A new test for detecting biomarkers for cancer and diabetes is more than 1000x more detailed and 100% faster than existing methods, report researchers. Named 2D Mass Spectrometry (2DMS), the test provides a new tool in the rapidly expanding field research into the structure and function of proteins: Proteomics.
Cancer usually begins in one location and then spreads, but in 3 percent to 5 percent of cancer patients, the tissue where a cancer begins is unknown. Family members of patients with cancers of unknown origin have a higher risk for getting those and other types of cancers, suggests a new study.
Pancreatic cancer tumors spill their molecular secrets into the blood stream, shedding their complete DNA and RNA wrapped inside protective lipid particles that make them ripe for analysis with a liquid biopsy, researchers at report.
A first-of-a-kind drug that interferes with the metabolic activity of gut microbes could one day treat heart disease in humans, according to a mouse study. Dietary supplementation with a compound that is naturally abundant in red wine and olive oil prevented gut microbes from turning unhealthy foods into metabolic byproducts that clog arteries.
Biologists have identified a network of proteins that guides neural synapse formation in Drosophila brains. They discovered that proteins from two different molecular subfamilies bind together selectively.
In recent decades much hope has been based on the development of personalized drug treatments, in which genetic tests determine the choice and optimal dose of medication for each individual patient. However, the real breakthrough is still to be seen, and now researchers show in two separate scientific papers that many more gene variants affect how a person responds to medication than previously thought – and thus that today’s analytical tools are too coarse.
Proteins are like bricks that form our cells and they are built by the orders given by our genetic material, DNA. In human diseases, eventually DNA alterations modify proteins and they don't do their normal function, either by excess or defect. But recently scientists have started to find alterations of proteins without an obvious damage of the gene that produces them.
A drug-like molecule can activate innate immunity and induce genes to control infection in a range of RNA viruses, including West Nile, dengue, hepatitis C, influenza A, respiratory syncytial, Nipah, Lassa and Ebola, according to new research.
An MRI contrast agent that can pass through the blood-brain barrier will allow doctors to detect deadly brain tumors called gliomas earlier, say researchers. This ability opens the door to make this fatal cancer treatable.
The role sphingolipids play in the death of cancer cells is now better understood, thanks to a recent study. The research traces how levels of various sphingolipids spike inside cancer cells when the cells are undergoing a highly organized form of cellular death called apoptosis.
Genetically identical sibling cells do not always behave the same way. So far this has been attributed to random molecular reactions. Now systems biologists have discovered an overlooked consequence of the spatial separation of cells into a nucleus and a cytoplasm. Building on top of this insight they could predict with supercomputers the activity of genes in individual human cells.
Integrins help cells communicate with and adapt to their environment. Also cancer cells depend on their properties to survive and spread throughout the body. Now scientists have successfully developed a small, highly active molecule that binds to a specific integrin that operates in many types of cancer. In the future it may allow patient-specific diagnoses and subsequent targeted treatment of tumor cells.
Una parte de la resistencia a los antibióticos de las bacterias patógenas se debe al uso de antibióticos de amplio espectro en las primeras etapas de la infección porque no se ha conseguido determinar qué especie concreta es la causante.
The mechanism by which tumor cells elevate levels of MDM4, a protein that is highly expressed in cancer cells but not in normal adult tissues, has been revealed by researchers. The team has also found that the mechanism can be interfered with antisense oligonucleotides to suppress cancer growth.
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