walking sends pressure waves through the arteries that increases blood supply to the brain. “New data now strongly suggest that brain blood flow is very dynamic,” said researcher Ernest Greene and his colleagues at New Mexico Highlands University.


Activities such as bicycling, walking and running may optimize brain function and overall sense of well-being during exercise, the researchers said.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

Engaging in regular physical activity, or simply walking or running on a daily basis, can help maintain overall health and wellness. New research studies suggest that the impact of a foot while performing exercise can actually increase circulation to the brain. For more information, please feel free to ask Dr. Jimenez or contact us at (915) 850-0900.

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April 19, 2017 5:42 PM
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Yoga For You & Back Pain | El Paso Back Clinic® • 915-850-0900

Yoga For You & Back Pain | El Paso Back Clinic® • 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it


El Paso, TX. Chiropractor Dr. Alex Jimenez looks at yoga for back pain.


Yoga increases strength and flexibility, but some find it to be a spiritual experience that brings serenity and delight.


I attended my first yoga class after I was 21 years old. At the time the goal was purely to appease my self so that I really could tell my family, buddies and coworkers that “I do yoga”. In my experience, yogis were “cool” and I enjoyed being linked with that healthy lifestyle. My twenty-something year old mind was in a self absorbed place and that I totally enjoyed the freedom of dedicating myself to yoga and other enjoyable ways to fill my time.


Yoga in its purest form is intended to help the individual’s head and body and is likely to be practiced without ego.


My First Yoga Course


I loved her gentle encouragement. As I visited my weekly yoga classes, bending and twisting and telling myself “I can perform this,” I began to reap the benefits. I was sleeping better. My body felt less angry and I noticed a calmness come over me. I felt more patient coping with the irritations of life, too.


Here is The Irony


Yoga is not something you do. It’s a thing that you encounter. Because over time, it gets engrained in you it’s called a practice. The teachings of Maureen were put in me like little seeds which didn’t actually completely thrive until much later in my life. To jogging, though I did not intentionally give up my usual yoga practice, around along the way it took a back seat. Those little seeds were there but lay dormant for now.


Running Was Different


I felt free moving swiftly across the road. Being goal oriented, I found monitoring my mileage to be a pleasing achievement. Running was pleasing for another reason, too. My best friend Linda was also a runner, and we’d meet most Sundays for long runs. We’d participated in occasional half marathons and put in 15 to 20 miles each week. Running that distance took time — two hours or even more.


Those small seeds were there all along. Like I mentioned, yoga has a way of becoming engrained in you.


My Back Pain & Yoga


If you’ve been following my site, you understand that I’ve had back pain through most of my entire life. Then, seemingly out of nowhere, my little friend “yoga” softly arrived back on the scene. My pain riddled body seemed to intuitively understand it needed yoga again. I began feeling the requirement to roll over on my back and pull my legs towards my chest, before getting out of bed in the early hours. (The knees-to-chest pose extends your lower back muscles and is a gentle, soothing approach to begin your day.) The reach that was straightforward felt energizing and really good.


There are quite a lot of advantages of yoga, especially for maintaining well-being and managing low back pain. A follow up experiment was conducted after 26 weeks, and those same yoga participants were experiencing increased function and less pain. For me, the continual practice of yoga has relieved my pain to the point that I no more need pain medication to get me.


Other Ways Yoga Relieves Back Pain

 

  • Yoga strengthens abdominal muscles both of which help support the spinal column and builds flexibility in the rear, when practiced regularly.
  • Holding yoga models, for up to a minute, helps stretch the muscles over time.
  • Properly stretching the muscles in the low back reduces stress over the area.
  • Yoga offers relief from pain, stress and anxiety. All low back pain suffers know this is a vicious cycle. You start with pain that doesn’t go away. You find out there is an anatomical reason for the pain. But, you still have the anxiety, and stress of worrying about how long this may last. Yoga can counter that triple threat.
  • Yoga improves posture. To maintain a strong, fit, flexible backbone great posture is vital. Seated and standing yoga poses help improve the alignment of the spine and also posture. Proper bearing reduces back pain and removes some of the pressure from your spinal column.


Yoga For Beginners


Thus, let’s get started with a couple of poses which are a cinch to do and great for preserving flexibility in your spine.Try these three poses daily for increased flexibility and your spine will thank you!


Knees to Chest Pose

 

  • Lie on your back with legs and arms stretched
  • Bring both knees to chest as you exhale. Clasp your hands around legs
  • Back is flat on the floor (mat)
  • If it’s comfortable for you, gently rock back and forth, which gives you a little massage


Cat/Cow Pose 

 

  • Begin on all fours in a tabletop position
  • Place your hands under your shoulders along with your knees under your hips
  • Like a cat, round your back up to arch on the exhale
  • Bring your chin to chest
  • On the inhale, drop your abdomen and raise your head, extending your sitting bones (sits) back up


Child’s Pose

 

  • Move from table top to a kneeling position. Rest your arms by your side, press your shoulders down and simultaneously reach your head tall
  • Slowly lower your buttocks towards your heels feeling a nice stretch in your lower back/hips.
  • Let your forehead rest on the floor
  • Place arms resting alongside your body
  • Or you can place arms above head, gently stretching as they are placed on the floor
  • If it is easier too, you can widen knees as you stretch out


Those small yoga seeds—put by my first teacher long past — have continued to grow/flourish. I’m a fully certified yoga teacher and revel in sharing my practice with students every week now. May you find peace on your own journey and pain relief too, one pose at a time. Namaste.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

There are quite a lot of advantages of yoga, especially for maintaining well-being and managing low back pain. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

jack henry's curator insight, April 2, 2024 6:25 AM


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April 5, 2017 9:36 PM
Scoop.it!

Flexibility Training Tips Call 915-850-0900

Flexibility Training Tips Call 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it



There are 3 primary parts of exercise: cardiovascular exercise, strengthening exercises, and flexibility training. And let’s face it—those first 2 typically get more emphasis. Cardiovascular exercise (running, for example—anything which gets your pulse up) and strength training (lifting weights) come with some rather immediate outcomes. They help us build muscle and lose weight , all while helping us be more fit. It takes longer to see those advantages.
But here’s the deal: flexibility becomes more significant as you grow old. Being limber can help battle those aches and pains related to aging; stretching can help you maintain better joint health. It can also make those daily jobs—carrying groceries, going up and down stairs etc. much easier.

However, you can’t wake up when you’re 64 and unexpectedly be equally as adaptable as you were when you were 24. It’s much better and even more efficient to work flexibility training into your workout routine throughout your own life.

(Rest assured: if you are 64 and were hoping to regain some of that youthful flexibility, you can start working it in your workout routine now. Simply be realistic concerning the outcomes. You will, most likely, never be as flexible as you once were, but working on flexibility at any age is rewarding.)

TALK TO YOUR PHYSICIAN OR PHYSICAL THERAPIST BEFORE BEGINNING A NEW WORKOUT PLAN

They could help you establish realistic targets and create a plan that best suits your life. You may want to think about working with a personal trainer to assist you ease into the brand new routine.

FLEXIBILITY TRAINING IS MORE THAN DOING A FEW STRETCHES

After a run is yes, better than nothing, doing a couple of hamstring stretches, but you won’t find as many long-term gains as you’d see from a flexibility plan that is more developed.

To get the most benefit from flexibility training, you should have a personalized program, one that takes into account your body and demands. As stated earlier, a personal trainer or physical therapist is able to help you develop the best plan for you.

And remember: the more time and attention you give to flexibility training, the more gains you’ll see—especially those long term gains.

TAKE YOUR ACTIONS INTO ACCOUNT

Think, also, about your daily life: does your job involve a lot of sitting or lifting?

A personalized flexibility training program is able to help you enhance your freedom (how well your joints move) and stability (keeping good posture and body alignment during actions in order for your body isn’t under undue strain). It can allow you to excel in sports or your activities, in addition to help you take good attention to your body on a daily basis.

GIVE SPECIAL FOCUS TO MUSCLES THAT FEEL TIGHT

The shoulders, chest, hamstrings, and hips are often tight, but you may find tightness in other regions depending on harms, pressure in your lifetime, or how rough a particular workout was. By tailoring your flexibility training to your body, you’ll prevent overstretching muscles—or muscles that are lost that need consideration.

YOUR BODY KNOWS WHAT’S BEST FOR IT

Listen to your body, and don’t push it too much when you’re stretching. Instead, ease into a stretch when you’ve reached the limit of what you are able to do at that point, and understand.

Also, you need to prevent ballistic stretches—that sort of extending where you bounce in and out of the stretch. That strategy isn’t as successful holding the stretch for about 10 to 30 seconds and then as slowly stretching your muscles.

YOU CAN BE CREATIVE WITH STRETCHING

Within the plan that was developed for you, you can use resistance balls, towels, or other props that’ll allow you to go deeper in your stretches. Assortment will also make you more likely to stay with your flexibility training plan.

WARM UP FOR STRETCHING

You may be a bit confused—isn’t stretching a warmup? How do you warm up for stretching? This is where a brisk walk or short jog can help: get your heart pumping and your muscles limber before stretching.

TAKE A FLEXIBILITY COURSE IN THE FITNESS CENTER

Assess your gym’s class program; it may be that they have a few flexibility or stretching classes. Sometimes these courses combine cardiovascular work, strength training, and flexibility work—all 3 parts of exercise in one class! Or you may take a class that’s exclusively focused on stretching.

YOUR MIND CAN STRETCH

Pilates and yoga are outstanding flexibility training trainings. Plus, they teach you about relaxation, meditation, and other head-body techniques—ways to help calm your body and emotions, which can, subsequently, make your body more receptive to being stretched.

STRETCHING IS SIGNIFICANT FOR EVERYONE

Maybe you’ve got this bogus organization with extending—that only individuals in rehabilitation do it or that it’s only for individuals who aren’t actually in shape (that’s: it’sn’t “real” exercise). Well, it’s time to go past that misconception. Everyone should stretch. Look for inspiration or proof at Olympic and professional athletes: they know that flexibility training is a key section of peak performance.

YOU MUST BE CONSISTENT

It needs to be part of your routine, for stretching to be as effective as possible. This isn’t something which you do for a few weeks and after that move on. Regular stretching and flexibility work—along with cardiovascular exercise and strength training —will assist you to take good care of your own body for years to come.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

As you grow older, flexibility becomes more important. Being limber can help combat those aches and pains associated with aging. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900 

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April 4, 2017 1:40 PM
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Exercise Helps Reverse Cellular Aging Process in Adults

Exercise Helps Reverse Cellular Aging Process in Adults | Mobility & Flexibility - Joint Movement | Scoop.it

High-intensity exercise may help older adults reverse certain aspects of the “cellular” aging process, a new study suggests.


It’s no secret that regular exercise is healthy for young and old alike. But researchers said the new findings point to particular benefits from “high-intensity interval training” for older adults. That’s the type of workout that combines brief bursts of vigorous exercise with periods of moderate activity: A person might, for example, go all-out on a stationary bike for a few minutes, ease up for the next few, and then start again.


In this study, older adults who performed that type of exercise showed greater changes at the cellular level, compared to those who worked out more moderately. Specifically, interval training gave a bigger boost to mitochondrial function in the muscle. Mitochondria are the “powerhouses” within body cells that break down nutrients to be used for energy. The training also revved up activity in more genes related to mitochondrial function and muscle growth.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

Participating in physical activities and exercise on a regular basis can provide many benefits to younger and older adults alike. High intensity exercise may additionally benefit middle-aged people because it can help reverse the body's cellular aging process. For more information, please feel free to ask Dr. Jimenez or contact us at (915) 850-0900.

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Chondromalacia Patellae, Chiropractic Treatment Can Help In El Paso, TX.

Chondromalacia Patellae, Chiropractic Treatment Can Help In El Paso, TX. | Mobility & Flexibility - Joint Movement | Scoop.it

Chiropractic care is extremely beneficial for many different conditions; some obvious while others are more obscure. Structural issues that affect the knees are often very responsive to chiropractic treatment. In the case of chondromalacia patellae and other knee problems, it has proven to reduce pain and help improve the condition considerably, providing the patient with increased mobility and flexibility.

Chondromalacia Patellae (aka Runner’s Knee)

Approximately 40 percent of injuries that runners experience are knee injuries. These injuries fall under the umbrella term of “runner’s knee.” This includes chondromalacia patellae which may also be referred to as patellofemoral pain syndrome (PFMS).

 

Other runner’s knee injuries include iliotibial band syndrome, and plica syndrome. Chondromalacia patellae is one of the more common forms of runner’s knee, along with PFMS. Rest and ice are typical remedies, but when that doesn’t work or when the pain and mobility difficulties return once the patient returns to normal activities, chiropractic care is often a good treatment option.

Chondromalacia Patella

The knee is a marvelous piece of machinery. It is constructed to take the impact of the body’s weight, bending, and moving. Under the kneecap is a layer of cartilage which acts as a natural shock absorber. Injury, overuse, aging, or other conditions can cause damage to that cartilage.

 

This condition causes pain and impaired mobility, most commonly when the knees are in use such as walking up or down stairs. The pain may decrease with rest and ice, but sometimes that simply isn’t enough. Traditional treatments include physical therapy, medication for pain, and surgery.

Symptoms

The most common symptom of chondromalacia patellae is pain in the front of the knee. It is often described as a dull ache that is deep in the knee. This pain is often made worse when the patient sits with their knee bent for a long time, when they squat or kneel, or when they walk up and down stairs.

 

The more the patient uses the knee, the worse it is. However, rest and ice can work fairly quickly to help relieve the pain. If the pain persists even with rest and ice, then more aggressive care is usually advised. While traditionally doctors prescribe medication and even surgery, more patients are gravitating to drug free, less invasive treatments for knee pain. Chiropractic is a viable option.

Causes & Risk Factors

The exact cause of chondromalacia patellae is not known. Doctors have been able to link the condition to several factors. Overuse of the knee places repetitive stress on the joint. This is commonly seen in sports or activities that involve a lot of jumping or running.

 

Poor muscle control is another common factor. The muscles that surround the knee and hip don’t function properly so that tracking of the kneecap is “off.” Injury is another common factor with chondromalacia patellae. When the kneecap endures a trauma such as fracture or dislocation.

 

There are several factors that may increase a person’s risk of developing chondromalacia patellae. Age is often noted in young adults and adolescents. Older individuals with knee pain are usually experiencing effects of arthritis.

 

Gender is another risk factor. Women develop the condition twice as often as men. Doctors theorize that this is due to the skeletal structure of a woman – the pelvis is wider which increases the angle where the bones of the knee joint meet.

