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Data from randomized trials support the ability of co-enzyme Q10 (CoQ10) to improve heart function in people with congestive heart failure (CHF), say scientists from Tulane University in New Orleans.
The authors caution that the results are based on a small number of clinical trials, and that the benefit may be greatest for less severe cases of heart failure. 
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A new understanding of the immune system may be paving the way for the development of a vaccine for heart disease.
“If successful, a tolerogenic vaccine could stop the inflammation component of heart disease,” [Dr. Klaus] stated. “This could probably be used in conjunction with the statins (cholesterol-lowering drugs) that have already taken a significant chunk out of the numbers of people with heart disease. Together, they could deliver a nice one-two punch that could be important in further reducing heart disease.”
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It is commonly understood that when a parent has died of heart disease at a young age (under 60), the offspring have a higher risk of having heart disease.
A recent study, which examined data on millions of people in Denmark over a period of 30 years, confirms this theory. In fact, the risk is also increased for individuals with a second-degree relative (grandparent, half-sibling) who died young.
And the risk can be compounded-- when two or more first-degree relatives died of heart problems before age 60, a person's own risk of early heart disease rose five-fold.
It's important to note that the power of lifestyle changes to offset this potential is not clear. So, while the risk may be increased for individuals with a family history, there is still a very likely benefit to making improvements in blood pressure, cholesterol, exercise, and diet. The increased risk should be used as information and perhaps inspiration to make improvements, but not as a foregone lifespan conclusion.
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New research shows that in a two-day-old mouse, a heart attack causes active stem cells to grow new heart cells and a few months after an attack, the heart is mostly repaired.
But in an adult mouse, recovery from such an attack leads to classic after-effects: scar tissue, permanent loss of function, and life-threatening arrhythmias. Via Dr. Stefan Gruenwald
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