The mantra that saturated fat is bad for you and must be removed to reduce the risk of heart disease has been drummed into public consciousness for almost four decades. But independent scientific evidence shows that this advice has paradoxically increased our cardiovascular risk. And the obsession with total cholesterol levels has led to the over- medication of millions with statins with yet more harm than benefit.
Recent prospective cohort studies have not supported any significant association between saturated fat intake and cardiovascular risk. If anything, saturated fat has been found to be protective in many studies.
Current government guidelines on dietary fat are not supported by independent evidence from randomized control trials.
First, there is no association between heart disease, blood cholesterol and dietary fat; and a meta-analysis of the RCTs available at the time the first guidelines were introduced confirmed that there was no relationship between dietary fat intake and deaths from CHD or all-causes, despite significant reduction in cholesterol levels in the intervention and control groups.
Second, low fat diets (in accordance with government guidelines) are actually bad for health in comparison with low carbohydrate, low glycaemic diets of the same calorific content.
Third, independent population studies showed that while low cholesterol level is a significant risk factor for cardiovascular and non-cardiac mortality in healthy individuals, high cholesterol level is not.
Finally, the advice to specifically increase omega-6 PFUAs intake may actually increase the risks of CHD and death.
In view of this evidence on the lack of association between heart disease, blood cholesterol and dietary fat, the mass medication of healthy people with cholesterol lowering statins has resulted in yet more harm than good (see Statins for the Healthy are Harmful ).
In one study that used experimental animals, magnesium supplementation inhibited the deposit of lipids in the walls of the aorta — that is, it inhibited plaque formation, a major factor in atherosclerosis.
Chronic fatigue syndrome/myalgic encephalomyelitis, an illness characterized by unexplained fatigue lasting 6 months or more, is often linked to dysfunctional neuroendocrine and immune responses, including abnormal production of several cytokines...
Recent evidence indicates that oxytocin is an anxiolytic hormone, as it decreases the release of stress hormones, while oxytocin imbalances have been associated with both anxiety (HJ Lee et al., Prog Neurobiol, 2009, 88:127) and depression.
San Francisco, California, Dec. 16, 2014 (GLOBE NEWSWIRE) -- Global autoimmune disease diagnostics market is expected to reach USD 14.17 billion by 2020, according to a new study by Grand View Research, Inc.
In this review, we will dissect the microbial actors thought to be involved in the HH as well as their immunomodulatory mechanisms as emphasized by experimental studies, with a particular attention on parasites. Thereafter, we will review the early clinical trials using helminthes’ derivatives focusing on autoimmune diseases.
Researchers say they have found 'unequivocal' evidence that chronic fatigue syndrome is not an imaginary illness, but a genuine condition that causes the immune system to go into overdrive, leaving patients feeling perpetually exhausted.
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