 

Individuals who participate in certain sports, such as those that involve a lot of jumping and running, are at an increased risk of developing the condition. This is particularly true if they suddenly increase their level of training.

Chiropractic Treatment

Successful chiropractic treatments for chondromalacia patellae include nutritional intervention as well as adjustments and stretches. The treatment is designed to stretch shortened hamstrings and adjust the sacroiliac joint.

 

The point of much of the treatment is to improve tracking of the kneecap and increase motor control. Some practitioners use soft tissue work to help patients with knee pain. The whole body approach that chiropractic care offers not only provides relief from knee pain, but often cures or reduces the condition itself.

 

If you or a loved one are suffering from knee pain, give us a call. Our Doctor of Chiropractic will do a thorough exam to determine the proper treatment protocol for your condition. You don’t have to live with pain. Again, give us a call. We’re here to help!

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. In the case of chondromalacia patellae and other knee problems, chiropractic treatment has proven to reduce pain and help improve the condition considerably, providing the patient with increased mobility and flexibility. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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Cheerleaders Benefit From Chiropractic Treatment In El Paso, TX.

Cheerleaders Benefit From Chiropractic Treatment In El Paso, TX. | Mobility & Flexibility - Joint Movement | Scoop.it

When we think of cheerleaders we usually think of pretty girls in colorful outfits, pom pom's in hand, cheering for their team. They are way more than that though. Cheerleaders are serious athletes.

 

It has taken a while, but the public is finally starting to realize just how true this is. Data collected by the National Center for Catastrophic Sports Injury Research (NCCSIR), between 1982 and 2009 cheerleaders made up for more than 70 percent of catastrophic injuries in women’s college sports. At the high school level that number exceeded 60 percent.

 

Some claim that this high percentage of injuring among cheerleaders to be due to lax regulations at the state level. Some states refuse to recognize cheerleading as a sport and organizations such as the NCAA don’t either.

 

This leaves an already potentially dangerous activity without proper oversight of the government or regulating agencies. It also means that some people who manage cheer squads and organize competitions are not required to get the same coaching and safety training standards that those in other sports do.

 

Since chiropractic treats the whole body through not just structurally via spinal adjustments, but also through soft tissue techniques, patients can receive complete care after an injury and during rehabilitation. Cheerleaders are finding that chiropractic treatment provides numerous benefits and can even make them better athletes.

Cheerleaders

Chiropractic Helps To Improve Range Of Motion

Chiropractic care helps to improve a person’s range of motion which in turn helps to prevent injury and relieve the pain of injury. Chiropractic is growing in its popularity within the field of sports medicine because of its effectiveness. A 2010 study published in The Journal of the Canadian Chiropractic Association showed that chiropractic made a significant difference in performance.

Chiropractic Helps To Prevent Injury

Even when no injury is present, athletes can benefit from chiropractic care. This is especially true in high impact sports like cheerleading where athletes push their bodies beyond limits through acrobatics and some of the strenuous tricks that they do in the course of their sport. Chiropractic helps to loosen muscles, making them more pliable and flexible, thus less prone to injury.

Chiropractic Helps To Relieve Pain From Injury

A study published in the Journal of Manipulative and Physiological Therapeutics in March 2011 shows that chiropractic for pain relief can alleviate the pain of hamstring injury. The study was conducted over the course of a football season and involved 43 professional cheerleaders for football various football teams.

 

Throughout the study the cheerleaders received specific exercise intervention. At the end of the study those who had reported pain due to a hamstring injury experienced significant pain reduction after receiving chiropractic treatment.

Chiropractic Can Increase Physical Strength

While chiropractic is typically considered a therapy for alleviating skeletal and muscular complaints, a study in 2011 showed that it can also improve physical strength. The study involved judo athletes competing at the national level who used cervical spinal manipulative therapy (SMT). The final results of the study showed a 16 percent improvement in grip strength among the athletes who received only three SMT sessions.

 

The case for chiropractic treatment for athletes is very strong. It can help during training, recovery from injury, rehabilitation, and at every point in between. Although cheerleaders are still in their infancy of being recognized as serious athletes, the case for chiropractic care as a viable sports medicine therapy to improve performance and recover from injury is significant. The benefits that cheerleaders can receive from chiropractic treatment, as we can see, can really give them something to cheer about both on the field and off.

Cheerleader Discusses Chiropractic Treatment

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Chiropractic treatment for cheerleaders can help during training, recovery from injury, rehabilitation, and at every point in between, the case for chiropractic care as a viable sports medicine therapy to improve performance and recover from injury is significant. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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February 26, 2018 9:09 PM
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Orthotics: What Chiropractic Patients Ought To Know | El Paso, TX

Orthotics: What Chiropractic Patients Ought To Know | El Paso, TX | Mobility & Flexibility - Joint Movement | Scoop.it

Orthotics: It's good to have options.

 

Individuals who suffer from a recurring medical condition, as well as those who experience an injury of one form or another, maintain the same overall goals; manage the pain, find a successful treatment option, and heal as quickly as possible. Fortunately, chiropractic care helps promote healing and strengthen the body by working on it in its entirety.

 

Experienced chiropractors understand there are some other treatments in addition to chiropractic care that help aid pain management, increase mobility, and decrease healing time. Depending on the condition, individuals may experience a wide array of benefits from blending these treatments into their chiropractic treatment.

 

One such treatment is orthotics or inserts. If life were a sandbox, chiropractic care and orthotics would be the best of friends. They treat muscle and skeletal conditions, as does chiropractic treatment. Some of the key benefits of utilizing orthotics as treatment include:

Greater Support: Orthotics

Orthotics created to "brace" the body part that is not at full performance strength allows it to heal faster.

Success In Keeping Certain Areas Immobile

Sometimes a person's injury requires little or no movement, and orthotics serve this scenario well.

Decreasing Weight Bearing On The Particular Body Part

Feet, for example, bear a great deal of the body's weight, making them one of the more difficult parts of the body to achieve healing. Inserts provide the weight bearing assistance needed to give the body time to repair and heal itself. 

Body Stabilization

If a part of the body is not functioning adequately, the entire body may be unstable. This is an unsafe situation that can actually cause other injuries. Orthotics are tools that stabilize the body by providing extra support.

Body Alignment Correction

A variety of injuries and other health conditions cause misalignment of the spine. Certain orthotics assist the body in achieving alignment over the course of time, especially when combined with chiropractic adjustments.

 

Used in the course of chiropractic treatment, orthotics provide a valuable factor in the person's recovery. Marrying the regimens of chiropractic care and orthotics supercharge the healing and recovery time.

 

Here's how:

 

Helps eliminate painful symptoms. With chiropractic visits working on the body as a whole, and orthotics offering support and stabilization, patients often show a decrease in painful symptoms faster than employing one or the other.

 

Increases the chance of returning to normal activity. Utilizing orthotics gives the area that is underperforming stabilization and support. This allows a person to more likely return to work and other daily activities faster than chiropractic treatment alone.

 

Minimizes reliance on medication. A chronically painful medical issue is quite difficult to manage without medication. Long-term use of certain medications can create health and addiction issues, leaving a person with one more problem to handle. The combination of chiropractic care and inserts empowers many individuals to lessen their dependence on drugs.

 

Maximizes quality of life. While being treated by a chiropractor, a patient’s body may take weeks or longer to stabilize before it completely heals. When inserts are coupled with chiropractic care, these same people are able to achieve a greater feeling of stability, and consequently, independence. This effect is perhaps the most significant benefit of employing the two practices, as quality of life is immeasurable.

 

No matter the injury or condition, an experienced chiropractor can determine the best regimen for each individual patient's needs. By consulting with chiropractors who also utilize inserts in their practices, most health issues can be tackled more effectively which, in return, provides even greater results.

Michael Strahan Shares Athletic TIPS

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. An experienced chiropractor can determine the best regimen for each individual patient's needs. By consulting with chiropractors who also utilize orthotics in their practices, most health issues can be tackled more effectively which, in return, provides even greater results. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

good health's curator insight, January 15, 2024 11:47 AM

Acquista Online La Prescrizione Di Perdita Di Peso
Crediamo che i farmaci a volte possano essere molto urgenti da assumere. Se hai urgente bisogno di farmaci, possiamo anche fornirti una consegna espressa,


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February 22, 2018 6:10 PM
Scoop.it!

The Knee | El Paso Chiropractor • Dr. Alex Jimenez D.C. 915-850-0900

The Knee | El Paso Chiropractor • Dr. Alex Jimenez D.C. 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it

The Knee | MRI may be requested for:

  • Ligament injuries
  • Meniscal tears and degeneration
  • Rheumatoid arthritis
  • Osteochondral fractures
  • Tendon disruptions

Bones & Cartilage Of The Knee

The knee joint is the largest, most complicated, and most vulnerable joint in the body, as it does not have a stable bony configuration. It consists of the tibiofemoral and patellofemoral articulations, which include the femur, tibia, and patella. The knee is a synovial joint that is enclosed by a ligament capsule. The capsule contains synovial fluid that keeps the joint lubricated (Figure 82). The knee provides flexible movement, but must also bear large weight and pressure loads. During walking, the knees support 1.5 times your body weight. When climbing stairs, they support 3-4 times your body weight. When squatting, your knees support 8 times your body weight.

 

 

 Figure 82. Anatomy of the knee.

 

The tibiofemoral articulation is a modified hinge joint that allows bending and straightening, but also allows for slight rotation. This articulation consists of the lateral and medial condyles of the femur resting on the lateral and medial aspects of the tibial plateau. The femoral condyles make up the distal portion of the femur, which is expanded in order to assist with weight distribution at the knee joint. The medial femoral condyle is typically larger and rounder. The condyles are united anteriorly to provide the articular surface for the patella, but they are separated posteriorly by the intercondylar notch. This notch, or fossa, is the attachment site for the cruciate ligaments, the ligaments of Humphrey and Wrisberg, and the frenulum of the patellar fat pad. A large part of the posterior distal femur is called the popliteal surface. This area is covered by fat, which separates it from the popliteal artery. The medial and lateral edges of the popliteal surface are attachment sites for muscles. Superior to the femoral condyles are the epicondyles, which are the attachment sites for muscles, tendons, and capsular ligaments. The medial epicondyle is the attachment site for the medial (or tibial) collateral ligament (Figure 83). The lateral femoral epicondyle is the attachment site for the lateral (or fibular) collateral ligament, as well as the tendon of the popliteus muscle, fibers of the iliotibial tract, and the lateral capsular ligament. Superior and posterior to the epicondyles is the most distal extent of the linea aspera, the bony ridge of the femur.

 

The tibia is the distal portion of the tibiofemoral articulation at the knee. The tibia is the second longest bone in the body, ranked just behind the femur. Its proximal end is flattened and expanded to provide a larger surface for the body weight that is transmitted through the femur. Like the femur, the proximal tibia has medial and lateral condyles. The medial condyle is larger, and somewhat flattened where it contacts the medial meniscus. The lateral condyle has a circular look to its femoral articular surface. The lateral tibial condyle articulates with the head of the fibula posteriorly, which is as close as the fibula comes to any involvement in the knee joint. Both the medial and lateral condyles rise in the center of the superior aspect of the tibia to form the intercondylar eminence. Posterior to this eminence are the attachments sites for the posterior horns of the medial and lateral menisci, which will be discussed with the ligaments of the knee. The medial and lateral tibial condyles, and the area of the intercondylar eminence are often grouped together and referred to as the tibial plateau (Figure 84). This is a critical weight-bearing area, and greatly affects the stability of the knee joint. The tibial tuberosity (or tubercle) is located on the anterior surface of the proximal tibial shaft. It has a smooth upper portion, and a roughened lower portion, which is the insertion site for the patellar tendon. The lateral side of the tibial tuberosity has a ridge for the attachment of fibers from the iliotibial tract. This is the strongest direct attachment site for the iliotibial tract. The IT tract, or band, helps in limiting lateral movement of the knee.

 

 

 Figure 84. Tibial plateau.

 

 Figure 83. Tibiofemoral anatomy. 

 

The patella is the third bone involved in the knee joint, specifically in the patellofemoral articulation. Patella means “little plate” in Latin, which describes the look and function of this sesamoid bone. The patella develops in the tendon of the quadriceps femoris muscle (Figure 85). It moves when the leg moves, and protects the knee joint by relieving friction between the bones and muscles when the knee is bent or straightened. The patellofemoral joint is a saddle-type synovial joint, allowing the patella to glide along the bottom front surface of the femur between the femoral condyles in the patellofemoral groove. Ossification of the patella is typically completed in females by age 10, and in males between the ages of 13-16. If the patella has more than one ossification center, and the additional center does not fuse, it is termed a bipartite patella (Figure 86).

 

Figure 86. Bipartite patella.

 

 

 

 Figure 85. Patella location. 

 

Articular, or hyaline, cartilage covers the ends of the bones involved in any joint. In the knee joint, this includes the distal end of the femur, the proximal end of the tibia, and the posterior aspect of the patella (Figure 87). In larger joints, this cartilage is approximately ¼” thick. Articular cartilage is white, shiny, rubbery, and slippery, enabling surfaces to slide against one another without damage. Articular cartilage is very flexible, due in part to its high water content, which also makes it highly visible on MRI. In contrast to the bones that it covers, articular cartilage has almost no blood vessels, so it is not good at repairing itself. Bones, on the other hand, have numerous blood vessels, and are good at self-repair.

 

 Figure 87. Articular cartilage.

 

Another type of cartilage is found between the femur and tibia- the fibrous cartilage that makes up the medial and lateral menisci. The menisci, also referred to as “articular disks”, wrap around the round ends of the femur to fill the space between the femur and tibia (Figure 88). Since the menisci are more fibrous in composition, they have tensile strength and can resist pressure. They can help spread the force from our body weight over a larger area. By helping with weight distribution, the menisci protect the articular cartilage on the ends of the bones from excessive forces. The menisci are fashioned to be thicker on their outsides, creating a shallow socket on the tibial surface. They act like a wedge on the rounded distal portion of the femur, improving the overall stability of the knee joint by preventing any “rolling” of the femur. Despite how strong they sound, the menisci can crack or tear when the knee is forcefully rotated or bent. The medial meniscus is fused with the medial collateral ligament, so it is less mobile than the lateral meniscus. It is often injured when the anterior or posterior cruciate ligaments are injured. The inner 2/3 of the medial meniscus receives a limited blood supply, so the entire meniscus is usually slow to heal. The lateral meniscus suffers from fewer injuries than the medial meniscus. Meniscal tears are one of the most common causes of knee pain, with suspected meniscal tears the most common indication for an MRI of the knee joint. 

