For many, social media is simply a place to post links to content they've created in hopes that thousands will see it, click through, and share with their followers. So they have profiles on every network, and every network looks exactly the same; line after line of self-promotion. This is not going to bring results.
Social marketing is a lot of work, and it takes time listening and responding. It's social, and anything social takes an investment of effort and skill.
Check out these 41 resources that will help you develop the skills needed to be effective on social media. You may want to bookmark this post so you can easily refer to it again later....
Au début de la recherche, c'est l'existence démontrable ou non d'un lien causal entre l'usage des TICE par les enseignants et le taux de réussite des élèves qui a d'abord retenu l'attention .... Une très complète recension des impacts des TIC ...
Via Marcel Lebrun
On his many failed experiments, Thomas Edison once said,
"I have learned fifty thousand ways it cannot be done and therefore I am fifty thousand times nearer the final successful experiment."
Elsewhere, we have dug into the data on startups that died (as well as those acquihired) and found they usually die 20 months after raising financing and after having raised about $1.3 million.
So we thought it would be useful to see how startup founders and investors describe their failures. While not exactly “50,000 ways it cannot be done,” below is a compilation of startup post-mortems that describe the factors that drove a startup’s demise.
Most of the failures have been told by the company’s founders, but in a few cases, we did find a couple from competitors, early employees, or investors including Roger Ehrenberg (now of IA Ventures) and Bruce Booth (Atlas Venture). They are in no particular order, and there is something to learn from each and every one of them....
Despite the importance of the co-receptor PD-1 in T cell immunity, the upstream signaling pathway that regulates PD-1 expression has not been defined. Glycogen synthase kinase 3 (GSK-3, isoforms α and β) is a serine-threonine kinase implicated in cellular processes. Here, we identified GSK-3 as a key upstream kinase that regulated PD-1 expression in CD8+ T cells. GSK-3 siRNA downregulation, or inhibition by small molecules, blocked PD-1 expression, resulting in increased CD8+ cytotoxic T lymphocyte (CTL) function. Mechanistically, GSK-3 inactivation increased Tbx21 transcription, promoting enhanced T-bet expression and subsequent suppression of Pdcd1 (encodes PD-1) transcription in CD8+ CTLs. Injection of GSK-3 inhibitors in mice increased in vivo CD8+ OT-I CTL function and the clearance of murine gamma-herpesvirus 68 and lymphocytic choriomeningitis clone 13 and reversed T cell exhaustion. Our findings identify GSK-3 as a regulator of PD-1 expression and demonstrate the applicability of GSK-3 inhibitors in the modulation of PD-1 in immunotherapy.
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