 

Figure 88. Superior view of menisci of right knee.

 

Symptoms that might indicate a problem with the bones of the knee joint include locking of the joint, the knee giving way, crackling or grinding felt in the joint, and pain and swelling. Locking of the joint can be indicative of a “loose body” (bone, cartilage, or foreign object) in the joint space, which can often be removed through arthroscopy (Figure 89). A knee that gives way can indicate that the patella is out of the patellofemoral groove, which leaves the knee unstable. Crackling and grinding at the joint can result from degenerative arthritis or osteoarthritis, as well as from a dislocating patella. An increase in pain with activity can occur due to a stress fracture or bone fracture. One of the pathologic conditions that can affect the bones of the knee joint is osteochondritis dissecans, which can affect the distal femur, and was discussed previously with the femur anatomy. Various types of arthritis manifest in the bones of the knee joint, including osteoarthritis, infectious arthritis, and rheumatoid arthritis. Chondromalacia patella, also known as patellofemoral syndrome or “runner’s knee” results from an irritation of the undersurface of the patella (Figure 91). If the patella is not tracking correctly in the patellofemoral groove, the articular cartilage may rub against the knee joint (Figure 90). The cartilage degenerates, and becomes irritated and painful. This condition is most common amongst young, healthy athletes, especially females and runners that are flat-footed. Treatment is typically rest and physical therapy to stretch and strengthen the quads and hamstrings. If surgery is required, it may be to perform a “lateral release”, as the abnormal tracking of the patella can cause a tightening of the lateral tissues of the knee. The lateral release procedure cuts the tight tissues, so the patella can return to its normal position and tracking. Osgood-Schlatter disease involves the anteriorly located tibial tuberosity, and the patellar tendon that inserts on that tuberosity (Figures 92, 93). This condition affects children during their growth spurts, and is typically found more in boys. During growth spurts, contractions of the quad muscle put additional stress on the patellar tendon at its attachment site on the tibial tuberosity. This can result in multiple subacute avulsion fractures and inflammation of the tendon. Excess bone growth occurs on the tuberosity, and a lump on the tuberosity can be seen and felt. This lump can become irritated and swollen, causing knee and leg pain. This condition is typically worsened with running, jumping, and climbing stairs. Osgood-Schlatter usually resolves with rest, ice, compression and elevation, as well as maturity of the youngster’s skeleton.

 

Figure 89. Intraarticular loose body.

 

Figure 90. Patellofemoral groove. 

 

Figure 91. Patellofemoral syndrome or “runner’s knee”.

 

Figure 92. Xray displaying Osgood-Schlatter disease.

 

Figure 93. MRI displaying Osgood- Schlatter disease.

Ligaments Of The Knee

Ligaments are the tough bands of tissue that connect bones. They are considered to be “viscoelastic”, meaning they can gradually lengthen under tension, but return to their original shape when the tension is removed. However, if they are stretched for a prolonged period of time, or past a certain point, the ligaments cannot retain their original shape, and may eventually tear or snap. This is one of the reasons that a dislocated joint should be re-located as quickly as possible. If the ligaments lengthen, they leave the joint weakened and prone to future dislocations. Controlled stretching exercises to lengthen ligaments, and make the joints more supple, are part of the daily routines of athletes, gymnasts, dancers, etc. Damaged ligaments can lead to unstable joints, wearing of the cartilage, and eventually osteoarthritis. The numerous ligaments of the knee joint are the most important structures in controlling stability of the knee. Many of these ligaments were mentioned in the femur anatomy section, as they have attachments on the distal femur. The more important ligaments will be reviewed here in greater detail, in regards to their functions in the knee joint. The main intracapsular ligaments are the anterior and posterior cruciates (Figures 94, 95). Intracapsular ligaments are not very common in synovial joints. They provide stability, but permit a larger range of motion as compared to capsular or extracapsular ligaments. The anterior cruciate ligament (ACL) stretches from the lateral femoral condyle to the anterior intercondylar area of the tibia, preventing the tibia from being pushed too far anterior relative to the femur. It is the more commonly injured of the cruciate ligaments, and can be torn during twisting and bending of the knee. Women are at higher risk for ACL ruptures due to the facts that the maximum diameter of the intercondylar fossa is in its posterior aspect (the ACL attaches anteriorly), and the overall width of the intercondylar fossa is smaller in females. The posterior cruciate ligament (PCL) stretches from the medial femoral condyle to the posterior intercondylar area of the tibia, preventing posterior displacement of the tibia relative to the femur. It is the stronger of the two cruciate ligaments, and is injured less frequently; however, it can be injured from direct force or trauma. The menisci are also considered to be intracapsular structures, with connections to ligaments inside and outside the joint capsule. Two of their intracapsular ligaments are the anterior and posterior transverse meniscomeniscal ligaments. They attach the medial and lateral menisci to each other at their anterior and posterior aspects. Posterior transverse meniscal ligaments are very rare- only 1-4% of knees will have them. Two additional intermeniscal ligaments are the medial and lateral oblique meniscomeniscal ligaments (Figure 96). Their names describe their anterior horn attachment sites; they attach on the posterior horn of the opposite meniscus (i.e. medial oblique meniscomeniscal attaches to the anterior horn of the medial meniscus and posterior horn of the lateral meniscus). The oblique meniscomeniscal ligaments both traverse the intercondylar notch, and pass between the anterior and posterior cruciate ligaments (Figure 97).

 

Figure 94. Cruciate ligaments and menisci.

 

Figure 95. Posterior view of cruciate ligaments of left knee.

 

Figure 96. Axial fatsat T2 FSE image with arrow indicating
oblique meniscal ligament coursing from anterior horn of
medial meniscus to posterior horn of lateral meniscus.

 

Figure 97. Sagittal dual-echo T2 through the intercondylar notch at the level of the posterior cruciate ligament (curved arrow); thin linear structure of low signal intensity inferior to PCL represents the oblique meniscomeniscal ligament (straight arrow); sometimes misinterpreted as displaced meniscal fragment.

 

The medial (or tibial) collateral ligament is considered a capsular ligament, as it is part of the articular capsule surrounding the synovial knee joint. It acts as mechanical reinforcement for the joint, protecting the knee from valgus force, or being bent open medially due to stress on the lateral side of the knee. The medial collateral ligament (MCL) is one of the most commonly injured of all knee ligaments, occurring in all sports, in all ages, and often times with medial meniscal tears (Figures 98-101). It has both superficial and deep components. Fibers from the superficial portion of the MCL attach to the medial epicondyle of the femur and the medial tibial condyle. Fibers from the deep medial collateral ligament attach to the medial meniscus. Proximal to the attachment point, this ligament is referred to as the meniscofemoral ligament, as it attaches the medial meniscus to the medial aspect of the femur. Distal to the meniscal attachment, the ligament is referred to as the meniscotibial (or coronary) ligament, as it attaches the medial meniscus to the medial aspect of the tibia. The meniscofemoral and meniscotibial are also referred to as the meniscocapsular or medial capsular ligaments, as they play an important role in anchoring peripheral parts of the medial meniscus in the medial side of the knee. The meniscotibial ligament is typically injured more often than the meniscofemoral ligament. The meniscotibial ligament attaches to the tibia several millimeters inferior to the articular cartilage. Its job is to stabilize and maintain the meniscus in its proper position on the tibial plateau. Disruption of the meniscotibial ligament can result in a floating meniscus or meniscal avulsion, while the meniscofemoral ligament may not be affected. The deep medial collateral ligament is short, and tightens quickly with rotation motions. It is often damaged, along with the ACL, when the mechanism of injury involves tibial rotation. Diagnosis and surgical repair of the deep medial collateral ligament can be challenging.

  

Figure 98. Normal MCL is linear, has low signal intensity.

 

Figure 99. Grade 1 sprain shows adjacent edema, no change in signal intensity of MCL.

 

 Figure 100. Grade 2 sprain or partial tear shows increased edema,

abnormal signal intensity, thickening or thinning of ligament.

 

Figure 101. Grade 3 involves complete disruption of ligaments or attachments.

 

In addition to fibers of the medial collateral ligament, the deep portion of the capsular compartment of the medial knee is the location of the medial knee’s posterior support. The posterior oblique ligament is attached proximally to the medially located adductor tubercle of the femur, and distally to the tibia and the posterior aspect of the knee joint capsule. If the posterior oblique is injured, it is usually torn from its femoral origin. The posterior oblique ligament provides static resistance to valgus loads as the knee moves into full extension, as well as dynamic stabilization to valgus forces (stress from lateral side) as the knee moves into flexion. It acts as an important restraint to posterior tibial translation in cases of posterior cruciate ligament injury. The posterior oblique ligament has three “arms”. Its superior capsular “arm” becomes continuous with the posterior knee capsule, and the proximal portion of the oblique popliteal ligament. The oblique popliteal ligament is also an important posterior stabilizing structure for the knee joint Figure 102). It extends from the posteromedial aspect of the tibia, running obliquely and laterally upward to insert near the lateral epicondyle of the femur.

 

Figure 102. Oblique popliteal ligament in posterior view of knee.

 

Figure 103. Medial (tibial) and lateral (fibular) collateral ligaments. 

  

The lateral (or fibular) collateral ligament is considered an extracapsular ligament. It helps to provide joint stability and protects the lateral side of the knee from varus forces, or inside bending forces that are directed at the medial side of the knee. Injuries to the lateral collateral ligament are less common than injuries to the medial collateral, as the opposite leg can guard against medial forces that can lead to lateral collateral injuries. Injuries can occur in sports such as soccer and rugby, where the knee is extended and unprotected during running. The lateral, or fibular, collateral ligament stretches obliquely downward and backward, from the lateral epicondyle of the femur to the head of the fibula (Figure 103). It is not fused with the capsular ligament or with the lateral meniscus, so it has increased flexibility and decreased incidence of injury when compared to the medial collateral ligament. Similar to the medial meniscus, the lateral meniscus has a meniscotibial, or coronary, ligament. It connects the inferior edges of the lateral meniscus to the periphery of the tibial plateau. The lateral meniscus also has a meniscofemoral ligament that extends from the posterior horn of the lateral meniscus to the lateral aspect of the medial femoral condyle. It is given two distinct names, based on its location in relation to the posterior cruciate ligament (PCL). The ligament of Humphrey passes in front of the posterior cruciate ligament. It is less than 1/3 the diameter of the posterior cruciate ligament, but may be confused for the posterior cruciate during arthroscopy. The ligament of Wrisberg passes behind the posterior cruciate ligament, and is about ½ of the posterior cruciate’s diameter (Figure 104). Its femoral origin often merges with the posterior cruciate ligament. Both ligaments are present in only about 6% of knees. Approximately 70% of people have one or the other of these ligaments, with the majority possessing the more posterior ligament of Wrisberg (Figure 105). MRI is the preferred imaging modality for medial collateral or lateral collateral ligament injuries, as it can detect any associated internal knee derangements, cruciate-collateral ligament injuries, or cartilage deficiencies.

 

Figure 104. Rendering of posterior knee, arrow indicates Ligament of Wrisberg; courses obliquely from lateral aspect of medial femoral condyle to posterior horn of lateral meniscus,

remains posterior to PCL.

 

Figure 105. Arrow indicates “Wrisberg pseudo-tear”; intermediate signal intensity line at junction of Ligament of Wrisberg and normal posterior horn of lateral meniscus; often mistaken for a meniscal tear.

 

The patellar ligament is the connection between the patella and the tibia, extending from the apex (inferior aspect) of the patella to the tibial tuberosity. Technically, it is connecting two bones, so it is a ligament. However, it is most often referred to as the patellar tendon, because the superficial fibers that cover the front of the patella and extend to the tibia are continuous with the central portion of the common tendon of the quadriceps femoris muscle. The posterior surface of the patellar ligament is separated from the synovial membrane of the knee joint by a large infrapatellar pad of fat. Injuries to the patellar ligament can occur from overuse, such as sports that involve jumping and quick directional changes, as well as running-related sports. This is the ligament that is injured in jumper’s knee (or patellar tendonitis), which begins with inflammation, and can lead to degeneration or rupture of the patellar ligament and the tissue around it (Figure 106). Patients with patellar ligament injuries typically complain of pain in the area below the kneecap, which will increase with walking, running, squatting, etc. They can often be treated in the same manner as other soft tissue injuries- with rest, ice, compression and elevation. The patellar ligament attachment at the tibial tuberosity is the site of Osgood-Schlatter disease, which was discussed previously.

 

Figure 106. Patellar tendonitis (jumper’s knee).

 

Along the sides of the patella and the patellar ligament are the medial and lateral patellar retinacula (Figure 107). They are fibrous tissue stabilizers for the patella that form from the medial and lateral portions of the quad tendons as they pass down to insert on either side of the tibial tuberosity. The lateral retinaculum is the thicker of the two, but both have superficial and deep layers. Within the deep layers are various ligaments (whose names indicate the structures they connect) that help support the patella in its position, relative to the femur below it. The deep layer of the lateral patellar retinaculum is the location where the lateral patellofemoral ligament meets the iliopatellar band, which is a tract of fibers from the iliotibial (IT) band that connects to the patella. The deep layer of the medial patellar retinaculum has three focal capsular thickenings, referred to as the medial patellofemoral, medial patellomeniscal, and medial patellotibial ligaments. The medial patellofemoral ligament is strong enough to influence patellar tracking, and acts as a major medial restraint. Imbalances in the forces that control patellar tracking during flexion and extension of the knee can lead to patellofemoral pain syndrome (runner’s knee), one of the most common causes of knee pain. This can result from overuse, trauma, muscle dysfunction, patellar hypermobility, and poor quadriceps flexibility. Typical symptoms include pain behind or around the patella that is increased with running, and activities that involve knee flexion. MRI is typically not necessary for this diagnosis. Physical therapy has been found to be effective for the treatment of patellofemoral pain syndrome.

 

Figure 107. Lateral and medial retinaculum.

Muscles & Tendons Of The Knee

The flexor and extensor muscles of the knee have been discussed previously, as the majority of them are the anterior and posterior muscles of the thigh. We will review the thigh muscles involved in knee movement, and add two muscles of the lower leg that also affect the knee. The quadriceps femoris muscles of the anterior thigh are the main knee extensors (Figure 108). As these muscles contract, the knee joint straightens. The tendons of the vastus medialis, vastus intermedius, vastus lateralis, and rectus femoris join at the superior aspect (base) of the patella to form the patellar tendon. This tendon continues over the patella and attaches it to the tibial tuberosity (since it is connecting bone to bone, it is sometimes called the patellar ligament). The quadriceps, along with the gluteal muscles, are responsible for the thrusting forces necessary for walking, running, and jumping. The quads also help control movement of the patella, as they are attached to it by the quadriceps tendons (Figure 109). The patella increases the force exerted by the quadriceps muscles as the knee is straightened.

 

Figure 108. Anterior thigh muscles - knee extensors.

  

Figure 109. Quadriceps controlling the patella.

 

The posterior thigh muscles, also known as the hamstrings, are the main knee flexors, with assistance from the sartorius, gracilis, gastrocnemius, and popliteus muscles. The knee bends when the hamstrings contract. The hamstring muscles give the knee joint the strength needed for propulsion in running and jumping. They also help to stabilize the knee by protecting the collateral and cruciate ligaments, especially when the knee twists. The three hamstring muscles have varying attachment sites around the knee joint (Figure 110). The biceps femoris attaches to the head of the fibula and the superolateral aspect of the tibia. The semitendinosus attaches on the anterior aspect of the tibia, medial to the tibial tuberosity, crossing over the medial collateral ligament. The tendon of the semitendinosus muscle is sometimes used for cruciate ligament reconstruction. The semimembranosus attaches at the posteriomedial aspect of the medial tibial condyle. The sartorius muscle is also a knee flexor, although it is an anterior thigh muscle. It inserts on the anterior medical aspect of the tibia. The gracilis muscle of the medial thigh is one of the hip adductors, but also plays a part in knee flexion. Like the semitendinosus tendon, the tendon of the gracilis is sometimes used for cruciate ligament reconstructions. The gracilis attaches to the medial aspect of the proximal tibia.

 

Figure 110. Posterior knee
muscles - knee flexors.

 

Additional flexors of the knee joint include some of the posterior muscles of the lower leg. The large superficial gastrocnemius muscle has a medial and a lateral head, which originate from the medial and lateral femoral condyles, respectively. It runs the length of the posterior lower leg, attaching to the calcaneus by the Achilles tendon. The gastrocnemius gives us the ability to flex our knee while our foot is flexed, as it connects to both joints. It is involved in standing, walking, running, and jumping. The popliteus is a deep posterior lower leg muscle that helps with knee flexion, and also rotates the tibia medially, which aids in knee stability. The popliteus originates from the outer margin of the lateral meniscus of the knee joint. It extends posteriorly and inserts on the medial aspect of the tibia, inferior to the medial tibial epicondyle.

 

The important tendons of the knee include the quadriceps, patellar, and hamstring tendons, and the iliotibial band (Figure 111). Tendons attach muscles to bones. These major knee tendons have all been discussed with either the bones or the muscles that they attach. The quadriceps tendon was mentioned with the quadriceps muscle as the muscle’s attachment to the patella. The quad tendon continues over the patella, then attaches the apex of the patella to the tibial tuberosity. It is then called the patellar tendon (or ligament). Hamstring tendons were discussed with the hamstring muscles, the posterior muscles that are flexors of the knee. Hamstring tendons are sometimes used for cruciate ligament reconstructions. Tendonitis, which is the inflammation of a tendon, is a common knee injury amongst athletes in a variety of sports. The iliotibial band (or IT tract) functions like a tendon, as it attaches the knee to the tensor fasciae latte muscle. The band is actually a fibrous reinforcement of the fascia lata, or deep tissue of the thigh. It runs from the ilium to the tibia. Proximally, it acts as a hip abductor, while distally it acts as lateral stabilization for the knee, and aids with medial rotation of the tibia. The IT band is in constant use during walking and running, which can lead to irritation at the point where it passes over the lateral femoral epicondyle. A ‘tight” IT band can cause inflammation and/or irritation at the femoral epicondyle, or at the point of insertion on the lateral tibial condyle. This condition is called IT band friction syndrome. It is common amongst runners, hikers, and cycling enthusiasts.

 

Figure 111. Tendons of the knee.

Nerves Of The Knee

The main nerves to the knee that come from the sacral plexus of nerves are the tibial nerve and the common peroneal nerve (Figure 112). Both are branches of the sciatic nerve, and begin posteriorly, slightly above the actual knee joint. Both of these nerves, or their branches, continue through the lower leg and foot, providing sensation and muscle control. The tibial and common peroneal nerves are also both involved in cutaneous innervation, which is the supply of nerves to the skin of the knee. The tibial nerve remains posterior and more medial, branching at the medial ankle to innervate the foot. The common peroneal nerve begins posterolaterally, moving anteriorly near the neck of the fibula. It then branches into the superficial and deep peroneal nerves, which continue their anterior descent to the foot. The tibial and common peroneal nerves are the most commonly injured nerves when a knee is dislocated. Nerves can grow back, but they do so at a rate of approximately ½ inch per month.

 

Figure 112. Sacral plexus nerves of knee.

 

Nerves from the lumbar plexus that affect the knee include the lateral femoral cutaneous, and the saphenous, which is a branch of the femoral nerve (Figure 113). The saphenous nerve travels more medially and gives off infrapatellar branches around the knee joint. Below the knee, the saphenous nerve sends branches to the skin of the anterior and medial lower leg. The lateral femoral cutaneous nerve sends an anterior branch to the skin of the anterior and lateral thigh, down to the area of the knee. Terminal filaments of this nerve communicate with the infrapatellar branch of the saphenous nerve, forming the peripatellar plexus.

  

Figure 113. Lumbar plexus nerves of knee.

Arteries & Veins Of The Knee

The popliteal artery, a branch of the superficial femoral artery, is the main arterial supply to the knee joint. It runs along the posterior aspect of the distal femur, behind the knee joint. At the supracondylar ridge, the popliteal artery gives off the blood supply to the knee, which consists of various genicular arteries (Figure 114). Inferior to the knee joint, the popliteal branches into the anterior and posterior tibial arteries, which supply the lower leg. The popliteal artery is a common site for both atherosclerosis and aneurysms, and is listed as the most common site for peripheral arterial aneurysms. Approximately 50% of these aneurysms are bilateral. Although they rarely rupture, popliteal aneurysms may serve as a focus for abrupt thrombotic occlusion of the involved popliteal artery, which can affect the foot on the same side. A thrombus within an aneurysm can also lead to a distal embolism. The genicular arteries are sources of continued blood flow to the knee and lower limb, in case of an obstructed popliteal artery. The descending genicular, also called the highest or supreme genicular, branches from the femoral artery, just superior to the popliteal branch. It supplies the adductor magnus and hamstring muscles, then joins with the network of genicular arteries around the knee joint. The middle genicular pierces the oblique popliteal ligament, and supplies the ligaments and synovial membrane inside the knee articulation (including the ACL and PCL). The sural artery joins the anastomoses of the genicular arteries, and also supplies muscles of the lower leg, including the large gastrocnemius muscle. The anastomotic pattern around the knee joint is supplied by the popliteal artery posteriorly, the descending genicular artery medially, and the descending branch of the lateral circumflex femoral artery laterally. The genicular arteries involved in the anastomosis are labeled as the medial and lateral superior geniculars, and the medial and lateral inferior geniculars.

 

Figure 114. Arteries of knee.

 

The major deep veins around the knee joint are the popliteal vein, and the anterior and posterior tibial veins (Figure 115). The popliteal vein begins at the junction of the tibial veins in the posterior aspect of the lower leg, just inferior to the knee joint. It ascends posteriorly, continuing as the femoral vein about halfway up the thigh. As deep veins typically follow the arteries, the genicular veins accompany the genicular arteries around the knee joint, then drain into the popliteal vein. The important superficial veins around the knee joint are the small and great saphenous veins. Superficial veins typically do not follow arteries, but rather travel with cutaneous nerves. The small saphenous ascends the lower leg posteriorly, angling from lateral to medial. It merges with the popliteal vein at a position slightly superior to the knee joint. The great saphenous vein, the longest vein in the body, has a medial and anterior course in the lower leg. It moves to a posterior position, but stays medial along the knee joint, moving alongside the medial epicondyle of the femur. The great saphenous then moves anteriorly again through the thigh. 

 

Figure 115. Veins of knee.

 

Varicose and “spider” veins are often seen in the leg in the posterior aspect of the knee joint. As mentioned previously, in the femoral vein discussion, veins have valves to ensure the “one-way” uphill flow of blood back to the heart (Figure 116). Communicating vessels, also called perforating veins, exist between the deep and superficial veins to help compensate for valves that may be incompetent, and are allowing blood reflux. If venous walls are weakened or dilated, the cusps of the valves can no longer close properly, and the valves can become incompetent. This leads to an increase in the weight of the column of blood for the veins that are “downstream” from the bad valve. Blood can pool in these veins, causing them to become varicose, where the veins swell, become tortuous, and even bulge through the skin surface. Reticular veins, which are smaller varicose veins that do not bulge through the skin, as well as very small “spider” veins are both typically less severe conditions, but both still involve the backwards flow of blood. Removal of severe varicose veins will actually help blood flow, as the blood will no longer be stagnant in the pooled areas.

 

Figure 116. Varicose veins around knee.

Bursae Of The Knee

The synovial knee joint is home to a large number of bursae (Figure 117). These are fluid sacs and synovial pockets that surround and sometimes communicate with the joint cavity. They facilitate friction-free movement between the bones and moving structures (tendon, muscle). Fluid or debris can collect in the bursa, or fluid can extend into the bursa from the adjacent joint in situations such as excessive friction, infection or direct trauma. This type of pathological enlargement of the bursa is referred to as bursitis, which can mimic several peripheral joint and muscle abnormalities. Radiologists must be able to accurately identify bursal pathology, especially amongst the numerous knee bursae (14 reported in some literature). We will identify a few of the more common bursa, beginning with the suprapatellar bursa. This bursa lies between a quadriceps tendon and the femur, superior to the patella (Figure 118). Fluid is commonly found here when patients have a joint effusion. Bursitis of the prepatellar bursa is also known as “housemaid’s knee”. It occurs from repetitive trauma from kneeling, as seen with housemaids, wrestlers, and carpet-layers. This bursa is found between the patella and the skin (Figure 119). Inflammation of the superficial infrapatellar bursa may be called “Clergyman’s knee”, another bursitis that can occur from excessive kneeling. This bursa is located between the distal third of the patellar tendon and the overlying skin (Figure 120).

 

Figure 117. Bursae in the knee.

  

Figure 118. T2 gradient
displaying suprapatellar
bursa.

  

Figure 119. T2
fatsat displaying
prepatellar bursa.

 

Figure 120. T2 fatsat
displaying infrapatellar
bursa.

 

The synovial sac of the knee joint sometimes forms a posterior bulge, known as a Baker’s cyst or popliteal cyst (Figure 121). It typically forms between the tendons of the medial head of the gastrocnemius muscle and the semimembranosus muscle, posterior to the medial femoral condyle. Baker’s cysts are not true cysts, as they typically maintain open communication with the synovial sac. However, they can pinch off, and they can rupture. They are usually asymptomatic, but can be indicative of another problem of the knee, such as arthritis or a meniscal tear. Aspiration of the synovial fluid can be performed if the cyst becomes problematic. Treatment is usually necessary if a Baker’s cyst ruptures, as it can cause acute pain behind the knee, and swelling of the calf muscles. A ruptured cyst can also mimic a DVT or thrombophlebitis. Ultrasound and MRI can both be used for confirmation of a Baker’s cyst (Figure 122).

 

Figure 121. Lateral view of Baker’s cyst.

  

Figure 122. Sagittal image of Baker’s cyst on MRI.

 

Scan Setups

The following are HMSA suggestions for knee imaging. Knee protocols should be designed to yield diagnostic images of the menisci, bones, articular cartilage, and all ligamentous structures of the knee. While many radiologists may require additional imaging of the ACL, protocols that are designed for optimal imaging of the cartilage and menisci should also produce adequate images of the ACL. Always check with your radiologist for his/her imaging preferences.

Axial Scans

When positioning axial slices for the knee, sagittal and coronal images can be used to insure inclusion of all pertinent anatomy. The slices should extend superiorly to include the entire patella, and inferiorly to include the tibial tuberosity and patellar tendon insertion. A presat can be placed over the unaffected lower extremity to reduce the possibility of wrap-around artifact, as seen in the coronal image in Figure 139.

 

Figure 139. Axial slice setup using sagittal and coronal images.

Coronal Scans

Coronal slices of the knee should include the anatomy from the posterior femoral condyles to the anterior portion of the patella. Visualize a line connecting the lateral and medial condyles of the femur. Typically, the coronal slices are angled so that they are parallel to that line, as seen in the axial image in Figure 140.

 

Figure 140. Coronal slice setup using axial and sagittal images.

Sagittal Scans

Sagittal slices should include the anatomy from the medial condyle to the lateral condyle. The slice group may be angled per your radiologist’s preference, but should remain perpendicular to the coronal slices. Typically, the slice group is angled so that it is parallel to the medial border of the femoral condyle, as seen in the axial image in Figure 141.

 

Figure 141. Sagittal slice setup using axial and coronal images.

 

In addition to routine oblique sagittal images, some radiologists prefer an additional sagittal scan of the ACL with thin slices and high spatial resolution. Axial and coronal images can be used for slice setup. Referenced literature recommends that the angle of the slice group should not exceed 10° from a line drawn perpendicular to the bicondylar line (line that connects the posterior femoral condyles), as seen in Figure 142.

 

Figure 142. Sagittal ACL slice setup using axial and coronal images.

 

References:

 

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Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. The knee joint is the largest, most complicated, and most vulnerable joint in the body, as it does not have a stable bony configuration. Climbing stairs, they support 3-4 times your body weight. When squatting, your knees support 8 times your body weight. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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February 9, 2018 6:53 PM
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Hips Positioning And MRI Anatomy

Hips Positioning And MRI Anatomy | Mobility & Flexibility - Joint Movement | Scoop.it

Hips Positioning & MRI Anatomy


MRI may be requested for:

  • Bone tumor
  • Osteoarthritis
  • Aseptic or avascular necrosis
  • Bursitis
  • Pain

Bones & Cartilage Of The Hips

The hips joints join the legs to the trunk of the body, and are formed by the femurs and pelvic bones. The hips are ball-and-socket type joints, where the femoral head (ball) fits into the cup-shaped acetabulum (socket) of the pelvis (Figure 1). When compared to the shoulder, which is also a ball-and-socket joint, the acetabulum is a deeper socket, and encompasses a greater area of the ball, or femoral head. This accommodation is necessary to provide stability for the hip, as it is a major weight-bearing joint, and one of the largest joints in the body. When not weight-bearing, the ball and socket of the hip joint are not perfectly fitted. However, as the hip joint bears more weight, the surface area contact increases, and the joint becomes more stable. When in a standing position, the body’s center of gravity passes through the center of the acetabula. While walking, weight-bearing stresses on the hips can be five times a person’s body weight. Healthy hips can support your weight and allow for pain-free movement. Hip injuries or disease can cause changes that affect your gait, as well as changes that affect the ability of the hips to distribute weight bearing. Abnormal stress is then placed on the joints that are above and below the hips. 

 

The three fused hips or innominate bones that form the acetabulum include the ilium, pubis, and ischium. The ilium forms the superior aspect, the pubis forms the inferior and anterior aspect, and the ischium forms the inferior and posterior aspect. The depth of the acetabulum socket is further increased by the attached fibrocartilaginous labrum (Figure 2). In addition to providing stability to the hip joint, the labrum allows flexibility and motion. Hip joint stability can be hampered by injuries resulting from playing sports, running, overuse, or falling, as well as by disease or tumor. MRI of the hips may be ordered to assess the joint(s) for internal derangement, fracture, or degenerative joint disease. A blow to the hip joint or a fall can result in dislocation of the hip, or a hip fracture. Osteoporosis or low bone density can also lead to hip fractures. Successful prevention and/or treatment of osteoporosis may be achieved through nutrition (adequate amounts of calcium, vitamin D and phosphorus), exercise, safety measures, and medications.

 

Articular cartilage covers the femoral head and the acetabulum (Figure 3). This cartilage is thin but tough, flexible, smooth and slippery, with a rubbery consistency. It absorbs shock, and allows the bones to move against each other easily and without pain. It is kept lubricated by synovial fluid, which is made in the synovial membrane (joint lining). Synovial fluid is both viscous and sticky. This fluid is what allows us to flex our joints under great pressure without wear. The articular cartilage of the hip is typically about ¼ inch thick, except in the posterior aspect of the hip socket (Figure 4). Here, the cartilage is thicker, as this area absorbs most of the force during walking, running, and jumping. MRI of the hip joint can detect problems involving both the articular cartilage and the fibrocartilaginous ring, or labrum. Cartilage has minimal blood vessels, so it is not good at repairing itself. Fraying, fissuring, and other abnormalities or defects of the cartilage can lead to arthritis in the hip joint. Contrast can be directly injected in the hip joint for a detailed look at the cartilage and labrum.

 

The femurs are the longest bones in the body, with large round heads that rotate and glide within the acetabula of the pelvis. The femoral head is particularly subject to pathologic changes if there is any significant alteration of blood supply (avascular necrosis). The femoral neck connects the head of the femur to the shaft. The neck ends at the greater and lesser trochanters, which are sites of muscle and tendon attachments. A disease characterized by an inadequate blood supply to the femoral head is Legg-Calve-Perthes disease, also known as LCP or simply Perthes disease. This is a degenerative disease of the hip joint that affects children, most commonly seen in boys ages two through twelve. One of the growth plates of the femoral head, the capital femoral epiphysis, is inside the joint capsule of the hip. Blood vessels that feed this epiphysis run along the side of the femoral neck, and are in danger of being torn or “pinched off” if the growth plate is damaged. This can result in a loss of blood supply to the epiphysis, leading to a deformity of the femoral head (Figure 5). The femoral head may become unstable and break easily, which can lead to incorrect healing and deformities of the entire hip joint (Figure 6). Treatment of Perthes disease is centered on the goal of returning the femoral head to a normal shape. Surgical and non-surgical treatments are used, based on the idea of “containment”- holding the femoral head in the acetabulum as much as possible, while still allowing motion of the hip joint for cartilage nutrition and healthy growth of the joint.

 

High level athletes and active individuals may be susceptible to a hip condition known as Femoro-Acetabular Impingement, or FAI. FAI is characterized by excessive friction in the hip joint. The femoral head and acetabulum rub abnormally, and can create damage to the articular or labral cartilage. FAI is also associated with labral tears, early hip arthritis, hyperlaxity and low back pain. FAI generally occurs in two forms: Cam and Pincer. The Cam form results in abnormal contact between the femoral head and the socket of the hip because the femoral head and neck relationship is aspherical (Figure 7). Males and those involved in significant contact sports typically display Cam impingement. Pincer impingement occurs when the acetabulum covers too much of the femoral head, resulting in the labral cartilage being pinched between the rim of the socket and the anterior femoral head-neck junction (Figure 8). Pincer impingement may be more common in women. Typically, these two forms exist together, and are labeled as “mixed impingement” (Figure 9).

 

Ewing’s sarcoma is a malignant bone tumor that may affect the pelvis and/or femur, thereby also affecting the stability of the hips. Like Perthes disease, Ewing’s sarcoma is more common in males, typically presenting in childhood or early adulthood. MRI is routinely used in the work-up of these malignant tumors to show bony and soft tissue extent of the tumor, and its relation to nearby anatomic structures (Figure 10). Contrast may be used to help determine the amount of necrosis within the tumor, which aids in determining the response to treatment before surgery.

 

 Figure 10. MRI demonstrating Ewing’s sarcoma.

Ligaments Of The Hips

Hip stability is further increased by three strong ligaments that encompass the hip joint and form the joint capsule. These ligaments connect the femoral head to the acetabulum, with names suggestive of the bones they connect. They include the pubofemoral and iliofemoral ligaments anteriorly, and the ischiofemoral ligament posteriorly (Figure 11). The iliofemoral ligament is the strongest ligament in the body. However, sports and overuse can still result in sprains of these sturdy ligaments of the joint capsules of the hips. A smaller ligament, the ligamentum teres, is an intracapsular ligament that connects the tip of the femoral head to the acetabulum (Figure 12). A small artery within this ligament brings some of the blood supply to the femoral head. Damage to the ligamentum teres, and its enclosed artery, can result in avascular necrosis.

Muscles & Tendons Of The Hips

The muscles of the thigh and lower back work together to keep the hip stable, in alignment, and able to move. The hip gains stability because the hip muscles do not attach right at the joint. Hip muscles allow the movements of flexion, extension, abduction, adduction, and medial and lateral rotation. To better understand the functions of the muscles surrounding the hip, they can be divided into groups based on their locations- anterior, posterior, and medial.

 

The anterior thigh muscles are the main hip flexors, and are located anterior to the hip joint. Seventy percent of the thigh’s muscle mass is made up of the quadriceps femoris muscle, so named because it arises from four muscle heads- the rectus femoris, vastus medialis, vastus intermedius, and vastus lateralis (Figures 13, 14). The rectus femoris is the only one of the “quad” muscles to cross the hip joint. The sartorius muscle is found anterior to the quadriceps, and also serves as an abductor and lateral rotator of the hip. The most powerful of the anterior thigh hip flexors is the iliopsoas, which originates in the low back and pelvis and attaches at the lesser trochanter. 

 

Posterior hip muscles include those of both the thigh and gluteal regions. The posterior thigh muscles are also known as the hamstrings- semimembranosus, semitendinosus, and biceps femoris (Figure 15). These muscles originate at the inferior pelvis, and are the extensors for the hip. They are active in normal walking motions. When the hamstrings are “tight”, they limit hip flexion when the knee joint is extended (bending forward from the waist with knees straight), and can limit lumbar movement, leading to back pain. The gluteal muscles include the gluteus maximus, medius, and minimus, six deep muscles that serve as lateral rotators, and the tensor fasciae latae. The three gluteals and the anterior sartorius muscle are all involved in abduction. The gluteus maximus is the main hip extensor, and is the most superficial of the gluteal muscles. It is involved in running and walking uphill, and assists with normal tone of the iliotibial band, which lies lateral to it. The gluteus medius and minimus both insert at the greater trochanter of the femur. The minimus is the deepest of the three gluteal muscles. Anterior to the gluteus minimus is the tensor fasciae latae muscle. It is a flexor and medial rotator of the hip, originating from the anterior superior iliac spine (ASIS) and inserting on the iliotibial band. The term “tensor fasciae latae” defines this muscle’s job- “muscle that stretches the band on the side”. This muscle helps the iliopsoas, gluteus medius, and gluteus minimus muscles during flexion, abduction and medial rotation of the thigh by making the iliotibial band taut, thereby steadying the trunk and stabilizing the hip (Figure 16). The iliotibial band or tract is not a muscle, but a thickened, fibrous band of deep fascia, or connective tissue. It is found at the lateral aspect of the thigh, and runs from the ilium to the tibia. It encloses the muscles and helps with lateral stabilization of the knee joint, as well as helping to maintain both hip and knee extension. Tightening of the iliotibial (IT) band typically causes more problems at the knee as opposed to the hip, but hip pain can result from the IT band rubbing as it passes over the greater trochanter.

 

The medial thigh (groin) muscles include five muscles of adduction, and one lateral rotator (Figures 17, 18). The lone lateral rotator is the obturator externus, which covers the external surface of the obturator foramen in the deep upper medial thigh. The adductors include the gracilis, the pectineus, and the adductor brevis, longus and magnus. The gracilis is the longest adductor, extending from the medial inferior aspect of the pubic bone, to the medial aspect of the tibia. The adductor magnus is the most massive of the medial muscles of the thigh.

 

The tendons and muscles of the hips are very powerful and create great forces, making them prone to inflammation and irritation. Tendonitis of the hip can result from repetitive movements involving the soft tissues surrounding the hip joint. Overuse of the hip joint in fitness workouts can lead to tendonitis. Tendons lose their elasticity as we age, resulting in swelling and irritation when the tendons are no longer “gliding” on their normal paths. Iliopsoas tendonitis plays a major role in snapping hip syndrome, or dancer’s hip. A snapping sensation when the hip is flexed and extended may be accompanied by an audible snapping or popping noise, as well as pain. This can be both an extra-articular and an intra-articular occurrence. Extra-articular snapping is often found in those patients with a leg length difference (the longer leg is symptomatic), those with tightness of the iliotibial band on the involved side, and those with weak hip abductors and external rotators. Lateral extra-articular snapping can be caused by the iliotibial band, tensor fascia latae or gluteus medius tendon as they slide back and forth across the greater trochanter (Figure 19). If any of these connective tissue bands thickens, they can “catch” on the greater trochanter during the motion of hip extension, thereby creating the “snapping” sensation and sound. Medial extra-articular snapping, which is less common, can occur when the iliopsoas tendon catches on the anterior inferior iliac spine, lesser trochanter, or iliopectineal ridge during hip extension. Intra-articular snapping hip syndrome is similar in many ways to the extra-articular type, but often involves an underlying mechanical problem in the lower extremity, and more intense pain. Intra-articular snapping may be indicative of a torn acetabular labrum, recurrent hip subluxation, a tear of the ligamentum teres, loose bodies, articular cartilage damage, or synovial chondromatosis (cartilage formations in the synovial membrane of the joint). Snapping hip syndrome is usually found in those ages 15-40, often in those in training for the military. It can also affect athletes, especially those involved in dance, gymnastics, soccer, and track and field. These athletes will all be performing repeated hip flexions, which can lead to tendonitis in the hip area. The repetitive motions of those involved in weightlifting and running generally lead to a thickening of the tendons in the hip region, rather than snapping hip syndrome. Prevention, or at least a lessening, of this syndrome may be found with increased stretching of the iliopsosas muscle or the iliotibial band. Surgery is usually not required, unless intra-articular pathology is present.

 

Figure 19. Hip muscles.

 

Tendon or muscle strains can occur suddenly, as in sports injuries, or they can develop over time, with symptoms including pain, swelling, muscle spasms, and difficulty moving certain muscles. MRI can be used to detect tendon and muscle tears and strains, as well as bone tumors and infection. MRI has shown good accuracy for the diagnosis of tears of the gluteus medius and gluteus minimus tendons, which are both abductor tendons of the hip. An association was found between these tears and areas of high signal intensity superior or lateral to the greater trochanter on T2-weighted images, tendon elongation in the gluteus medius, and tendon discontinuity (Figure 20). STIR and fat-suppressed T2-weighted coronal images are very sensitive for detection of areas of high signal intensity superior to the greater trochanter. Coronal T1-weighted images demonstrate tendon elongation in the gluteus medius (Figure 21). Axial images may prove superior for localizing involvement to individual abductor tendons and confirming tendon discontinuity (Figure 22). Tears of the abductor tendons may be the leading cause of greater trochanteric pain syndrome.

 

Figure 20. Sag. T2 shows high signal intensity superior to greater trochanter (gt) corresponding to swollen bursa (*).

 

Figure 21. Coronal STIR shows high signal intensity superior to greater trochanter in bursa (*) between gluteus medius (me) and gluteus minimus (mi) tendons.

 

 Figure 22. Axial T2 shows high signal intensity corresponding to fluid replacing distal rt. gluteus medius tendon (black arrow); normal left tendon (white arrow).

Nerves Of The Hips

The nerves of the hip supply the various muscles in the hip area. The major nerves include the femoral, obturator, and lateral femoral cutaneous nerves anteriorly, and the large sciatic nerve posteriorly (Figure 23). The femoral nerve innervates the quadriceps femoris and sartorius, and is the sensory nerve to the anterior thigh. Trauma to this nerve usually occurs in the pelvis, as it passes through or near the psoas muscle. The obturator nerve passes along the lateral pelvic wall and through the obturator foramen, then splits into branches that supply the adductor muscle group. This nerve can also be subject to trauma in the pelvis due to its passage through the obturator foramen. The lateral femoral cutaneous nerve is a sensory nerve that travels along the anterolateral aspect of the thigh. It supplies sensation to the skin surface of the thigh. This is the single nerve involved in a painful condition called meralgia paresthetica, which is characterized by tingling, numbness, and burning pain in the outer part of the thigh. Meralgia paresthetica results from focal entrapment of the lateral femoral cutaneous nerve as it passes through the tunnel formed by the lateral attachment of the inguinal ligament and the ASIS. The posterior sciatic nerve passes deep to the gluteus maximus into the posterior thigh, where it innervates the hamstring muscles, on its way down to the lower leg and foot. The sciatic nerve is approximately as big around as the thumb, and is the largest single nerve in the human body. It can be injured in cases of posterior hip dislocation. Pressure on this nerve can cause nerve pain, numbness, tingling and weakness (sciatica symptoms) in the buttocks, leg, or foot, depending on the site of origin of the sciatic nerve compression.

 

Figure 23. Anterior and posterior views of the nerves of the hip.

Arteries & Veins Of The Hips

The arterial blood vessels that supply the hips are branches of the internal and external iliacs. The internal iliac artery gives off the superior and inferior gluteals, and the obturator artery. The inferior gluteal flows to the posterior aspect of the hip joint and proximal femur, where it joins a branch of the femoral artery. The obturator artery runs through the obturator foramen, and sends its acetabular branch to the ligamentum teres as part of the blood supply to the femoral head. The external iliac becomes the femoral artery, which has numerous branches that supply the hip and proximal femur. The largest femoral branch is the profunda femoris, which branches superiorly into the medial and lateral circumflex femorals (Figure 24). The circumflex femorals and the inferior gluteal artery contribute to the anastomoses to supply the femoral head, femoral neck, and the hip joint. The medial circumflex also has an acetabular branch to the ligamentum teres. Congenital anomalies in the hip anastomoses, degenerative processes, and trauma can all compromise the blood supply to the hip joint area.

 

Figure 24. Anterior and posterior views of the arteries of the hip.

 

Venous flow in the hip and proximal femur typically follows the arterial flow, including the same names for the vessels. The deep veins of the hip and thigh can be the origination of a deep vein thrombosis, which can result in a pulmonary embolus. This can be caused by immobility after hip surgery, sitting in cars or airplanes for extended trips, being overweight, or slow or low blood flow. These blood clots can break off, travel through the larger veins of the thigh and hip, continue through the heart, and become lodged in the smaller vessels of the lung. MRI is being used more frequently to diagnose this very serious condition.

Bursae Of The Hips

The hip joint is surrounded by bursae, similar to the shoulder. These fluid-filled sacs are lined with a synovial membrane, which produces synovial fluid. Their function is to lessen the friction between tendon and bone, ligament and bone, tendons and ligaments, and between muscles. There may be as many as 20 bursae around the hip. If they become infected or inflamed, the result is a painful condition called bursitis. Common hip bursae that may become inflamed include the greater trochanteric bursa, the iliopsoas bursa, and the ischial bursa (Figure 25). The greater trochanteric bursa is sandwiched between the greater trochanter of the femur, and the muscles and tendons that cross over it. If this bursal sac becomes inflamed, patients experience pain with every step they take, as each step requires the tendon to move over the femur at the hip joint. A tight iliotibial band can also cause irritation of the greater trochanteric bursa. Iliopsoas bursitis can result from irritation of the bursa found between the hip joint and the iliopsoas muscle that passes in front of it. Another common site for bursitis is the ischial bursa, which acts as a lubricating pad between tendons and the ischial tuberosity, which is the bony prominence of the pelvis that you sit on. The ischial bursa acts to prevent destruction of the tendons as they move over the ischial tuberosity. Prolonged sitting can cause ischial bursitis. Inflammation around the ischial tuberosity can irritate the sciatic nerve, and trigger symptoms similar to sciatica. Hip bursitis is seen in runners and athletes in sports that involve excessive running (soccer, football, etc.). It can also be caused by an injury (traumatic bursitis), and is seen in post-op hip replacement and hip surgery patients. Treatment for hip bursitis typically includes rest, anti-inflammatory medications, and ice. It may become necessary to aspirate the bursa, which can be combined with a cortisone injection. MRI may be needed if the diagnosis is unclear, or if the problem does not resolve with normal treatments.

 

Figure 25. Bursae of the hip.

Axial Scans

When positioning unilateral axial slices for the hip, a coronal image can be used to ensure inclusion of all pertinent anatomy. The slices should extend superiorly to include the entire femoral head and acetabulum, and inferiorly to include anatomy below the lesser trochanter. The slices should be aligned perpendicular to the shaft of the femur, as seen in the coronal image in Figure 39.

 

Figure 39. Axial slice setup using sagittal and coronal images.

 

For bilateral axial hip slice setup, parameters may have to be altered to maintain adequate resolution with the larger FOV that is required (Figure 40). The slice group may require angulation to maintain alignment of the femoral heads on the resultant images.

 

Figure 40. Bilateral axial slice setup using a coronal image.

Coronal Scans

Coronal slices of the hip should cover the area from the posterior margin to the anterior margin of the femoral head. The area from the proximal margin of the femoral shaft to the greater sciatic notch should be included in the image (Figure 41). Slices may be angled so that they are parallel to the femoral neck. Thinner slices may be requested for coronal scanning.

 

Figure 41. Coronal slice setup using axial and sagittal images.

Sagittal Scans

Sagittal slices of the hip should extend past the greater trochanter laterally, and through the acetabulum medially. The slices should be aligned along the long axis of the femur, and perpendicular to the coronal slices, as seen in the coronal image in Figure 42. Two different slice groups will be necessary when performing bilateral sagittal scans.

  

Figure 42. Sagittal slice setup using coronal and axial images.

Hips Arthrography

MR hip arthrography is often times referred to as the gold standard for assessment of the labrum of the hip. The most clinically significant abnormal findings that result from hip arthrography are labral detachments and tears. Detachment of the labrum, which is more common than a labral tear, can be diagnosed from the appearance of the injected contrast at the acetabular-labral interface (Figure 43). A labral tear can result in injected contrast appearing within the substance of the labrum (Figure 44). Contrast injection is necessary to differentiate torn or detached labra from other pathologic conditions, which may have separate signal intensities. The sensitivity and accuracy for the diagnosis of labral tears and detachment with MR arthrography vs. nonarthrographic MR is 90%. Hip arthrography with MR can also depict intrarticular loose bodies, osteochondral abnormalities, and abnormalities of soft-tissue structures.

 

Hip arthrography can be performed under fluoro in the x-ray dept., with the patient being moved to the MRI dept. for further imaging, or the entire procedure can be performed in the MRI suite, if MR compatible supplies are available for interventional techniques. The patient should be securely positioned with the hips in internal rotation.

 

T1-weighted imaging is performed post-contrast to visualize the high signal of the intraarticular contrast. T1 gradient echo sequences offer the benefits of thin sections, elimination of partial volume averaging, and increased detection of small tears. Fatsat sequences are helpful in increasing the contrast between the injected contrast and the adjacent soft tissue. STIR or fatsat T2 sequences performed in the coronal plane may help to detect unsuspected pathologic conditions in the soft tissue and adjacent osseous structures.

 

Post-contrast axial oblique images have been shown to optimize the detection of the most common sports-related acetabular labral tears, which are anterior or anterosuperior in location. Using a mid-coronal localizer, the axial oblique slices should be prescribed parallel to the long axis of the femoral neck.

  

Figure 43. Labral detachment as seen in a fat-suppressed T1-wtd. sag. image; arrowheads indicate involvement of anterior and anterosuperior labrum.

 

Figure 44. Labral tear as seen in a T1-wtd. image; arrowheads indicate enlarged labrum; short arrow indicates linear intralabral collection of contrast material; long arrow indicates communication between the joint and the iliopsoas bursa.

 

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Retrieved from http://www.eee.nuigalway.ie/documents/go_anatomy_of_the_plantar_venous_plexus_manuscript.pdf Morton’s neuroma. (Last modified 8August2012). Retrieved from http://en.wikipedia.org/wiki/Morton%27s_metatarsalgia

 

References Anatomy Pics:

 

Figures 1, 5, 6, 24- http://www.orthopediatrics.com/docs/Guides/perthes.html Figures 2, 3, 11, 12, 14, 15, 16, 18, 23, 25- http://www.activemotionphysio.ca/Injuries-Conditions/Hip/Hip-Anatomy/a~299/article.html Figure 4- http://hipkneeclinic.com/images/uploaded/hipanatomy_xray.jpg Figures 7, 8, 9- http://hipfai.com/ Figure 10- http://en.wikipedia.org/wiki/File:Ewing%27s_sarcoma_MRI_nci-vol-1832-300.jpg Figure 13- http://www.chiropractic-help.com/Patello-Femoral-Pain-Syndrome.html Figure 17- http://www.thestretchinghandbook.com/archives/ezine_images/adductor.jpg Figure 19- http://media.summitmedicalgroup.com/media/db/relayhealth-images/hipanat.jpg Figures 20-22- http://www.ajronline.org/content/182/1/137.full.pdf+html Figure 43, 44- http://radiographics.rsna.org/content/20/suppl_1/S43.full Figure 45- http://www.exploringnature.org/db/detail.php?dbID=24&detID=2768 Figures 46-48- http://www.ajronline.org/content/185/1/166.full.pdf Figure 49- http://arrs.org/shopARRS/products/s11p_sample.pdf Figure 50- http://www.thestretchinghandbook.com/archives/medial-collateral-ligament.php Figures 51, 52- http://www.radsource.us/clinic/0712 Figures 53, 54- http://www.osteo-path.co.uk/BodyMap/Thighs.html Figure 55- http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1963576/ Figure 56- http://legacy.owensboro.kctcs.edu/gcaplan/anat/Notes/API%20Notes%20M%20%20Peripheral%20Nerves.htm Figure 57- http://www.keywordpictures.com/keyword/lateral%20cutaneous%20nerve%20of%20thigh/ Figure 58- http://home.comcast.net/~wnor/postthigh.htm Figure 59- http://becomehealthynow.com/glossary/CONG437.htm Figure 60- http://fitsweb.uchc.edu/student/selectives/Luzietti/Vascular_pvd.htm Figure 61- http://www.fashion-res.com/peripheral-vascular-disease-with-stenting-in-the/ Figure 62- http://www.wpclipart.com/medical/anatomy/blood/femoral_artery_and_branches_in_leg.png.html Figure 63- http://www.globalteleradiologyservices.com/Deep_Vein_Thrombosis_Overview.htm Figure 64- http://www.vascularultrasound.net/vascular-anatomy/veins/lower-extremity-veins Figure 82- http://www.jeffersonhospital.org/diseases-conditions/knee-ligament-injury.aspx?disease=658f267f-75ab-4bde-8781-f2730fafa958 Figure 83- http://javierjuan.ifunnyblog.com/anatomybackofknee/ Figure 84- http://www.kneeandshouldersurgery.com/knee-disorders/tibial-osteotomy.html Figure 85- http://www.disease-picture.com/chondromalacia-patella-physical-therapy/ Figure 86- http://www.eorthopod.com/content/bipartite-patella Figure 87- http://www.orthogate.org/patient-education/knee/articular-cartilage-problems-of-the-knee.html Figure 88- http://www.webmd.com/pain-management/knee-pain/menisci-of-the-knee-joint Figure 89- http://sumerdoc.blogspot.com/2008_07_01_archive.html Figure 90- http://www.concordortho.com/patient-education/topic-detail-popup.aspx?topicID=55befba2d440dc8e25b85747107b5be0 Figure 91- http://trialx.com/curebyte/2011/08/16/pictures-for-chondromalacia-patella/ Figure 92- http://radiopaedia.org/images/1059 Figure 93- http://radiologycases.blogspot.com/2011/01/osgood-schlatter-disease.html Figure 94- http://www.physioquestions.com/2010/09/07/knee-injury-acl-part-i/ Figure 95- http://www.jeffersonhospital.org/diseases-conditions/knee-ligament-injury.aspx?disease=4e3fcaf5-0145-43ea-820f-a175e586e3c8 Figures 96, 97- http://radiology.rsna.org/content/213/1/213.full Figures 98-101- http://appliedradiology.com/Issues/2008/12/Articles/Imaging-the-knee--Ligaments.aspx Figure 102- http://radiopaedia.org/images/408156 Figure 103- http://aftabphysio.blogspot.com/2010/08/joints-of-lower-limb.html Figures 104, 105- http://www.radsource.us/clinic/0310 Figure 106- http://nwrunninglab.com/patellar-tendonitis.html Figure 107- http://www.aafp.org/afp/2007/0115/p194.html Figure 108- http://www.reboundsportspt.com/blog/tag/knee-pain Figure 109- http://www.norwellphysicaltherapy.com/Injuries-Conditions/Knee/Knee-Issues/Quadriceps-Tendonitis-of-the-Knee/a~1803/article.html Figure 110- http://kneeguru.co.uk/KNEEnotes/node/479 Figure 111- http://www.magicalrobot.org/BeingHuman/2010/03/fascia-bones-and-muscles Figure 112- http://home.comcast.net/~wnor/postthigh.htm Figures 113, 115, 157-159- http://ipodiatry.blogspot.com/2010/02/anatomy-of-foot-and-ankle_26.html Figure 114- http://medchrome.com/basic-science/anatomy/the-knee-joint/ Figure 116- http://www.sharecare.com/question/what-are-varicose-veins Figure 117- http://mendmyknee.com/knee-and-patella-injuries/anatomy-of-the-knee.php Figures 118-120- http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3177464/ Figure 121- http://www.riversideonline.com/health_reference/Disease-Conditions/DS00448.cfm Figure 122- http://arthritis.ygoy.com/2011/01/01/what-is-an-arthritis-knee-cyst/ Figure 143- http://usi.edu/science/biology/mkhopper/hopper/BIOL2401/LABUNIT2/LabEx11week6/tibiaFibulaAnswer.htm Figure 144- http://web.donga.ac.kr/ksyoo/department/education/grossanatomy/doc/html/fibula1.html Figure 145- http://becomehealthynow.com/popups/ligaments_tib_fib_bh.htm Figure 146- http://www.parkwayphysiotherapy.ca/article.php?aid=121 Figure 147- http://aidmyankle.com/high-ankle-sprains.php Figure 148- http://legsonfire.wordpress.com/what-is-compartment-syndrome/ Figures 149, 152- http://www.stepbystepfootcare.ca/anatomy.html Figures 150, 151- http://www.gla.ac.uk/ibls/US/fab/tutorial/anatomy/jiet.html Figure 153- http://www.athletictapeinfo.com/?s=tennis+leg Figure 154- http://radsource.us/clinic/0608 Figure 155- http://www.eorthopod.com/content/achilles-tendon-problems Figure 156- http://achillesblog.com/assumptiondenied/not-a-rupture/ Figure 181- http://www.orthopaedicclinic.com.sg/ankle/a-patients-guide-to-ankle-anatomy/ Figure 182- http://www.activemotionphysio.ca/article.php?aid=47 Figure 183- http://www.ajronline.org/content/193/3/687.full Figures 184, 186- http://www.eorthopod.com/content/ankle-anatomy Figure 185- http://www.crossfitsouthbay.com/physical-therapy/learn-yourself-a-quick-anatomy-reference/ankle/ Figures 187, 227- http://www.activemotionphysio.ca/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html Figure 188- http://inmotiontherapy.com/article.php?aid=124 Figures 189, 190- http://home.comcast.net/~wnor/ankle.htm Figure 191- http://skillbuilders.patientsites.com/Injuries-Conditions/Ankle/Ankle-Anatomy/a~47/article.html Figure 192- http://metrosportsmed.patientsites.com/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html Figure 193- http://musc.edu/intrad/AtlasofVascularAnatomy/images/CHAP22FIG30.jpg Figure 194- http://musc.edu/intrad/AtlasofVascularAnatomy/images/CHAP22FIG31B.jpg Figure 195- http://veinclinics.com/physicians/appearance-of-vein-disease/ Figure 196- http://mdigradiology.com/services/interventional-services/varicose-veins.php Figure 216- http://kidport.com/RefLib/Science/HumanBody/SkeletalSystem/Foot.htm Figure 217- http://www.joint-pain-expert.net/foot-anatomy.html Figure 218- http://www.thetoedoctor.com/turf-toe-symptoms-and-treatment/ Figures 219, 220- http://radsource.us/clinic/0303 Figure 221- http://www.ajronline.org/content/184/5/1481.full Figure 222- http://www.answers.com/topic/arches Figure 223- http://www.mayoclinic.com/health/medical/IM00939 Figure 224- http://radsource.us/clinic/0904 Figure 225- http://www.ortho-worldwide.com/anfobi.html Figure 226- http://www.coringroup.com/lars_ligaments/patientscaregivers/your_anatomy/foot_and_ankle_anatomy/ Figure 228- http://www.stepbystepfootcare.ca/anatomy.html Figure 229- http://iupucbio2.iupui.edu/anatomy/images/Chapt11/FG11_18aL.jpg Figure 230- http://www.ajronline.org/content/184/5/1481.full.pdf Figure 231- http://metrosportsmed.patientsites.com/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html Figure 232- http://www.painfreefeet.com/nerve-entrapments-of-the-leg-and-foot.html Figures 233, 234- http://emedicine.medscape.com/article/401417-overview Figure 235- http://web.squ.edu.om/med-Lib/MED_CD/E_CDs/anesthesia/site/content/v03/030676r00.HTM Figure 236- http://www.nysora.com/peripheral_nerve_blocks/classic_block_tecniques/3035-ankle_block.html Figure 237- http://ultrasoundvillage.net/imagelibrary/cases/?id=122&media=464&testyourself=0 Figure 238- http://www.joint-pain-expert.net/foot-anatomy.html Figure 239- http://jap.physiology.org/content/109/4/1045.full Figure 240- http://microsurgeon.org/secondtoe Figure 241- http://elu.sgul.ac.uk/rehash/guest/scorm/406/package/content/common_iliac_veins.htm

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Hips: healthy hip joints support the body's weight and allow for pain-free movement. Hip injuries or disease can cause changes that affect the gait, as well as changes that affect the ability of the hips to distribute weight. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

Sciencekeys's comment, July 28, 2020 10:24 AM
https://sciencekeys.com/nerves-of-the-abdomen-lower-back-and-pelvis/
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Sprains And Strains 3 Differences: Chiropractic Can Help | Back Clinic ®

Sprains And Strains 3 Differences: Chiropractic Can Help | Back Clinic ® | Mobility & Flexibility - Joint Movement | Scoop.it

As experienced chiropractors, we like answering the questions we receive from our patients. A common inquiry is "what is the difference between a sprain and a strain?" Sprains and strains are injuries to the musculoskeletal system that are commonly diagnosed conditions, and are two separate issues people frequently mix up. We will attempt to explain away some of the confusion today.

 

Let's look at three ways sprains and strains differ from each other.

1. Sprains & Strains Afflict Different Parts Of The Body.

According to the National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIH), a sprain is a stretch or tear of a ligament which provides joint stability. A strain is a stretch or tear of a muscle or tendon in the area where it is turning into a muscle.

2. Sprains & Strains Are Most Often Caused From Different Actions.

Falling or twisting the wrong way typically causes a sprain, because the movement forces a joint into an awkward position and ends up stretching or tearing the ligament. Twisting an ankle, falling down the stairs, or trying to catch yourself on an icy walkway are all ways to end up with a sprain.

 

A strain often results from overexertion or trauma, and repetitive movement. Lifting an item that is too heavy, jumping into an exercise routine that is too strenuous, or performing repetitive movements in either a sport or work are ways an individual can end up suffering from a strain.

3. Sprains & Strains Generally Affect Different Areas Of The Body.

Sprains occur at parts of the body that are injured when falling or suddenly twisting. According to the U.S. National Library of Medicine, ankle sprains alone number around 2 million each year.

 

Ankles, wrists, knees, and fingers are all areas that are frequently sprained. Strains, on the other hand, commonly occur in the back, shoulder, or hamstring, as both of these areas are affected by overexertion or repetitive movement.

 

Although sprains and strains are different injuries, they do have some similarities. This is most likely why people get them mixed up.

Let's discuss a few commonalities of sprains and strains.

Both share common symptoms.  Both injuries can bring on pain, swelling, and limited movement at the injury site. The pain can be moderate or intense, depending on the severity of the injury. Sprains and strains both benefit from ice packs, rest, and elevation.

 

They can require surgery.  Most diagnosed strains and sprains heal on their own with time, but a serious tear can require surgery to repair. With both injuries, it's important to visit a doctor if an individual experiences severe pain and swelling, and decreased mobility.

 

Both can benefit from chiropractic care.  Chiropractors can work wonders on the neck and back, but chiropractic care can assist in lessening the impact of a strain or sprain injury, too.

 

The benefits of seeing a chiropractor for both sprains and sprains are twofold. Chiropractic treatments promote healing of the injured area as well as help strengthen the areas around the injury to decrease the chance of future injuries.

 

These types of injuries can sideline individuals from their activities, no matter if they are athletes or regular guys doing yard work. It's vital to take steps to avoid sprains and strains in the first place.

 

Always properly stretch and avoid overexertion to prevent strains. Take pains to clear walkways and stairways to avoid falls or sudden twisting movements to decrease the risk of sprains.

Washington Cheerleader Talks Chiropractic

If you end up with a strain or sprain, contact us for a consultation. We have extensive experience in working with patients suffering from sprains and strains.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Sprains and strains are injuries to the musculoskeletal system that are commonly diagnosed conditions, and are two separate issues people frequently mix up. We will attempt to explain away some of the confusion today. Contact us for a consultation. We have extensive experience. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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December 12, 2017 6:30 PM
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Suffer From Arthritis: Chiropractic Can Help | El Paso Back Clinic ®

Suffer From Arthritis: Chiropractic Can Help | El Paso Back Clinic ® | Mobility & Flexibility - Joint Movement | Scoop.it

Even though chiropractic excels in wellness care, it is becoming more common for people to visit chiropractors to treat a variety of different kinds of pain. Because of this, chiropractic adjustments provide many benefits to people and patients who suffer from a wide variety of conditions like arthritis. In today’s article, we’ll explore how chiropractic can help patients who suffer from arthritis and give you additional suggestions on how to alleviate the pain that’s associated with it.

Arthritis: What a Chiropractor Does

A Doctor of Chiropractic, also known as a chiropractor, is a health professional that focuses primarily on wellness care instead of sickness care. Their specialty focuses on adjusting the spine to correct misalignments that may be impinging on nerves.

 

Regular visits to a chiropractor can not only restore health throughout the body but also help alleviate back pain and other symptoms associated with an improperly aligned spinal column. They can also work with their patients to plan exercise routines and alterations in diet to assist management of inflammation and pain. Most insurance carriers cover visits to a chiropractor on at least some level.

What Is Arthritis?

Put simply, arthritis is inflammation in the joints which result in joint pain, stiffness and limited range of movement. There are over 200 different varieties of the ailment. While it is generally associated with age, it can also affect young people. It can strike almost any area of the body, with each region having a different cause and name. In some cases, can cause damage to soft tissues and muscles, like the heart and lungs.

 

Osteoarthritis, also called degenerative joint disease, is the most common type of arthritis. It results from repeated trauma to the joint and becomes more common in the elderly.

 

Other common forms of include:

  • Rheumatoid arthritis, the second most common form in which the body’s immune system attacks the joint.
  • Psoriatic arthritis, another autoimmune form.
  • Ankylosing spondylitis, also a type where the body attacks itself.
  • Septic arthritis, which is caused by a viral or bacterial infection of the joint.

Diagnosis

Diagnosing arthritis involves a complete and thorough examination. If a chiropractor feels the need to co-manage the case, a medical work-up by a rheumatologist may be recommended. This can include radiology (x-ray) or an MRI, urine and blood analysis and physical examinations.

 

It is important to have your condition properly diagnosed so you can more effectively treat the symptoms of the disease.

Chiropractors and Arthritis

The most common treatment for arthritis is medication, which can take down the inflammation and swelling and reduce pain. However, chiropractors can be of great help in managing arthritis. While medications work, it has long-term health risks such as impairing healing, damage to the stomach lining and internal bleeding.

 

By visiting a chiropractor you may be able to reduce your reliance upon these medications while managing your pain and symptoms naturally. A chiropractor can:

  • Improve your range of motion by keeping your spine in line
  • Improve endurance and flexibility
  • Increase your strength and muscle tone
  • Help you develop a dietary and nutritional plan to reduce inflammation naturally

In addition, chiropractors can recommend an exercise regimen that’s conducive to arthritis. According to the American Chiropractic Association, this is a vital component in managing your arthritis symptoms.

Treating the Symptoms

Please understand that chiropractors cannot cure arthritis. At this time, there is no cure for this ailment. They can, however, help to alleviate the symptoms and slow the progression of the illness. They may use spinal adjustments in conjunction with other treatments to address the disease. These options can include:

  • Hot and cold therapy
  • Ultrasound treatments
  • Massage
  • Electronic muscle stimulation
  • Physical rehabilitation
  • Magnet therapy

The Best Results

With an inflammatory disease like arthritis, the best results are achieved from attacking it at all angles. This means working with your chiropractor and rheumatologist to combine treatments, if necessary. In addition to their care, a healthy diet and active exercise regime will help move you in the right direction toward a healthier outcome.

 

If you or a loved one are suffering with, don’t hesitate to give us a call today. We’re here to help in any way we can!

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

El Paso, TX. Arthritis, a chiropractor may be able to reduce your reliance upon medications while managing your pain and symptoms naturally. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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October 19, 2017 9:13 PM
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Yoga Standing Poses | Doctor Alexander Jimenez D.C.

Yoga Standing Poses | Doctor Alexander Jimenez D.C. | Mobility & Flexibility - Joint Movement | Scoop.it

 By Kyran Doyle In Yoga

 

Yoga is a great way to improve flexibility and strength in your body. In this article we will go over some standing poses to use in your practice.

YOGA: MOUNTAIN POSE (TADASANA)

The mountain pose is the foundation of all standing poses. It might not look like much but the mountain pose is an important starting position, resting pose and tool to improve posture which leads to many other standing poses in yoga.

STANDING FORWARD BEND (UTTANASANA)

Standing forward bend is a smooth transition from mountain pose and you will find a deep stretch in the entire back body.

WARRIOR I POSE (VIRABHADRASANA I)

There are three variations of the warrior pose of which this is number I.

WARRIOR II (VIRABHADRASANA II)

Warrior II stretches and strengthens the body in the one movement, allowing you to feel like a strong warrior. This pose will build strength your ankles, legs, glutes, core, back and shoulders.

REVERSE WARRIOR (VIPARITA VIRABHADRASANA)

Reverse Warrior is a variation of the warrior II pose that provide a great stretch in the side body.

 

 

 

Gymnastics & Chiropractic

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

Yoga is a great way to improve flexibility and strength in your body. We will go over some standing yoga poses to use in your practice. For Answers to any questions you may have please call Dr. Jimenez at 915-850-090

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May 9, 2017 5:56 PM
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Tunnel Vision: Medial Nerve Entrapment & Elbow Pain | El Paso Back Clinic® • 915-850-0900

Tunnel Vision: Medial Nerve Entrapment & Elbow Pain | El Paso Back Clinic® • 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it


Doctor of Chiropractic, Dr. Alex Jimenez looks at the etiology and clinical presentation of cubital tunnel syndrome, and also provides rehabilitation guidelines.


Cubital Tunnel Syndrome

Medial elbow pain is a commonly reported in throwing athletes. Due to the high valgus stress put on the elbow during the throwing motion, as many as 69% of baseball players under the age of 19 report pain in this region(1). Diagnosis can be complex and the clinician needs to consider a wide variety of pathologies in this region including medial collateral ligament (MCL) strain or rupture, flexor pronator tendon strain, posteromedial impingement and cubital tunnel syndrome – to name a but few.


Cubital tunnel syndrome is the second most common nerve entrapment of the upper limb and occurs as a result of compression or ischemia of the ulnar nerve as it passes through the cubital tunnel (see Figure 1)(2).
 
Table 1: Results Of Investigations

 

  • Investigation Summary of results
  • X-ray No abnormality detected
  • MRI  Accessory anconeus epitrochlearis muscle may be causing impingement of the ulnar nerve. No focal thickening or increased signal seen in the ulnar nerve.
  • Nerve conduction test NAD


The presence of an anconeus epitrochlearis muscle has been described as a ‘rare but possible’ cause of cubital tunnel syndrome(3-5). In this article, we will look at cubital tunnel syndrome by considering a case of compression of the ulnar nerve by the anconeus epitrochlearis muscle in a female national level water polo player.


Clinical Presentation


A 20-year old, right handed female water polo player presented with right medial elbow pain. She reported a 12-month
history of intermittent elbow pain and burning, as well as occasional paraesthesia into her 4th and 5th digit. She could not recall any incident/trauma to her elbow. These symptoms would be worse after national camps or tours of several weeks duration (daily water based training or games), and she would require treatment in the later stages of these tours. However, once she returned to state/club training (when water-based training was not as regular) her symptoms would resolve. Previous physiotherapy treatment had involved cervical spine mobilisation, neural mobilisation and soft tissue work. Previously these treatments had resolved her symptoms completely and she was able to return to a full training or playing load.


Gradually over the course of the year however, her symptoms were becoming more regular and she was starting to get intermittent symptoms at state training and occasionally during activities of daily living (leaning on elbow). Recently she had taken three months off training (due to the removal of a benign lump in her breast).

Table 2: Post-Operative Rehab Plan

 

  • Week post op Water polo based return to play timeline (key milestones)
  • Week 0-2 Cast (back slab)at 90deg flexion
  • Week 2+ Commenced active ROM and nerve sliders
  • Week 4 Full extension (lacked 20deg elbow F) Was able to commence light swimming starting at 600m and progress distance to 2km over two weeks
  • Week 7 Was advised by surgeon that could commence light passing however some elbow pain returned following a moderate swim session so that delayed passing
  • Week 8 Returned to swimming with no pain
  • Week 9 Commenced light passing; 20 passes of 5 metres
  • Weeks 10-12 Elbow pain reoccurred from passing so was advised rest from passing until clinical signs and symptoms settled. Able to continue swimming
  • Week 13 Recommenced passing 20 x 5m – 3 times per week
  • Week 16 Commenced light shooting 20 shots at open goal 3 times per week
  • Week 17 Shooting into goal with goalie and light wrestling in water
  • Week 18 Full training
  • Week 20 Returned to play domestic competition

 

Physiotherapy Treatment During This Process Involved:

  • Neural sliders with a ulnar nerve bias (see Figure 3)
  • Lower cervical and upper thoracic mobilisation
  • Soft tissue work throughout scalenes, pec minor and periscapular muscles


Load was gradually progressed dependant on clinical signs and symptoms. Symptoms were monitored via subjective questioning (pain) and clinical signs were monitored via elbow range of motion and brachial plexus provocation testing with ulnar nerve bias. If signs or symptoms worsened then training was decreased until baseline was again reached.

However, unlike previous episodes – where rest would relieve her symptoms – her symptoms during this time had increased. During this time she had also commenced studying physiotherapy and her study load had increased. This included a practical component which had recently involved neural mobilisation techniques which she felt aggravated her pain. She now had pain leaning on her elbow, and felt that her grip strength had decreased, especially when using a pen or holding cup. She also reported almost constant paresthesia into
her 5th digit and ulna side of her 4th digit.

 

Objective Assessment


An objective assessment revealed the following:

  • Minor wasting of the hypothenar muscles of her right hand
  • Cervical spine – slightly limited ROM and no reproductionof elbow symptoms Elbow – full ROM extensionand flexion Valgus stress test of the elbow – no pain or laxity
  • Ulnar nerve Tinels test – +ve
  • ULNTT(ulnarnervebias) –verylimited++
  • Sensation– mildly reduced light touch sensationof 5th digit and medial aspect of 4th digit (9/10 sensation)


At this point she was referred to sports physician who arranged investigations. The results are shownin Table 1.
Following these results, she was referred to an orthopaedic surgeon for their opinion. Considering the worsening of symptoms and the lack of improvement from conservative treatment, surgery was advised. Surgery was performed a week later and the anconeus epitrochlearis muscle was excised. Intraoperatively, the surgeon assessed the stability of the nerve (once the muscle was removed) and he felt that it was stable, so no nerve transposition was required. There then followed a post- operative rehabilitation plan in preparation for returning to water polo. This is outlined in Table 2.


Discussion


Cubital tunnel syndrome is a well- recognised compression neuropathy of the upper extremity, which causes motor and
sensory symptoms in the distribution of the ulnar nerve (see Table 3). As a result of this compression, patients will complain of paraesthesia or anaesthesia of the 4th (medial half) and 5th digit, as well as weakness of the hand (especially performing fine motor tasks). Wasting of the muscles of the hypothenar eminence may also be observed(6).


Cubital tunnel syndrome can be caused by a variety of space occupying lesions including the presence of an anconeus epitrochlearis muscle(3). The anconeus epitrochlearis is a congenital accessory muscle with a prevalence of between 4 and 34% in cadaver studies(3). It exists bilaterally in 25% of patients with cubital tunnel syndrome(1) and lies superficial to the ulnar nerve, attaching from the inferior surface of the medial epicondyle to the medial cortex of the olecranon (see figure 5)(2).
Anconeus epitrochlearis runs a similar anatomical course as the cubital tunnel retinaculum. This muscle becomes taut in flexion as well as assisting the triceps muscles in preventing ulnar nerve subluxation(4).


Table 3: Ulnar Nerve Innervations, Distal To Elbow

 

  • Motor function: Muscles of the hand (apart from thenar muscles and the lateral two lumbricals), as well as flexor carpi ulnaris and medial half of flexor digitorum profundus.
  • Sensory function: Anterior and posterior aspect 5th digit and ulnar side of 4th digit and associated palm area.

 

  • Decompressive procedures (with or without a medial epicondylectomy)
  • Ulnar nerve transpositionprocedures (subcutaneous, intramuscular or submuscular depending onwhere the nerve is placed)


The anconeus epitrochlearis muscle has been reported as the cause of ulnar nerve compression in 3-16% of patients with cubital tunnel syndrome in operative studies(5). Bagatur et al reported that when compared with idiopathic cubital tunnel syndrome, anconeus epitrochlearis associated ulnar neuropathy occurs at a younger age and has a more rapid progress. Also, in these patients, medial elbow symptoms are more pronounced than sensory symptoms(3). Oedema of the anconeus epitrochlearis muscle has also been noted as a feature on MRI scans(1); however this was not present in this patient.
The aim of treatment of ulnar nerve entrapment should be based not only on the relief of symptoms, but also to prevent further neurological damage.


Conservative measures should always be the first line of treatment for cubital tunnel syndrome. Conservative treatment should include activity modification (throwing technique), avoidance of local pressure (leaning on elbow), avoidance of prolonged elbow flexion (narrowing of the tunnel), neural sliders and interface mobilisation/soft tissue work. Medication for the neurological pain sensation (anti- depressives and anti-epileptics) may also be considered. Usually these methods should be trialed over a 6-month period. However, if these measures fail or neurological symptoms are worsening, then operative treatment has been shown to have good outcome(1).
The surgical treatment of cubital tunnel syndrome can be divided into two broad categories(7):


In cases where compression of the ulnar nerve is caused by the presence of an anconeus epitrochlearis muscle, surgical intervention has been described as either excision of the muscle or (if the nerve is determined to be unstable once the muscle has been removed) excision plus anterior transpositionof the ulnar nerve(7).


Several cases surgical excision of the anconeus epitrochlearis muscle have been reported in the literature, and all of these show full resolution of symptoms and a good functional and clinical outcome(7). No previous case could be found in an elite- level water polo player. Morgenstein et al describe a case report of a elite baseball pitcher who returned to full velocity pitching at 3 months post anconeus epitrochlearis excision and ulnar nerve decompression(6). Li et al also described 3 baseball pitchers who had excision of anconeus epictrochlearis (1 of which also had medial ulnar collateral ligament reconstruction); all of these returned to their pre-injury status withinone year(1).


Conclusion


Diagnosis of medial elbow pain can be complex in an overhead athlete. The anconeus epitrochlearis muscle should be considered as a potential cause of symptoms in patients with cubital tunnel symptoms. In patients with neurological symptoms who have tried conservative treatment surgical, excision of the anconeus epitrochlearis is warranted. Several case studies show that throwing athletes have all returned to pre-injury level at one year post treatment. In this case the player returned to domestic waterpolo competitionafter five months.


References
     1. Orthopaedics 2012. Vol 35 No 7 e1129 – e1132
     2. Hand Surgery 2012. Vol 17, No1 1-2
     3. Orthopedics 2016. Vol 39 (5) e988-e991
     4. Journal of Shoulder and Elbow Surgery 2009. Vol 18 e21-e23
     5. Turkish Neurosurgery 2014. Vol 24 No 2 266- 271
     6. Journal of Hand Surgery 2014. Vol 41 (2) 227- 228
     7.Clinical Rheumatology 2012. 31: 1139-1142

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

Medial elbow pain is a commonly reported in throwing athletes. As many as 69% of baseball players under the age of 19 report pain in the area. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900

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The Impact of Exercise on Brain Health

The Impact of Exercise on Brain Health | Mobility & Flexibility - Joint Movement | Scoop.it

Can a new exercise regimen boost your brain health if you’re over 50?

 

Possibly, suggests a new research review that found middle-age folks can improve their thinking and memory skills by adopting regular moderate-to-vigorous routines involving aerobic and resistance exercise.

 

“When we combined the available data from [39 previous] studies, we were able to show that undertaking physical exercise was able to improve the brain function of people aged 50 and over,” said study lead author Joseph Northey. He’s a doctoral candidate and teaching fellow at the University of Canberra Research Institute for Sport and Exercise in Australia.

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

As people age, it's important for them to continue exercising in order to maintain overall health and wellness. While physical activity in older individuals can be beneficial, new studies suggest exercise can help with memory. For more information, please feel free to ask Dr. Jimenez or contact us at (915) 850-0900.

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Scooped by Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP
April 17, 2017 7:50 PM
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Taking Advil For Joint Pain Can Actually Make It Worse Call 915-850-0900

Taking Advil For Joint Pain Can Actually Make It Worse Call 915-850-0900 | Mobility & Flexibility - Joint Movement | Scoop.it


El Paso TX. Chiropractor Dr. Alex Jimenez takes a look at medication for joint pain and how they can make the pain worse.


Non-steroidal anti-inflammatory drugs (NSAIDs) are as common as candy, a staple of every home medicine cabinet and tossed casually in desk drawers, purses, and briefcases. Many people take these drugs, which include ibuprofen (sold as Motrin and Advil), naproxen (Aleve), and aspirin, at the first sign of a headache or muscle cramps — and they are a daily ritual for many people living with arthritis.

But few people realize that NSAIDs carry a black-box warning, the strictest warning issued by the Food and Drug Administration. “Most people think that the government or FDA would not allow something dangerous on the market, especially since most of them are over-the-counter and [used] without a prescription,” says integrative medicine expert Sunil Pai, MD, author of An Inflammation Nation. “A black-box warning is the FDA’s attempt to let you know that you can end up in a casket if you are unlucky enough to suffer one of a medication’s serious reactions.”

Not only have NSAIDs been linked to a slew of serious side effects, including ulcers, hearing loss, allergic reactions and miscarriages, but they can actually worsen some of the conditions, such as arthritis, they are supposed to help.

“The scientific literature makes it abundantly clear that NSAIDs…have a significant negative effect on cartilage,” which accelerates the deterioration of arthritic joints, says Pai. “NSAIDs have no beneficial effect on [cartilage] and speed up the very disease for which they are most used and prescribed.”

Even worse, NSAIDs do not address the underlying conditions that cause pain and inflammation, such as a leaky gut, and can even exacerbate them. Stress, infections, alcohol, and a poor diet can all irritate the gut lining and lead to a leaky gut, but so can NSAIDs.

“If you use a full therapeutic dose of NSAIDs for two weeks, there is a 75 percent chance you will develop a leaky gut that doesn’t go away when you stop taking the drug, Leo Galland, MD, tellsExperience Life magazine.

6 SIMPLE DIETARY INTERVENTIONS TO FIGHT & HEAL A LEAKY GUT


So, how can people with acute or chronic inflammatory conditions fight pain naturally? Some simple dietary interventions go a long way towards fighting inflammation and healing a leaky gut.

1. TRY AN ELIMINATION DIET

Removing common foods that can irritate the gut, including gluten, sugar, dairy, processed foods and soy, can jumpstart the healing process. Sugar (and refined grains, which turn to sugar in the body), for example, is one of the single biggest drivers of inflammation and its downstream consequences.

When sugar cravings strike, try roasting root vegetables or sweet potatoes. Roasting concentrates the natural sweetness of the plant, but the fiber slows down sugar absorption in the bloodstream.

2. EAT WHOLE FOODS

Michael Pollen’s recommendation – “Eat food. Not too much. Mostly plants.” – Is great advice when it comes to naturally fighting inflammation. Eating a Standard American Diet (SAD) — high in processed foods, unhealthy fats, and sugars — is like pouring kerosene on inflammation’s fire. Eating whole foods, rich in phytonutrients, helps put out that fire.

One fun way to eat more plants? Strive to “eat the rainbow,” or get at least one whole food from all the different colors of the rainbow each day:

  • Red (pomegranates, strawberries, tomatoes)
  • Orange (sweet potatoes, carrots)
  • Yellow (lemon, squash)
  • Green (avocado, Brussels sprouts, green tea)
  • Blue/purple (berries, olives)
  • White/tan/brown (garlic, onion, mushrooms).

 

Animal protein doesn’t need to be avoided if it’s grass-fed and pastured. Instead, try to reverse the ratio on your dinner plate: Make meat the side dish and vegetables the main course.

3. SUPPLEMENT WITH GLUTAMINE

Glutamine helps heal your gut by fueling the cells in your gut lining. You could think of it as a leaky gut superhero. “Glutamine heals the intestinal lining more than any other nutrient,” Liz Lipski, Ph.D., CCN, author of Digestive Wellness, tells Experience Life.

4. GET YOUR OMEGA-3S

Omega-3 fatty acids are natural inflammation fighters. Good whole food sources of omega-3s include wild-caught fish, grass-fed meat, pastured eggs, algae, and seeds such as hemp, chia, and flax. A high-quality omega-3 supplement is also worth considering. Even on a largely whole-foods-based, it can be hard to get the recommended daily amount of omega-3s.

5. DRINK BONE BROTH

Bone broth is one of the best natural sources of collagen, a protein found in abundance in our ligaments, tendons, bones, and skin. The collagen in broth is easily absorbed by our tissues and can not only help promote healthier connective tissue and ease joint pain, but it can also help heal a leaky gut. The best bone broth is homemade but increasingly high-quality bone broth is available for purchase at cooperatives and health food stores.

6. CONSIDER BOTANICAL FIRST AID

Many plants are powerful inflammation fighters. Turmeric may be the best known and most studied. Recent research suggests that the active ingredient in turmeric (called curcumin) has anti-inflammatory, antioxidant, antiviral, antibacterial, antifungal, and anticancer activities on par with commonly prescribed arthritis drugs like Enbrel and Humira.

A lot of other plants and plant compounds show similar activity in the body, including ginger, bromelain (an enzyme found in pineapple), capsaicin (the active ingredient in hot peppers), and ginger. Consult your healthcare practitioner before taking botanical supplements.

SOURCE:

http://www.drfranklipman.com/problem-nsaids-yes-mean-advil/

Dr. Alex Jimenez DC, APRN, FNP, IFMCP, CFMP's insight:

Many people take pain medication, which include ibuprofen (sold as Motrin and Advil), naproxen (Aleve), and aspirin. For Answers to any questions you may have please call Dr. Jimenez at 915-850-0900 

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Yoga Can Provide Relief from Back Pain

Yoga Can Provide Relief from Back Pain | Mobility & Flexibility - Joint Movement | Scoop.it

For people experiencing low back pain, the thought of exercise often seems daunting. But yoga may be a natural fit in the quest to relieve an aching back, a new review indicates.


The findings come from an analysis of 12 studies that included more than 1,000 participants with lower back pain. The studies compared yoga to physical therapy or patient education.


There was some evidence that yoga led to small improvements in pain, and small to moderate improvements in back function at three and six months.


“We found that the practice of yoga was linked to pain relief and improvement in function,” said review author L. Susan Wieland. She is an assistant professor of family and community medicine at the University of Maryland.


“For some patients suffering from chronic non-specific low back pain, yoga may be worth considering as a form of treatment,” Wieland added in a university news release.

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